Expression of a dominant negative TrkB receptor, T1, reveals a requirement for presynaptic signaling in BDNF-induced synaptic potentiation in cultured hippocampal neurons
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Long-Term Potentiation and MemoryNeurotrophins: roles in neuronal development and functionExercise promotes the expression of brain derived neurotrophic factor (BDNF) through the action of the ketone body β-hydroxybutyrateSynaptic secretion of BDNF after high-frequency stimulation of glutamatergic synapses.BDNF enhances quantal neurotransmitter release and increases the number of docked vesicles at the active zones of hippocampal excitatory synapsesBDNF-mediated neurotransmission relies upon a myosin VI motor complexSynaptic and extrasynaptic localization of brain-derived neurotrophic factor and the tyrosine kinase B receptor in cultured hippocampal neurons.Functional interactions between steroid hormones and neurotrophin BDNFA new aspect of the TrkB signaling pathway in neural plasticity.Neurotrophin-mediated potentiation of neuronal injury.Retrograde signaling at central synapsesBoth pre- and postsynaptic activity of Nsf prevents degeneration of hair-cell synapsesBrain-derived neurotrophic factor enhances calcium regulatory mechanisms in human airway smooth muscle.Postsynaptic GluA1 enables acute retrograde enhancement of presynaptic function to coordinate adaptation to synaptic inactivity.BDNF regulates the expression and distribution of vesicular glutamate transporters in cultured hippocampal neurons.Local presynaptic activity gates homeostatic changes in presynaptic function driven by dendritic BDNF synthesisSynaptic reliability correlates with reduced susceptibility to synaptic potentiation by brain-derived neurotrophic factor.Nerve growth factor acutely reduces chemical transmission by means of postsynaptic TrkA-like receptors in squid giant synapse.TrkB gene transfer does not alter hippocampal neuronal loss and cognitive deficits following traumatic brain injury in micePresynaptic and postsynaptic NMDA receptors mediate distinct effects of brain-derived neurotrophic factor on synaptic transmission.Control of synaptic vesicle endocytosis by an extracellular signalling molecule.Endogenous truncated TrkB.T1 receptor regulates neuronal complexity and TrkB kinase receptor function in vivo.Differential activity-dependent secretion of brain-derived neurotrophic factor from axon and dendrite.In vivo evidence that truncated trkB.T1 participates in nociception.BDNF deletion or TrkB impairment in amygdala inhibits both appetitive and aversive learning.The link between inflammation, synaptic transmission and neurodegeneration in multiple sclerosis.Brain-derived neurotrophic factor in the airways.Neurotrophin Signaling and Stem Cells-Implications for Neurodegenerative Diseases and Stem Cell Therapy.Neurotrophic factor neurotrophin-4 regulates ameloblastin expression via full-length TrkB.Pre-synaptic TrkB in basolateral amygdala neurons mediates BDNF signaling transmission in memory extinction.Mechanisms of BDNF regulation in asthmatic airway smooth muscle.Brain-derived neurotrophic factor-induced potentiation of Ca(2+) oscillations in developing cortical neurons.TRPC3 regulates release of brain-derived neurotrophic factor from human airway smooth muscle.TrkB-T1 receptors on Muller cells play critical role in brain-derived neurotrophic factor-mediated photoreceptor protection against phototoxicity.Neurotrophins act at presynaptic terminals to activate synapses among cultured hippocampal neurons.BDNF reduces miniature inhibitory postsynaptic currents by rapid downregulation of GABA(A) receptor surface expression.Long-term potentiation in the dentate gyrus of the rat hippocampus is accompanied by brain-derived neurotrophic factor-induced activation of TrkB.Rapid BDNF-induced retrograde synaptic modification in a developing retinotectal system.Brain-derived neurotrophic factor triggers a rapid glutamate release through increase of intracellular Ca(2+) and Na(+) in cultured cerebellar neurons.Regulation of TRKB surface expression by brain-derived neurotrophic factor and truncated TRKB isoforms.
P2860
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P2860
Expression of a dominant negative TrkB receptor, T1, reveals a requirement for presynaptic signaling in BDNF-induced synaptic potentiation in cultured hippocampal neurons
description
1998 nî lūn-bûn
@nan
1998 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Expression of a dominant negat ...... n cultured hippocampal neurons
@ast
Expression of a dominant negat ...... n cultured hippocampal neurons
@en
Expression of a dominant negat ...... cultured hippocampal neurons.
@nl
type
label
Expression of a dominant negat ...... n cultured hippocampal neurons
@ast
Expression of a dominant negat ...... n cultured hippocampal neurons
@en
Expression of a dominant negat ...... cultured hippocampal neurons.
@nl
prefLabel
Expression of a dominant negat ...... n cultured hippocampal neurons
@ast
Expression of a dominant negat ...... n cultured hippocampal neurons
@en
Expression of a dominant negat ...... cultured hippocampal neurons.
@nl
P2093
P2860
P356
P1476
Expression of a dominant negat ...... n cultured hippocampal neurons
@en
P2093
P2860
P304
10884-10889
P356
10.1073/PNAS.95.18.10884
P407
P50
P577
1998-09-01T00:00:00Z