Slow toxins, biologic markers, and long-latency neurodegenerative disease in the western Pacific region.
about
Is neurodegenerative disease a long-latency response to early-life genotoxin exposure?The cycad genotoxin MAM modulates brain cellular pathways involved in neurodegenerative disease and cancer in a DNA damage-linked mannerEvidence of active nerve cell degeneration in the substantia nigra of humans years after 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine exposureGenotoxicants target distinct molecular networks in neonatal neuronsAnimal models of brain maldevelopment induced by cycad plant genotoxinsConservation Implications of Chamorro Consumption of Flying Foxes as a Possible Cause of Amyotrophic Lateral Sclerosis-Parkinsonism Dementia Complex in GuamDamage and repair of nerve cell DNA in toxic stress.On the decline and etiology of high-incidence motor system disease in West Papua (southwest New Guinea).Biomagnification of cyanobacterial neurotoxins and neurodegenerative disease among the Chamorro people of GuamReturn of the cycad hypothesis - does the amyotrophic lateral sclerosis/parkinsonism dementia complex (ALS/PDC) of Guam have new implications for global health?Beyond Guam: the cyanobacteria/BMAA hypothesis of the cause of ALS and other neurodegenerative diseases.Genetic epidemiology of amyotrophic lateral sclerosis.PARADOXICAL RESPONSES TO NEUROTOXIC STERYL GLYCOSIDES: INSIGHTS FROM A CELLULAR MODEL OF ALSPDC.Biotransfer of β-N-methylamino-L-alanine (BMAA) in a eutrophicated freshwater lake.Are human neurodegenerative disorders linked to environmental chemicals with excitotoxic properties?Does the cycad genotoxin MAM implicated in Guam ALS-PDC induce disease-relevant changes in mouse brain that includes olfaction?Spatial clustering of amyotrophic lateral sclerosis and the potential role of BMAA.A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.Overview of the potent cyanobacterial neurotoxin β-methylamino-L-alanine (BMAA) and its analytical determination.Disease progression in amyotrophic lateral sclerosis: predictors of survival.Comparative human and rat neurospheres reveal species differences in chemical effects on neurodevelopmental key events.Production of the neurotoxin BMAA by a marine cyanobacterium.BMAA inhibits nitrogen fixation in the cyanobacterium Nostoc sp. PCC 7120.Acute basal ganglia infarction in propionic acidemia.The natural non-protein amino acid N-β-methylamino-L-alanine (BMAA) is incorporated into protein during synthesis.Cyanobacterial neurotoxin BMAA in ALS and Alzheimer's disease.Motor neuron disease on Guam: geographic and familial occurrence, 1956-85.Nodding syndrome in Uganda is a tauopathy
P2860
Q26824755-28D9F371-7BB7-4A62-9DE3-2B7BF51D52DAQ27335106-074C05B5-9E64-4073-8FB5-4E6869F423EDQ28145614-5886E81B-0FBE-4C57-B1A0-030A05C9B9BBQ28392440-5B4F0C5D-CA0E-4EDF-AA1B-F8B1A5DDA5D2Q28655111-82D31085-B415-42FE-888F-350AC94D72DCQ29041676-A27482AA-0895-4E3D-8F3E-7D0A5E356F5BQ30759422-FD5C1BD6-FC7B-478C-9ECB-FF7A54559E0AQ33221450-5EABD550-0AB6-422F-93AB-6646657C21A0Q33973850-0E26A7C6-AE66-4E7C-90BD-FEE745FF117FQ33988530-B9FACB8E-15E6-4621-AFF0-134A76CFC45EQ34020244-83D0AE70-5EEC-410F-8AD3-CEA3E3F865D2Q35083574-CD38D7D8-D692-4E56-A51F-A25995135CD4Q35176825-344D9B71-A302-458A-AC3A-C8B0DB47564EQ35228202-D150469D-046A-4B92-ADD7-B109B1FB8327Q35230778-087CF109-3727-4CF5-B4B5-B943937C5D32Q35836177-35589C51-7DFE-4214-A8DB-E3E18AC0500BQ37972904-4A7EB35F-089D-4CF0-AFF3-0C15502C7241Q38734183-8F01CE19-A9B4-44F1-BCC9-96D6E9F90C42Q38960608-C67D9D25-5D2A-480E-8911-1BF472625FCFQ40642209-B20DBEA4-A8BE-4816-83FE-1B2F026B7545Q40689918-C37283CE-EDA7-487F-A67A-D2A25EBEB8BFQ41917017-44C9D97E-C969-46C4-8286-EDAC9C6D5890Q42591398-EFF1DC78-59D3-460C-83BA-C189102F3A1DQ44957122-C495CB6F-A952-4FB1-9604-1692B2DB17E0Q45191700-E30E08B5-05DF-4E81-B3E7-D5A297FF1771Q45292859-DE6E3C60-43FB-411F-A506-83D683782221Q50999191-06EC8049-C95B-490F-A336-330E3291C1FBQ56909433-8C498EC8-F4DD-48C4-A51A-E5B54624A690
P2860
Slow toxins, biologic markers, and long-latency neurodegenerative disease in the western Pacific region.
description
1991 nî lūn-bûn
@nan
1991 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Slow toxins, biologic markers, ...... in the western Pacific region.
@ast
Slow toxins, biologic markers, ...... in the western Pacific region.
@en
Slow toxins, biologic markers, ...... in the western Pacific region.
@nl
type
label
Slow toxins, biologic markers, ...... in the western Pacific region.
@ast
Slow toxins, biologic markers, ...... in the western Pacific region.
@en
Slow toxins, biologic markers, ...... in the western Pacific region.
@nl
prefLabel
Slow toxins, biologic markers, ...... in the western Pacific region.
@ast
Slow toxins, biologic markers, ...... in the western Pacific region.
@en
Slow toxins, biologic markers, ...... in the western Pacific region.
@nl
P2093
P1433
P1476
Slow toxins, biologic markers, ...... in the western Pacific region.
@en
P2093
A C Ludolph
P S Spencer
P304
62-6; discussion 66-8
P356
10.1212/WNL.41.5_SUPPL_2.62
P407
P433
P577
1991-05-01T00:00:00Z