In a murine tuberculosis model, the absence of homeostatic chemokines delays granuloma formation and protective immunity. - Wikidata - wikimedia - TriplyDB

In a murine tuberculosis model, the absence of homeostatic chemokines delays granuloma formation and protective immunity.

In a murine tuberculosis model, the absence of homeostatic chemokines delays granuloma formation and protective immunity.

http://www.wikidata.org/entity/Q33565121

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Mathematical modeling of tuberculosis bacillary counts and cellular populations in the organs of infected mice.CXCR5⁺ T helper cells mediate protective immunity against tuberculosis CD34 is required for dendritic cell trafficking and pathology in murine hypersensitivity pneumonitis Inducible Bronchus-Associated Lymphoid Tissue: Taming Inflammation in the Lung Lactoferrin: A Modulator for Immunity against Tuberculosis Related Granulomatous Pathology Ectopic lymphoid follicles: inducible centres for generating antigen-specific immune responses within tissues Profiling early lung immune responses in the mouse model of tuberculosis.Unexpected role for IL-17 in protective immunity against hypervirulent Mycobacterium tuberculosis HN878 infection.Chemokine gene expression in lung CD8 T cells correlates with protective immunity in mice immunized intra-nasally with Adenovirus-85A To achieve an earlier IFN-γ response is not sufficient to control Mycobacterium tuberculosis infection in mice.The stress-response factor SigH modulates the interaction between Mycobacterium tuberculosis and host phagocytes Conservation and divergence in Toll-like receptor 4-regulated gene expression in primary human versus mouse macrophages.An interferon-related signature in the transcriptional core response of human macrophages to Mycobacterium tuberculosis infection.Lipocalin 2 regulates inflammation during pulmonary mycobacterial infections.A lympho-follicular microenvironment is required for pathological prion protein deposition in chronically inflamed tissues from scrapie-affected sheep The role of B cells and humoral immunity in Mycobacterium tuberculosis infection Mouse model of tuberculosis The development and function of mucosal lymphoid tissues: a balancing act with micro-organisms.Expression of the leptin receptor outside of bone marrow-derived cells regulates tuberculosis control and lung macrophage MHC expression.ICOS and Bcl6-dependent pathways maintain a CD4 T cell population with memory-like properties during tuberculosis.The DosR Regulon Modulates Adaptive Immunity and Is Essential for Mycobacterium tuberculosis Persistence.DosS Is required for the complete virulence of mycobacterium tuberculosis in mice with classical granulomatous lesions Interleukin 27R regulates CD4+ T cell phenotype and impacts protective immunity during Mycobacterium tuberculosis infection.The tuberculous granuloma: an unsuccessful host defence mechanism providing a safety shelter for the bacteria?Infection with Mycobacterium tuberculosis induces the Warburg effect in mouse lungs.Chemokines shape the immune responses to tuberculosis.Effects of B Cell Depletion on Early Mycobacterium tuberculosis Infection in Cynomolgus Macaques.Mucosal pre-exposure to Th17-inducing adjuvants exacerbates pathology after influenza infection.CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis.Understanding delayed T-cell priming, lung recruitment, and airway luminal T-cell responses in host defense against pulmonary tuberculosis.Protection versus pathology in tuberculosis: recent insights.The role of viruses in autoreactive B cell activation within tertiary lymphoid structures in autoimmune diseases.Location, location, location: the impact of migratory heterogeneity on T cell function.Immunological mechanisms contributing to the double burden of diabetes and intracellular bacterial infections.Chemokines in tuberculosis: the good, the bad and the ugly.The balance between protective and pathogenic immune responses in the TB-infected lung.The onset of adaptive immunity in the mouse model of tuberculosis and the factors that compromise its expression.Understanding the pathophysiology of the human TB lung granuloma using in vitro granuloma models.IL-23 is required for long-term control of Mycobacterium tuberculosis and B cell follicle formation in the infected lung.Cytokines and Chemokines in Mycobacterium tuberculosis Infection.

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P2860

In a murine tuberculosis model, the absence of homeostatic chemokines delays granuloma formation and protective immunity.

http://www.wikidata.org/entity/Q33565121

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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In a murine tuberculosis model ...... ation and protective immunity.

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Cynthia A Martino

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David L Woodland

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Gary M Winslow

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Jeffrey J Fountain

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Shabaana A Khader

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Troy D Randall

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P2860

B cell-attracting chemokine 1, a human CXC chemokine expressed in lymphoid tissues, selectively attracts B lymphocytes via BLR1/CXCR5

A chemokine expressed in lymphoid high endothelial venules promotes the adhesion and chemotaxis of naive T lymphocytes

A chemokine-driven positive feedback loop organizes lymphoid follicles

CXCL13 is required for B1 cell homing, natural antibody production, and body cavity immunity

Immunology of tuberculosis

Differential expression of FIZZ1 and Ym1 in alternatively versus classically activated macrophages.

Chemokines and cell migration in secondary lymphoid organs.

The reduced expression of 6Ckine in the plt mouse results from the deletion of one of two 6Ckine genes.

IL-23 compensates for the absence of IL-12p70 and is essential for the IL-17 response during tuberculosis but is dispensable for protection and antigen-specific IFN-gamma responses if IL-12p70 is available.

Inducible bronchus-associated lymphoid tissue (iBALT) in patients with pulmonary complications of rheumatoid arthritis.

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Javier Rangel-Moreno

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