Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis - Wikidata - wikimedia - TriplyDB

Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis

Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis

http://www.wikidata.org/entity/Q33579424

about

Mactinin: a modulator of the monocyte response to inflammation Differentiation of monocytes into multinucleated giant bone-resorbing cells: two-step differentiation induced by nurse-like cells and cytokines Mechanisms of bone loss in inflammatory arthritis: diagnosis and therapeutic implications.Mice lacking monocyte chemoattractant protein 1 have enhanced susceptibility to an interstitial polymicrobial infection due to impaired monocyte recruitment Ligation of TLR5 promotes myeloid cell infiltration and differentiation into mature osteoclasts in rheumatoid arthritis and experimental arthritis.Angiopoietin-like 4 is over-expressed in rheumatoid arthritis patients: association with pathological bone resorption Should EMT of Cancer Cells Be Understood as Epithelial-Myeloid Transition?Human arthroplasty derived macrophages differentiate into osteoclastic bone resorbing cells Human tumour-associated macrophages differentiate into osteoclastic bone-resorbing cells.Mechanisms of TNF-alpha- and RANKL-mediated osteoclastogenesis and bone resorption in psoriatic arthritis.The effect of vascular endothelial growth factor on osteoclastogenesis in rheumatoid arthritis Do sex hormones modulate the synovial macrophages in rheumatoid arthritis?Synovial macrophage-osteoclast differentiation in inflammatory arthritis Receptor activator NF-kappaB ligand (RANKL) expression in synovial tissue from patients with rheumatoid arthritis, spondyloarthropathy, osteoarthritis, and from normal patients: semiquantitative and quantitative analysis.Identification of a human peripheral blood monocyte subset that differentiates into osteoclasts.Identification of cell types responsible for bone resorption in rheumatoid arthritis and juvenile rheumatoid arthritis Genetic mouse models for bone studies--strengths and limitations Upregulation of tumor necrosis factor receptor-associated factor 6 correlated with synovitis severity in rheumatoid arthritis Periodontitis and rheumatoid arthritis: a review.Regulation of bone destruction in rheumatoid arthritis through RANKL-RANK pathways Atorvastatin inhibits osteoclastogenesis by decreasing the expression of RANKL in the synoviocytes of rheumatoid arthritis Giant cells in pigmented villo nodular synovitis express an osteoclast phenotype.Activated γδ T cells inhibit osteoclast differentiation and resorptive activity in vitro.Immune regulation of bone loss by Th17 cells.The role of osteoprotegerin (OPG) and estrogen receptor (ER-α) gene polymorphisms in rheumatoid arthritis.Histone deacetylase inhibitors as suppressors of bone destruction in inflammatory diseases.Hypoxia-inducible factor 1-alpha does not regulate osteoclastogenesis but enhances bone resorption activity via prolyl-4-hydroxylase 2.Osteoimmunology: memorandum for rheumatologists.Kinetics of bone protection by recombinant osteoprotegerin therapy in Lewis rats with adjuvant arthritis.Suppressive effect of iron on concanavalin A-induced multinucleated giant cell formation by human monocytes.The osteoclast: a potential therapeutic target of bone and joint destruction in rheumatoid arthritis.Structural, cellular, and molecular evaluation of bone erosion in experimental models of rheumatoid arthritis: assessment by μCT, histology, and serum biomarkers.Collagen-induced arthritis: severity and immune response attenuation using multivalent N-acetyl glucosamine.Identification of RANKL in osteolytic lesions of the facial skeleton.Neutrophil Microvesicles from Healthy Control and Rheumatoid Arthritis Patients Prevent the Inflammatory Activation of Macrophages.RANKL: A therapeutic target for bone destruction in rheumatoid arthritis.A correlative study of clinical and histological findings of revision hip arthroplasty for rheumatoid arthritis and inflammatory joint disease.Osteoclast differentiation factor induces synovial macrophage-osteoclast differentiation in rheumatoid arthritis.Dual regulation of osteoclast differentiation by periodontal ligament cells through RANKL stimulation and OPG inhibition.Macrophage-osteoclast differentiation and bone resorption in osteoarthrotic subchondral acetabular cysts.

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P2860

Human osteoclast formation and bone resorption by monocytes and synovial macrophages in rheumatoid arthritis

http://www.wikidata.org/entity/Q33579424

description

1996 nî lūn-bûn

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1996 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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1996年の論文

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1996年論文

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1996年論文

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1996年论文

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Human osteoclast formation and ...... phages in rheumatoid arthritis

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Human osteoclast formation and ...... phages in rheumatoid arthritis

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Human osteoclast formation and ...... phages in rheumatoid arthritis

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Human osteoclast formation and ...... phages in rheumatoid arthritis

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Annals of the Rheumatic Diseases

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Human osteoclast formation and ...... phages in rheumatoid arthritis

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A Sabokbar

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N A Athanasou

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S Neale

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Y Fujikawa

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P2860

Abundant calcitonin receptors in isolated rat osteoclasts. Biochemical and autoradiographic characterization

The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis

Monocyte activation in early onset rheumatoid arthritis

Leucocyte integrin and CR1 expression on peripheral blood leucocytes of patients with rheumatoid arthritis

Synovial tissue macrophages and joint erosion in rheumatoid arthritis.

Bone resorption by cells isolated from rheumatoid synovium.

Origin of osteoclasts: mature monocytes and macrophages are capable of differentiating into osteoclasts under a suitable microenvironment prepared by bone marrow-derived stromal cells.

Resorption of bone by isolated rabbit osteoclasts.

Immunophenotypic differences between osteoclasts and macrophage polykaryons: immunohistological distinction and implications for osteoclast ontogeny and function.

Synovium as a source of interleukin 6 in vitro. Contribution to local and systemic manifestations of arthritis

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