Transient calcium release induced by successive increments of inositol 1,4,5-trisphosphate.
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Single-cell imaging of graded Ins(1,4,5)P3 production following G-protein-coupled-receptor activationHeteroligomers of type-I and type-III inositol trisphosphate receptors in WB rat liver epithelial cellsThree additional inositol 1,4,5-trisphosphate receptors: molecular cloning and differential localization in brain and peripheral tissuesComplexity in biological signaling systems.Micromolar and submicromolar Ca2+ spikes regulating distinct cellular functions in pancreatic acinar cells.Stereospecific inositol 1,4,5-[32P]trisphosphate binding to isolated rat liver nuclei: evidence for inositol trisphosphate receptor-mediated calcium release from the nucleus.Inositol trisphosphate receptor Ca2+ release channels.Redox-regulated heterogeneous thresholds for ligand recruitment among InsP3R Ca2+-release channels.Fast kinetics of calcium liberation induced in Xenopus oocytes by photoreleased inositol trisphosphateSimplification and analysis of models of calcium dynamics based on IP3-sensitive calcium channel kinetics.Highly cooperative Ca2+ elevations in response to Ins(1,4,5)P3 microperfusion through a patch-clamp pipette.Quantal calcium release and calcium entry in the pancreatic acinar cell.Transient inositol 1,4,5-trisphosphate-induced Ca2+ release: a model based on regulatory Ca(2+)-binding sites along the permeation pathwayCalcium release in HSY cells conforms to a steady-state mechanism involving regulation of the inositol 1,4,5-trisphosphate receptor Ca2+ channel by luminal [Ca2+].Rapid ligand-regulated gating kinetics of single inositol 1,4,5-trisphosphate receptor Ca2+ release channels.'Quantal' Ca(2+) release reassessed--a clue to oscillation and synchronization.Reversible desensitization of inositol trisphosphate-induced calcium release provides a mechanism for repetitive calcium spikes.From puffs to global Ca2+ signals: how molecular properties shape global signals.IP(3) receptors: toward understanding their activation.Cytosolic inositol 1,4,5-trisphosphate dynamics during intracellular calcium oscillations in living cellsInternal calcium release and activation of sea urchin eggs by cGMP are independent of the phosphoinositide signaling pathway.Graded recruitment and inactivation of single InsP3 receptor Ca2+-release channels: implications for quantal [corrected] Ca2+release.Phasic characteristic of elementary Ca(2+) release sites underlies quantal responses to IP(3).Ca2+ homeostasis in the agonist-sensitive internal store: functional interactions between mitochondria and the ER measured In situ in intact cells.Regulation of inositol trisphosphate receptors by luminal Ca2+ contributes to quantal Ca2+ mobilizationMechanisms responsible for quantal Ca2+ release from inositol trisphosphate-sensitive calcium stores.Phytic acid in health and disease.Do submaximal InsP3 concentrations only induce the partial discharge of permeabilized hepatocyte calcium pools because of the concomitant reduction of intraluminal Ca2+ concentration?Control of inositol 1,4,5-trisphosphate-induced Ca2+ release by cytosolic Ca2+.Calmodulin trapping by calcium-calmodulin-dependent protein kinase.Quantal Ca2+ mobilization by ryanodine receptors is due to all-or-none release from functionally discrete intracellular stores.Inhibition of the cerebellar inositol 1,4,5-trisphosphate-sensitive Ca2+ channel by ethanol and other aliphatic alcohols.Synergistic effects of inositol 1,3,4,5-tetrakisphosphate on inositol 2,4,5-triphosphate-stimulated Ca2+ release do not involve direct interaction of inositol 1,3,4,5-tetrakisphosphate with inositol triphosphate-binding sites.A steady-state mechanism can account for the properties of inositol 2,4,5-trisphosphate-stimulated Ca2+ release from permeabilized L1210 cells.A minimal gating model for the cardiac calcium release channel.Multiple mechanisms by which protein kinase A potentiates inositol 1,4,5-trisphosphate-induced Ca2+ mobilization in permeabilized hepatocytes.Slow kinetics of inositol 1,4,5-trisphosphate-induced Ca2+ release: is the release 'quantal' or 'non-quantal'?Model for receptor-controlled cytosolic calcium oscillations and for external influences on the signal pathway.Different modulatory effects of elevated cyclic AMP on cytosolic Ca2+ spikes induced by phenylephrine or vasopressin in single rat hepatocytes.Quantal responses to inositol 1,4,5-trisphosphate are not a consequence of Ca2+ regulation of inositol 1,4,5-trisphosphate receptors.
P2860
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P2860
Transient calcium release induced by successive increments of inositol 1,4,5-trisphosphate.
description
1990 nî lūn-bûn
@nan
1990 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@ast
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@en
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@nl
type
label
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@ast
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@en
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@nl
prefLabel
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@ast
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@en
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@nl
P2860
P356
P1476
Transient calcium release indu ...... inositol 1,4,5-trisphosphate.
@en
P2093
P2860
P304
P356
10.1073/PNAS.87.10.3841
P407
P577
1990-05-01T00:00:00Z