REDD1 is essential for stress-induced synaptic loss and depressive behavior. - Wikidata - wikimedia - TriplyDB

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

http://www.wikidata.org/entity/Q33591105

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Stress weakens prefrontal networks: molecular insults to higher cognition GABA interneurons mediate the rapid antidepressant-like effects of scopolamine.Is REDD1 a Metabolic Éminence Grise?Targeting Prefrontal Cortical Systems for Drug Development: Potential Therapies for Cognitive Disorders RTP801/REDD1: a stress coping regulator that turns into a troublemaker in neurodegenerative disorders Synaptic plasticity and depression: new insights from stress and rapid-acting antidepressants.Social defeat stress causes depression-like behavior with metabolite changes in the prefrontal cortex of rats.How do antidepressants work? New perspectives for refining future treatment approaches.The Evidence for Altered BDNF Expression in the Brain of Rats Reared or Housed in Social Isolation: A Systematic Review Ketamine and rapid-acting antidepressants: a window into a new neurobiology for mood disorder therapeutics Emerging treatment mechanisms for depression: focus on glutamate and synaptic plasticity.Chronic Stimulation of Alpha-2A-Adrenoceptors With Guanfacine Protects Rodent Prefrontal Cortex Dendritic Spines and Cognition From the Effects of Chronic Stress Ketamine as a promising prototype for a new generation of rapid-acting antidepressants.Ribosomal protein S6 kinase 1 signaling in prefrontal cortex controls depressive behavior.Spine synapse remodeling in the pathophysiology and treatment of depression.Involvement of normalized NMDA receptor and mTOR-related signaling in rapid antidepressant effects of Yueju and ketamine on chronically stressed mice.Pathogenesis of depression: Insights from human and rodent studies.Neuronal correlates of depression.Synergistic antidepressant-like effect of ferulic acid in combination with piperine: involvement of monoaminergic system.High-Fat Diet Induced Anxiety and Anhedonia: Impact on Brain Homeostasis and Inflammation.Sex-specific variation in signaling pathways and gene expression patterns in human leukocytes in response to endotoxin and exercise.Molecular and Cellular Mechanisms of Rapid-Acting Antidepressants Ketamine and Scopolamine Synaptic Plasticity, Metaplasticity and Depression.Cav1.2 channels mediate persistent chronic stress-induced behavioral deficits that are associated with prefrontal cortex activation of the p25/Cdk5-glucocorticoid receptor pathway.New perspectives on the involvement of mTOR in depression as well as in the action of antidepressant drugs.KETAMINE'S MECHANISM OF ACTION: A PATH TO RAPID-ACTING ANTIDEPRESSANTS.Effects of escitalopram and paroxetine on mTORC1 signaling in the rat hippocampus under chronic restraint stress.Synaptic alterations associated with depression and schizophrenia: potential as a therapeutic target.L-type Ca2+ channels in mood, cognition and addiction: integrating human and rodent studies with a focus on behavioural endophenotypes.Prefrontal Cortex GABAergic Deficits and Circuit Dysfunction in the Pathophysiology and Treatment of Chronic Stress and Depression.mTORC1 senses stresses: Coupling stress to proteostasis.From Gene to Behavior: L-Type Calcium Channel Mechanisms Underlying Neuropsychiatric Symptoms.Age- and Sex-Dependent Impact of Repeated Social Stress on Intrinsic and Synaptic Excitability of the Rat Prefrontal Cortex.Cacna1c in the Prefrontal Cortex Regulates Depression-Related Behaviors via REDD1.The long-lasting antidepressant effects of rapastinel (GLYX-13) are associated with a metaplasticity process in the medial prefrontal cortex and hippocampus.Identification of genes and gene pathways associated with major depressive disorder by integrative brain analysis of rat and human prefrontal cortex transcriptomes.Suppression of REDD1 in osteoarthritis cartilage, a novel mechanism for dysregulated mTOR signaling and defective autophagy The effects of chronic stress on health: new insights into the molecular mechanisms of brain-body communication.Neural Plasticity Is Involved in Physiological Sleep, Depressive Sleep Disturbances, and Antidepressant Treatments.Circuit and synaptic mechanisms of repeated stress: Perspectives from differing contexts, duration, and development.

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P2860

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

http://www.wikidata.org/entity/Q33591105

description

2014 nî lūn-bûn

@nan

2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@ast

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@en

type

label

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@ast

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@en

prefLabel

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@ast

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@en

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Nature Medicine

P1476

REDD1 is essential for stress-induced synaptic loss and depressive behavior.

@en

P2093

Bhavya Voleti

http://www.w3.org/2001/XMLSchema#string

Catharine Duman

http://www.w3.org/2001/XMLSchema#string

Christopher Rex

http://www.w3.org/2001/XMLSchema#string

Craig A Stockmeier

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George K Aghajanian

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Jaime G Maldonado-Aviles

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Kristie T Ota

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Ronald S Duman

http://www.w3.org/2001/XMLSchema#string

Rong-Jian Liu

http://www.w3.org/2001/XMLSchema#string

Steve Boikess

http://www.w3.org/2001/XMLSchema#string

P2860

Prevalence, severity, and comorbidity of 12-month DSM-IV disorders in the National Comorbidity Survey Replication

Identification of a novel hypoxia-inducible factor 1-responsive gene, RTP801, involved in apoptosis.

mTOR signaling: at the crossroads of plasticity, memory and disease

Stress blunts serotonin- and hypocretin-evoked EPSCs in prefrontal cortex: role of corticosterone-mediated apical dendritic atrophy

Genetic variant BDNF (Val66Met) polymorphism alters anxiety-related behavior

Brain-derived neurotrophic factor induces mammalian target of rapamycin-dependent local activation of translation machinery and protein synthesis in neuronal dendrites

Decreased expression of synapse-related genes and loss of synapses in major depressive disorder

mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists

Effect of brain-derived neurotrophic factor haploinsufficiency on stress-induced remodeling of hippocampal neurons.

Chronic behavioral stress induces apical dendritic reorganization in pyramidal neurons of the medial prefrontal cortex.

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531-535

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10.1038/NM.3513

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P433

5

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20

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Ralph J. DiLeone

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2014-04-13T00:00:00Z

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261609736

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24728411

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4016190

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