Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection. - Wikidata - wikimedia - TriplyDB

Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.

Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.

http://www.wikidata.org/entity/Q33597147

about

Cryptococcus and Phagocytes: Complex Interactions that Influence Disease Outcome Scavenger receptor A modulates the immune response to pulmonary Cryptococcus neoformans infection.Role of IL-17A on resolution of pulmonary C. neoformans infection The absence of serum IgM enhances the susceptibility of mice to pulmonary challenge with Cryptococcus neoformans.Cryptococcus neoformans hyperfilamentous strain is hypervirulent in a murine model of cryptococcal meningoencephalitis.PI3K/Akt signaling pathway modulates influenza virus induced mouse alveolar macrophage polarization to M1/M2b.TLR9 signaling is required for generation of the adaptive immune protection in Cryptococcus neoformans-infected lungs.Fatal disseminated Cryptococcus gattii infection in New Mexico STAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.Interleukin-17 is not required for classical macrophage activation in a pulmonary mouse model of Cryptococcus neoformans infection.Immune modulation mediated by cryptococcal laccase promotes pulmonary growth and brain dissemination of virulent Cryptococcus neoformans in mice.Liposomal targeting of prednisolone phosphate to synovial lining macrophages during experimental arthritis inhibits M1 activation but does not favor M2 differentiation.Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.Effect of cytokine interplay on macrophage polarization during chronic pulmonary infection with Cryptococcus neoformans.Preferential macrophage recruitment and polarization in LPS-induced animal model for COPD: noninvasive tracking using MRI.Cryptococcus neoformans-induced macrophage lysosome damage crucially contributes to fungal virulence.The Assembly of EDC4 and Dcp1a into Processing Bodies Is Critical for the Translational Regulation of IL-6 Is Development of a Vaccine against Cryptococcus neoformans Feasible?Cryptococcal heat shock protein 70 homolog Ssa1 contributes to pulmonary expansion of Cryptococcus neoformans during the afferent phase of the immune response by promoting macrophage M2 polarization Cryptococcus neoformans Infection in Mice Lacking Type I Interferon Signaling Leads to Increased Fungal Clearance and IL-4-Dependent Mucin Production in the Lungs.IL-23 dampens the allergic response to Cryptococcus neoformans through IL-17-independent and -dependent mechanisms.Development of protective inflammation and cell-mediated immunity against Cryptococcus neoformans after exposure to hyphal mutants.Cryptococcal genotype influences immunologic response and human clinical outcome after meningitis.STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformans The Cnes2 locus on mouse chromosome 17 regulates host defense against cryptococcal infection through pleiotropic effects on host immunity Virulence factors identified by Cryptococcus neoformans mutant screen differentially modulate lung immune responses and brain dissemination.Protective immunity against pulmonary cryptococcosis is associated with STAT1-mediated classical macrophage activation.Cryptococcus neoformans growth and protection from innate immunity are dependent on expression of a virulence-associated DEAD-box protein, Vad1.Adjunctive interferon-γ immunotherapy for the treatment of HIV-associated cryptococcal meningitis: a randomized controlled trial.Macrophage M1/M2 polarization dynamically adapts to changes in cytokine microenvironments in Cryptococcus neoformans infection.Modulation of Macrophage Inflammatory Nuclear Factor κB (NF-κB) Signaling by Intracellular Cryptococcus neoformans The intracellular life of Cryptococcus neoformans.Impact of surfactant protein D, interleukin-5, and eosinophilia on Cryptococcosis.Interleukin-17A enhances host defense against cryptococcal lung infection through effects mediated by leukocyte recruitment, activation, and gamma interferon production Mechanisms of cryptococcal virulence and persistence.Induction of protective immunity against cryptococcosis.Classical versus alternative macrophage activation: the Ying and the Yang in host defense against pulmonary fungal infections.Immunity to Cryptococcus neoformans and C. gattii during cryptococcosis Role of dendritic cell-pathogen interactions in the immune response to pulmonary cryptococcal infection.Innate host defenses against Cryptococcus neoformans.

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Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.

http://www.wikidata.org/entity/Q33597147

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Pulmonary infection with an in ...... age activation and protection.

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Pulmonary infection with an in ...... age activation and protection.

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The American Journal of Pathology

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Pulmonary infection with an in ...... age activation and protection.

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Floyd L Wormley

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Mattie L Young

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Sailatha Ravi

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Sarah E Hardison

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P2860

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

IL-4 dependent alternatively-activated macrophages have a distinctive in vivo gene expression phenotype

Alternative activation of macrophages

Toll-like receptors in the induction of the innate immune response

Alternative activation of macrophages: an immunologic functional perspective

Cryptococcosis in the era of AIDS--100 years after the discovery of Cryptococcus neoformans.

A macrophage protein, Ym1, transiently expressed during inflammation is a novel mammalian lectin.

Differential expression of FIZZ1 and Ym1 in alternatively versus classically activated macrophages.

Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosis

Alternative versus classical activation of macrophages.

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40894808

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20056835

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2808084

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