Inhibition of glycogen synthase kinase 3beta (GSK3beta) decreases inflammatory responses in brain endothelial cells - Wikidata - wikimedia - TriplyDB

Inhibition of glycogen synthase kinase 3beta (GSK3beta) decreases inflammatory responses in brain endothelial cells

Inhibition of glycogen synthase kinase 3beta (GSK3beta) decreases inflammatory responses in brain endothelial cells

http://www.wikidata.org/entity/Q33597201

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miRNAs Participate in MS Pathological Processes and Its Therapeutic Response Poly(ADP-ribose) polymerase-1 inhibition in brain endothelium protects the blood-brain barrier under physiologic and neuroinflammatory conditions.Gsk3 Signalling and Redox Status in Bipolar Disorder: Evidence from Lithium Efficacy Modulation of GSK-3β activity in Venezuelan equine encephalitis virus infection Glycogen synthase kinase-3β inhibition in the medial prefrontal cortex mediates paradoxical amphetamine action in a mouse model of ADHD.Dynamic modulation of innate immune response by varying dosages of lipopolysaccharide (LPS) in human monocytic cells.P2X4 receptor regulates alcohol-induced responses in microglia Inhibition of glycogen synthase kinase 3β promotes tight junction stability in brain endothelial cells by half-life extension of occludin and claudin-5.GSK3β is increased in adipose tissue and skeletal muscle from women with gestational diabetes where it regulates the inflammatory response Anti-inflammatory effect of targeted delivery of SOD to endothelium: mechanism, synergism with NO donors and protective effects in vitro and in vivo.Craniula: A cranial window technique for prolonged imaging of brain surface vasculature with simultaneous adjacent intracerebral injection.Cultured cells of the blood-brain barrier from apolipoprotein B-100 transgenic mice: effects of oxidized low-density lipoprotein treatment.Activation of cannabinoid receptor 2 attenuates leukocyte-endothelial cell interactions and blood-brain barrier dysfunction under inflammatory conditions Protective Effects of Kaempferol against Myocardial Ischemia/Reperfusion Injury in Isolated Rat Heart via Antioxidant Activity and Inhibition of Glycogen Synthase Kinase-3β.Glycogen synthase kinase 3β inhibition prevents monocyte migration across brain endothelial cells via Rac1-GTPase suppression and down-regulation of active integrin conformation.miR-98 and let-7g* protect the blood-brain barrier under neuroinflammatory conditions Increased GSK-3β expression in DRG microglia in response to sciatic nerve crush.Potential involvement of glycogen synthase kinase (GSK)-3β in a rat model of multiple sclerosis: evidenced by lithium treatment Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease.Regulation by glycogen synthase kinase-3 of inflammation and T cells in CNS diseases ECIS technology reveals that monocytes isolated by CD14+ve selection mediate greater loss of BBB integrity than untouched monocytes, which occurs to a greater extent with IL-1β activated endothelium in comparison to TNFα.Activation of Cannabinoid Type Two Receptors (CB2) Diminish Inflammatory Responses in Macrophages and Brain Endothelium.Isoflurane Is More Deleterious to Developing Brain Than Desflurane: The Role of the Akt/GSK3β Signaling Pathway.Tumour necrosis factor-α-mediated disruption of cerebrovascular endothelial barrier integrity in vitro involves the production of proinflammatory interleukin-6.The deubiquitinating enzyme USP48 stabilizes TRAF2 and reduces E-cadherin-mediated adherens junctions.Role of glycogen synthase kinase 3 in ischemia-induced blood-brain barrier disruption in aged female rats.Electroacupuncture attenuates cerebral ischemia-reperfusion injury in diabetic mice through adiponectin receptor 1-mediated phosphorylation of GSK-3β.Proteomics landscape of radiation-induced cardiovascular disease: somewhere over the paradigm.Characterization of human fetal brain endothelial cells reveals barrier properties suitable for in vitro modeling of the BBB with syngenic co-cultures.Targeting glycogen synthase kinase-3β to prevent hyperoxia-induced lung injury in neonatal rats.Lower phosphorylated glycogen synthase kinase-3B levels in platelets of patients with schizophrenia: increment by olanzapine treatment.

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Inhibition of glycogen synthase kinase 3beta (GSK3beta) decreases inflammatory responses in brain endothelial cells

http://www.wikidata.org/entity/Q33597201

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Inhibition of glycogen synthas ...... ses in brain endothelial cells

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The American Journal of Pathology

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Inhibition of glycogen synthas ...... ses in brain endothelial cells

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Anil Papugani

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Holly Dykstra

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Ming Zhang

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Nancy Reichenbach

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Ronald F Tuma

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Servio H Ramirez

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Shongshan Fan

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Yuri Persidsky

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P2860

Glycogen synthase kinase 3: more than a namesake

Glycogen synthase kinase-3 (GSK3): inflammation, diseases, and therapeutics

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

The blood-brain barrier in health and chronic neurodegenerative disorders

GSK3 inhibitors show benefits in an Alzheimer's disease (AD) model of neurodegeneration but adverse effects in control animals

Mononuclear phagocyte immunity and the neuropathogenesis of HIV-1 infection.

Altered vascular permeability and early onset of experimental autoimmune encephalomyelitis in PECAM-1-deficient mice.

Postinsult treatment with lithium reduces brain damage and facilitates neurological recovery in a rat ischemia/reperfusion model.

Glycogen synthase kinase 3: a drug target for CNS therapies.

Rho-mediated regulation of tight junctions during monocyte migration across the blood-brain barrier in HIV-1 encephalitis (HIVE)

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