Phospholipase cbeta4 is specifically involved in climbing fiber synapse elimination in the developing cerebellum.
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Synapse elimination in the developing cerebellumPhosphoinositides: tiny lipids with giant impact on cell regulationmGluR1/TRPC3-mediated Synaptic Transmission and Calcium Signaling in Mammalian Central Neurons.LTD-like molecular pathways in developmental synaptic pruningDisturbance of cerebellar synaptic maturation in mutant mice lacking BSRPs, a novel brain-specific receptor-like protein familyA human homeotic transformation resulting from mutations in PLCB4 and GNAI3 causes auriculocondylar syndrome.Signaling and cross-talk by C5a and UDP in macrophages selectively use PLCbeta3 to regulate intracellular free calcium.Cerebellar plasticity and motor learning deficits in a copy-number variation mouse model of autism.Formation of hippocampal synapses on patterned substrates of a laminin-derived synthetic peptide.A positive feedback loop linking enhanced mGluR function and basal calcium in spinocerebellar ataxia type 2Clostridium difficile toxin B differentially affects GPCR-stimulated Ca2+ responses in macrophages: independent roles for Rho and PLA2.Oligodendrocytes as regulators of neuronal networks during early postnatal development.In vivo functions of heterotrimeric G-proteins: studies in Galpha-deficient mice.Postsynaptic P/Q-type Ca2+ channel in Purkinje cell mediates synaptic competition and elimination in developing cerebellumA chemokine receptor CXCR2 macromolecular complex regulates neutrophil functions in inflammatory diseasesRequirement of TrkB for synapse elimination in developing cerebellar Purkinje cellsβ-III spectrin is critical for development of purkinje cell dendritic tree and spine morphogenesis.Architecture and development of olivocerebellar circuit topography.Disruption of cerebellar microzonal organization in GluD2 (GluRδ2) knockout mouse.Type-1 metabotropic glutamate receptor in cerebellar Purkinje cells: a key molecule responsible for long-term depression, endocannabinoid signalling and synapse eliminationEndocannabinoid-mediated control of synaptic transmission.Glutamate-receptor-like molecule GluRδ2 involved in synapse formation at parallel fiber-Purkinje neuron synapses.Development of an anatomical technique for visualizing the mode of climbing fiber innervation in Purkinje cells and its application to mutant mice lacking GluRδ2 and Ca(v)2.1.Climbing fiber synapse elimination in cerebellar Purkinje cells.Phosphoinositide pathway and the signal transduction network in neural developmentGq protein alpha subunits Galphaq and Galpha11 are localized at postsynaptic extra-junctional membrane of cerebellar Purkinje cells and hippocampal pyramidal cells.Presynaptic Membrane Receptors Modulate ACh Release, Axonal Competition and Synapse Elimination during Neuromuscular Junction Development.Cellular and circuit mechanisms underlying spinocerebellar ataxias.Type-1 metabotropic glutamate receptor signaling in cerebellar Purkinje cells in health and disease.Retrograde BDNF to TrkB signaling promotes synapse elimination in the developing cerebellum.Arc/Arg3.1 is a postsynaptic mediator of activity-dependent synapse elimination in the developing cerebellum.Effects of FAK ablation on cerebellar foliation, Bergmann glia positioning and climbing fiber territory on Purkinje cells.Phospholipase C beta3 deficiency leads to macrophage hypersensitivity to apoptotic induction and reduction of atherosclerosis in mice.Deficient long-term synaptic depression in the rostral cerebellum correlated with impaired motor learning in phospholipase C beta4 mutant mice.Close correlation between the birth date of Purkinje cells and the longitudinal compartmentalization of the mouse adult cerebellum.Sodium imaging of climbing fiber innervation fields in developing mouse Purkinje cells.Postsynaptic GABAB receptor signalling enhances LTD in mouse cerebellar Purkinje cells.Serotonergic control of Purkinje cell maturation and climbing fibre elimination by 5-HT3 receptors in the juvenile mouse cerebellum.Impaired presynaptic function and elimination of synapses at premature stages during postnatal development of the cerebellum in the absence of CALEB (CSPG5/neuroglycan C).Maturation, Refinement, and Serotonergic Modulation of Cerebellar Cortical Circuits in Normal Development and in Murine Models of Autism.
P2860
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P2860
Phospholipase cbeta4 is specifically involved in climbing fiber synapse elimination in the developing cerebellum.
description
1998 nî lūn-bûn
@nan
1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@ast
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@en
type
label
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@ast
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@en
prefLabel
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@ast
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@en
P2093
P2860
P356
P1476
Phospholipase cbeta4 is specif ...... in the developing cerebellum.
@en
P2093
Hashimoto K
Kurihara H
Offermanns S
Watanabe M
P2860
P304
15724-15729
P356
10.1073/PNAS.95.26.15724
P407
P577
1998-12-01T00:00:00Z