Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
about
The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?The Antigastric Cancer Activity of San Leng Powder Extract Induces Apoptosis in Balb/C Bearing-SGC-7901 Mice.Elevated cleaved caspase-3 is associated with shortened overall survival in several cancer types.DNA Damage Response Proteins and Oxygen Modulate Prostaglandin E2 Growth Factor Release in Response to Low and High LET Ionizing Radiation.The Role of Cathelicidin LL-37 in Cancer Development.Caspase 3 in dying tumor cells mediates post-irradiation angiogenesis.A novel prognostic biomarker SPC24 up-regulated in hepatocellular carcinoma.Extracellular Reactive Oxygen Species Drive Apoptosis-Induced Proliferation via Drosophila Macrophages.Antitumor effect of sunitinib in human prostate cancer cells functions via autophagy.The Sound of Silence: Signaling by Apoptotic CellsEmerging targets for combination therapy in melanomas.CRLX101, a Nanoparticle-Drug Conjugate Containing Camptothecin, Improves Rectal Cancer Chemoradiotherapy by Inhibiting DNA Repair and HIF1α.Killers creating new life: caspases drive apoptosis-induced proliferation in tissue repair and disease.Microenvironmental Effects of Cell Death in Malignant Disease.Apoptotic Caspases in Promoting Cancer: Implications from Their Roles in Development and Tissue Homeostasis.Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy Outcome.A molecular signature for anastasis, recovery from the brink of apoptotic cell death.Tumor-promoting function of apoptotic caspases by an amplification loop involving ROS, macrophages and JNK in Drosophila.Resolvins suppress tumor growth and enhance cancer therapy.Berberine inhibits the chemotherapy-induced repopulation by suppressing the arachidonic acid metabolic pathway and phosphorylation of FAK in ovarian cancer.Grhl3 modulates epithelial structure formation of the circumvallate papilla during mouse development.Platelet-Activating Factor Receptor Ligands Protect Tumor Cells from Radiation-Induced Cell Death.Exploitation of Apoptotic Regulation in Cancer.Radiotherapy-induced cell death activates paracrine HMGB1-TLR2 signaling and accelerates pancreatic carcinoma metastasis.Radiation-Induced RhoGDIβ Cleavage Leads to Perturbation of Cell Polarity: A Possible Link to Cancer Spreading.HMGB1 released by irradiated tumor cells promotes living tumor cell proliferation via paracrine effect.Caspase-independent cell death does not elicit a proliferative response in melanoma cancer cells.Repeated exposure of epithelial cells to apoptotic cells induces the specific selection of an adaptive phenotype: Implications for tumorigenesis
P2860
Q26749064-3D88FB19-2FE0-4B45-80C7-470B6808799DQ33711200-E413E046-AA6F-4EBB-AA54-CF8442290953Q34121507-24872782-0174-40A0-BD89-9B2C2C3B89F0Q36349712-BABED3D8-A6D8-4EBC-B941-6582A06A755AQ36465085-897DA8D9-9B17-4C88-A216-C0B476C2558DQ36544842-AB0B8567-EDCA-429E-B169-736F3FCA6183Q36562391-55ACB84E-3F58-4F38-95C7-FF9389613866Q36614093-773D543A-B425-41BE-88D2-0D4A42FEF414Q37734570-1E07FB2C-39BB-4590-B4C2-230FA6DDDFA5Q38393445-538D9600-1051-4D5B-B179-45A09D58F981Q38602788-A1A940D5-F581-4E69-AF88-CA7AB665CC20Q38736426-518945D3-44E2-4A6C-B1F5-1CD29E993E94Q38864895-088E87CD-1B70-4C2C-A98B-2B5B4C7EDFBEQ38936695-0C43DF62-3C4B-45B6-BA2B-862F05DA253AQ38936698-728D2461-1BDB-4C67-8BC9-818BDCCFF6A5Q39269601-78898F72-7B32-4C30-9238-09491788E39BQ45952373-6AA959A1-F2F6-4289-9039-3E7814C4A410Q46314637-8F6AEF7F-1E44-4876-B63A-971D287BC954Q47225724-ACC5360D-8B4C-4518-B6F7-CA83DEC9FAD4Q47679295-199F939F-5278-499D-95B4-6FCD8A2BA391Q48373324-F12BD309-6430-48B9-B1F7-70AD6403F094Q49829026-12F8825E-48F6-48D2-B523-725A04A0C245Q52356831-C6B6033B-D1C2-4868-BA18-707F6898AD8CQ52609417-6B0E3704-362E-4AB2-A17B-694F2FBEC16FQ52882972-F4998FC4-733A-4E18-B082-A22B833A3BB2Q55343196-5ED0B2BA-B0B5-48F5-816A-E7CC6A69F42EQ55511537-CA66E900-FDEE-4E11-B3EC-FDE6899FDA4CQ56889940-7EDC5ADC-0AC7-408B-8AE9-E3815574E41B
P2860
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@ast
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@en
type
label
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@ast
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@en
prefLabel
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@ast
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@en
P2093
P2860
P356
P1476
Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.
@en
P2093
Anne L Donato
Chuan-Yuan Li
Mary A Zimmerman
Qian Huang
Xinjian Liu
P2860
P2888
P304
P356
10.1038/JID.2014.18
P407
P577
2014-01-16T00:00:00Z
P5875
P6179
1014099519