Differences in the kinetics of gamma-H2AX fluorescence decay after exposure to low and high LET radiation. - Wikidata - wikimedia - TriplyDB

Differences in the kinetics of gamma-H2AX fluorescence decay after exposure to low and high LET radiation.

Differences in the kinetics of gamma-H2AX fluorescence decay after exposure to low and high LET radiation.

http://www.wikidata.org/entity/Q33612780

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Comments on potential health effects of MRI-induced DNA lesions: quality is more important to consider than quantity Establishment of a γ-H2AX foci-based assay to determine biological dose of radon to red bone marrow in rats pATM and γH2AX are effective radiation biomarkers in assessing the radiosensitivity of 12C6+ in human tumor cells.The RABiT: a rapid automated biodosimetry tool for radiological triage. II. Technological developments Induction and repair of DNA double-strand breaks assessed by gamma-H2AX foci after irradiation with pulsed or continuous proton beams.Endogenously induced DNA double strand breaks arise in heterochromatic DNA regions and require ataxia telangiectasia mutated and Artemis for their repair.Recruitment kinetics of DNA repair proteins Mdc1 and Rad52 but not 53BP1 depend on damage complexity.Glutathione depletion and carbon ion radiation potentiate clustered DNA lesions, cell death and prevent chromosomal changes in cancer cells progeny.Evaluation of the efficacy of radiation-modifying compounds using γH2AX as a molecular marker of DNA double-strand breaks Induction of H2AX phosphorylation in tumor cells by gossypol acetic acid is mediated by phosphatidylinositol 3-kinase (PI3K) family.Use of the γ-H2AX assay to investigate DNA repair dynamics following multiple radiation exposures.Comparison of RBE values of high-LET α-particles for the induction of DNA-DSBs, chromosome aberrations and cell reproductive death.Effects of targeted phosphorylation site mutations in the DNA-PKcs phosphorylation domain on low and high LET radiation sensitivity.Assessing 'radiosensitivity' with kinetic profiles of γ-H2AX, 53BP1 and BRCA1 foci.A new hand-held microfluidic cytometer for evaluating irradiation damage by analysis of the damaged cells distribution.Proteomic Analysis Implicates Dominant Alterations of RNA Metabolism and the Proteasome Pathway in the Cellular Response to Carbon-Ion Irradiation Enhancement of IUdR Radiosensitization by Low-Energy Photons Results from Increased and Persistent DNA Damage.Detection of γH2AX foci in mouse normal brain and brain tumor after boron neutron capture therapy.The potential value of the neutral comet assay and γH2AX foci assay in assessing the radiosensitivity of carbon beam in human tumor cell lines.High linear-energy-transfer radiation can overcome radioresistance of glioma stem-like cells to low linear-energy-transfer radiation.Bringing the heavy: carbon ion therapy in the radiobiological and clinical context.Repair of DNA damage induced by accelerated heavy ions--a mini review.Efficient Rejoining of DNA Double-Strand Breaks despite Increased Cell-Killing Effectiveness following Spread-Out Bragg Peak Carbon-Ion Irradiation.Understanding the potentiality of accelerator based-boron neutron capture therapy for osteosarcoma: dosimetry assessment based on the reported clinical experience.Differential gene expression in human fibroblasts after alpha-particle emitter (211)At compared with (60)Co irradiation.Multispectral imaging flow cytometry reveals distinct frequencies of γ-H2AX foci induction in DNA double strand break repair defective human cell lines.A Process of Resection-Dependent Nonhomologous End Joining Involving the Goddess Artemis.H2AX phosphorylation level in peripheral blood mononuclear cells as an event-free survival predictor for bladder cancer.Sulforaphane enhances irradiation effects in terms of perturbed cell cycle progression and increased DNA damage in pancreatic cancer cells.In-depth Characterization of a TCR-specific Tracer for Sensitive Detection of Tumor-directed Transgenic T Cells by Immuno-PET Differences in Phosphorylated Histone H2AX Foci Formation and Removal of Cells Exposed to Low and High Linear Energy Transfer Radiation.Gamma-H2AX foci in cells exposed to a mixed beam of X-rays and alpha particles.Inhibition of the HDAC/Suv39/G9a pathway restores the expression of DNA damage-dependent major histocompatibility complex class I-related chain A and B in cancer cells Increased cell survival and cytogenetic integrity by spatial dose redistribution at a compact synchrotron X-ray source.Radiation-Induced Cardiovascular Disease: Mechanisms and Importance of Linear Energy Transfer.Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry.Live Dynamics of 53BP1 Foci Following Simultaneous Induction of Clustered and Dispersed DNA Damage in U2OS Cells.

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P2860

Differences in the kinetics of gamma-H2AX fluorescence decay after exposure to low and high LET radiation.

http://www.wikidata.org/entity/Q33612780

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Differences in the kinetics of ...... to low and high LET radiation.

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Differences in the kinetics of ...... to low and high LET radiation.

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Differences in the kinetics of ...... to low and high LET radiation.

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Differences in the kinetics of ...... to low and high LET radiation.

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Differences in the kinetics of ...... to low and high LET radiation.

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Differences in the kinetics of ...... to low and high LET radiation.

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Anja Mittag

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Attila Tarnok

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Thomas E Schmid

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