IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis. - Wikidata - wikimedia - TriplyDB

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

http://www.wikidata.org/entity/Q33618905

about

Immunotherapy for the Adjunctive Treatment of Sepsis: From Immunosuppression to Immunostimulation. Time for a Paradigm Change?Innate immune programing by endotoxin and its pathological consequences Epigenetic regulation of immune cell functions during post-septic immunosuppression.Intrauterine inflammation, insufficient to induce parturition, still evokes fetal and neonatal brain injury Epigenetics, bioenergetics, and microRNA coordinate gene-specific reprogramming during acute systemic inflammation.Sepsis induces specific changes in histone modification patterns in human monocytes.Epigenetic regulation of tumor necrosis factor α (TNFα) release in human macrophages by HIV-1 single-stranded RNA (ssRNA) is dependent on TLR8 signaling.Study of Protein Phosphatase 2A (PP2A) Activity in LPS-Induced Tolerance Using Fluorescence-Based and Immunoprecipitation-Aided Methodology.Epigenetic Regulation of Tolerance to Toll-Like Receptor Ligands in Alveolar Epithelial Cells.Impaired innate mucosal immunity in aged mice permits prolonged Streptococcus pneumoniae colonization.PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis Genetic predisposition to respiratory infection and sepsis.When host defense goes awry: Modeling sepsis-induced immunosuppression.Role of dendritic cell-pathogen interactions in the immune response to pulmonary cryptococcal infection.Epigenetic Alterations Induced by Bacterial Lipopolysaccharides.Wear particles promote endotoxin tolerance in macrophages by inducing interleukin-1 receptor-associated kinase-M expression.Induction of endotoxin tolerance in vivo inhibits activation of IRAK4 and increases negative regulators IRAK-M, SHIP-1, and A20.The STAT4/MLL1 Epigenetic Axis Regulates the Antimicrobial Functions of Murine Macrophages.[Epigenetic regulation in sepsis : current state of knowledge].Microglial activation of the NLRP3 inflammasome by the priming signals derived from macrophages infected with mycobacteria.Association between gene polymorphisms of IRAK-M and the susceptibility of sepsis.The IL-33-PIN1-IRAK-M axis is critical for type 2 immunity in IL-33-induced allergic airway inflammation.Sepsis and Nosocomial Infection: Patient Characteristics, Mechanisms, and Modulation

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IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

http://www.wikidata.org/entity/Q33618905

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@ast

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@en

type

label

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@ast

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@en

prefLabel

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@ast

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@en

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JOURNAL.PONE.0011145

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P1476

IRAK-M regulates chromatin remodeling in lung macrophages during experimental sepsis.

@en

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Eric Rich

http://www.w3.org/2001/XMLSchema#string

Haitao Wen

http://www.w3.org/2001/XMLSchema#string

Kenneth Lyn-Kew

http://www.w3.org/2001/XMLSchema#string

Michael W Newstead

http://www.w3.org/2001/XMLSchema#string

Steven L Kunkel

http://www.w3.org/2001/XMLSchema#string

Theodore J Standiford

http://www.w3.org/2001/XMLSchema#string

Urvashi Bhan

http://www.w3.org/2001/XMLSchema#string

Xianying Zeng

http://www.w3.org/2001/XMLSchema#string

P2860

IRAK-M is a novel member of the Pelle/interleukin-1 receptor-associated kinase (IRAK) family

Identification and characterization of murine IRAK-M

IRAK-M is a negative regulator of Toll-like receptor signaling

Rapid up-regulation of IRAK-M expression following a second endotoxin challenge in human monocytes and in monocytes isolated from septic patients

Histone H3 lysine 4 methylation patterns in higher eukaryotic genes

Stability and flexibility of epigenetic gene regulation in mammalian development

The pathophysiology and treatment of sepsis

Cytokines and anticytokines in the pathogenesis of sepsis.

Epigenetics in human autoimmunity. Epigenetics in autoimmunity - DNA methylation in systemic lupus erythematosus and beyond.

Clinical aspects: from systemic inflammation to 'immunoparalysis'.

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e11145

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scholarly article

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10.1371/JOURNAL.PONE.0011145

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6

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2010-06-16T00:00:00Z

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44807083

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20585389

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