Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)). - Wikidata - wikimedia - TriplyDB

Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).

Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).

http://www.wikidata.org/entity/Q33619335

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Mitochondria, cholesterol and cancer cell metabolism The Permeability Transition in Plant Mitochondria: The Missing Link Calcium signaling as a mediator of cell energy demand and a trigger to cell death Glutathione and mitochondria Alterations in voltage-sensing of the mitochondrial permeability transition pore in ANT1-deficient cells.ACTH Action on StAR Biology Interdependence of tetrapyrrole metabolism, the generation of oxidative stress and the mitigative oxidative stress response The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection Cell death and autophagy in tuberculosis Peripheral benzodiazepine receptor/translocator protein global knock-out mice are viable with no effects on steroid hormone biosynthesis.Minireview: translocator protein (TSPO) and steroidogenesis: a reappraisal.Conditional steroidogenic cell-targeted deletion of TSPO unveils a crucial role in viability and hormone-dependent steroid formation.Translocator protein 18 kDa (TSPO): old dogma, new mice, new structure, and new questions for neuroprotection.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Global Deletion of TSPO Does Not Affect the Viability and Gene Expression Profile.Discovery, Synthesis, and Optimization of Diarylisoxazole-3-carboxamides as Potent Inhibitors of the Mitochondrial Permeability Transition Pore.Mitochondrial Translocator Protein (TSPO) Function Is Not Essential for Heme Biosynthesis Translocator Protein (TSPO) Affects Mitochondrial Fatty Acid Oxidation in Steroidogenic Cells.The hGFAP-driven conditional TSPO knockout is protective in a mouse model of multiple sclerosis.Functional gains in energy and cell metabolism after TSPO gene insertion.The role of hexokinase in cardioprotection - mechanism and potential for translation.The mitochondrial permeability transition pore and its adaptive responses in tumor cells.The mitochondrial translocator protein and arrhythmogenesis in ischemic heart disease Lost in translocation: the functions of the 18-kD translocator protein.Translocator protein (18 kDa): an update on its function in steroidogenesis.Mitochondrial involvement in myocyte death and heart failure.Shutting down the pore: The search for small molecule inhibitors of the mitochondrial permeability transition.Mitochondrial Ca2+ and regulation of the permeability transition pore.TSPO ligand residence time influences human glioblastoma multiforme cell death/life balance.The Mitochondrial Permeability Transition Pore and ATP Synthase.Translocator protein (TSPO) ligands for the diagnosis or treatment of neurodegenerative diseases: a patent review (2010-2015; part 1).Investigation of TSPO variants in schizophrenia and antipsychotic treatment outcomes.Mitochondrial permeability transition pore (MPTP) desensitization increases sea urchin spermatozoa fertilization rate.Methyl jasmonate leads to necrosis and apoptosis in hepatocellular carcinoma cells via inhibition of glycolysis and represses tumor growth in mice The Arabidopsis abiotic stress-induced TSPO-related protein reduces cell-surface expression of the aquaporin PIP2;7 through protein-protein interactions and autophagic degradation.Commentary: SPG7 is an essential and conserved component of the mitochondrial permeability transition pore CRISPR/CAS9-mediated Tspo gene mutations lead to reduced mitochondrial membrane potential and STEROID FORMation in MA-10 mouse tumor Leydig cells.Sifting through the surfeit of neuroinflammation tracers.Reconceptualization of translocator protein as a biomarker of neuroinflammation in psychiatry.

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Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).

http://www.wikidata.org/entity/Q33619335

description

2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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Regulation of the mitochondria ...... tor Protein of 18 kDa (TSPO)).

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JBC.M114.549634

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Journal of Biological Chemistry

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Regulation of the mitochondria ...... ator Protein of 18 kDa (TSPO))

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P2093

Andrea Carpi

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Elizabeth Blachly-Dyson

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Fernanda Ricchelli

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Michael Forte

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Randall Sewell

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Roberta Menabò

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P2860

Identification and validation of the mitochondrial F1F0-ATPase as the molecular target of the immunomodulatory benzodiazepine Bz-423

Cholesterol binding at the cholesterol recognition/ interaction amino acid consensus (CRAC) of the peripheral-type benzodiazepine receptor and inhibition of steroidogenesis by an HIV TAT-CRAC peptide

Isolation of the mitochondrial benzodiazepine receptor: association with the voltage-dependent anion channel and the adenine nucleotide carrier

Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice.

The molecular biology, biochemistry, and physiology of human steroidogenesis and its disorders

Structure of the yeast F1Fo-ATP synthase dimer and its role in shaping the mitochondrial cristae.

Peripheral-type benzodiazepine receptor: structure and function of a cholesterol-binding protein in steroid and bile acid biosynthesis

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

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13769-13781

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10.1074/JBC.M114.549634

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289

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Justina Sileikyte

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2014-04-01T00:00:00Z

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261294679

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