AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome. - Wikidata - wikimedia - TriplyDB

AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.

AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.

http://www.wikidata.org/entity/Q33640163

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Herpesvirus Late Gene Expression: A Viral-Specific Pre-initiation Complex Is Key Extracellular vesicles during Herpes Simplex Virus type 1 infection: an inquire Epstein-Barr virus and host cell methylation: regulation of latency, replication and virus reactivation Regulation of the latent-lytic switch in Epstein-Barr virus Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis The B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1 Micro RNAs of Epstein-Barr virus promote cell cycle progression and prevent apoptosis of primary human B cells.CpG-methylation regulates a class of Epstein-Barr virus promoters.Noise cancellation: viral fine tuning of the cellular environment for its own genome replication.Epstein-Barr virus (EBV) Rta-mediated EBV and Kaposi's sarcoma-associated herpesvirus lytic reactivations in 293 cells.Viral latency and its regulation: lessons from the gamma-herpesviruses.Epigenetic control of viral life-cycle by a DNA-methylation dependent transcription factor.Genome-wide DNA methylation as an epigenetic consequence of Epstein-Barr virus infection of immortalized keratinocytes The EBV immunoevasins vIL-10 and BNLF2a protect newly infected B cells from immune recognition and elimination Role for early-differentiated natural killer cells in infectious mononucleosis BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.A new model of Epstein-Barr virus infection reveals an important role for early lytic viral protein expression in the development of lymphomas.The effect of Epstein-Barr virus Latent Membrane Protein 2 expression on the kinetics of early B cell infection Sulfated small molecules targeting eBV in Burkitt lymphoma: from in silico screening to the evidence of in vitro effect on viral episomal DNA.An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cells Unexpected patterns of Epstein-Barr virus transcription revealed by a high throughput PCR array for absolute quantification of viral mRNA.Tegument protein control of latent herpesvirus establishment and animation.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latency Epstein-Barr virus Zta-induced immunomodulators from nasopharyngeal carcinoma cells upregulate interleukin-10 production from monocytes Epstein-Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression.DNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.The lytic phase of epstein-barr virus requires a viral genome with 5-methylcytosine residues in CpG sites.Genome-wide analysis of Epstein-Barr virus Rta DNA binding RNAs in Epstein-Barr virions control early steps of infection.An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.Epstein-Barr virus interactions with the Bcl-2 protein family and apoptosis in human tumor cells.Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replication.The B-cell-specific transcription factor and master regulator Pax5 promotes Epstein-Barr virus latency by negatively regulating the viral immediate early protein BZLF1.Epstein-Barr viral miRNAs inhibit antiviral CD4+ T cell responses targeting IL-12 and peptide processing.ERK/c-Jun Recruits Tet1 to Induce Zta Expression and Epstein-Barr Virus Reactivation through DNA Demethylation B lymphocytes from patients with a hypomorphic mutation in STAT3 resist Epstein-Barr virus-driven cell proliferation c-Jun/c-Fos heterodimers regulate cellular genes via a newly identified class of methylated DNA sequence motifs.Epstein-Barr virus genetics: talking about the BAC generation.How to control an infectious bead string: nucleosome-based regulation and targeting of herpesvirus chromatin.

P2860

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P2860

AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.

http://www.wikidata.org/entity/Q33640163

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

@ast

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

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type

label

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

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AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

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prefLabel

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

@ast

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

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P1433

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P2860

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P356

PNAS.0911948107

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

AP-1 homolog BZLF1 of Epstein- ...... tic state of the viral genome.

@en

P2093

Anne Schmeinck

http://www.w3.org/2001/XMLSchema#string

Dagmar Pich

http://www.w3.org/2001/XMLSchema#string

Markus Kalla

http://www.w3.org/2001/XMLSchema#string

Martin Bergbauer

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Wolfgang Hammerschmidt

http://www.w3.org/2001/XMLSchema#string

P2860

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

A versatile nanotrap for biochemical and functional studies with fluorescent fusion proteins

Epigenotypes of latent herpesvirus genomes

ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus

Methylated DNA recognition during the reversal of epigenetic silencing is regulated by cysteine and serine residues in the Epstein-Barr virus lytic switch protein

Early events associated with infection of Epstein-Barr virus infection of primary B-cells

Epigenotypes of Latent Herpesvirus Genomes

Epstein-Barr virus: 40 years on

Chromosome-wide and promoter-specific analyses identify sites of differential DNA methylation in normal and transformed human cells

Immediate early and early lytic cycle proteins are frequent targets of the Epstein-Barr virus-induced cytotoxic T cell response

P304

850-855

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scholarly article

P356

10.1073/PNAS.0911948107

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P407

P433

2

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P478

107

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P577

2009-12-22T00:00:00Z

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41040431

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P698

20080764

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P921

Epstein–Barr virus

P932

2818922

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