Adaptive mutation sequences reproduced by mismatch repair deficiency.
about
Atypical Role for PhoU in Mutagenic Break Repair under Stress in Escherichia coliMechanisms of stationary phase mutation: a decade of adaptive mutationGenome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutationThe SOS response regulates adaptive mutation.Transient and heritable mutators in adaptive evolution in the lab and in nature.Evidence that stationary-phase hypermutation in the Escherichia coli chromosome is promoted by recombination.The roles of REV3 and RAD57 in double-strand-break-repair-induced mutagenesis of Saccharomyces cerevisiae.Adaptive mutation in Saccharomyces cerevisiae.Mismatch repair protein MutL becomes limiting during stationary-phase mutation.Mutation as a stress response and the regulation of evolvability5-Methylcytosine is not a mutation hot spot in nondividing Escherichia coli.Promoter-creating mutations in Pseudomonas putida: a model system for the study of mutation in starving bacteriaFrameshift intermediates in homopolymer runs are removed efficiently by yeast mismatch repair proteinsHypermutability of homonucleotide runs in mismatch repair and DNA polymerase proofreading yeast mutants.Stable DNA replication: interplay between DNA replication, homologous recombination, and transcriptionAdaptive mutation: general mutagenesis is not a programmed response to stress but results from rare coamplification of dinB with lac.Stress-induced mutagenesis in bacteria.Stationary-phase mutation in the bacterial chromosome: recombination protein and DNA polymerase IV dependence.The cellular ecology of progressive neoplastic transformation: a clonal analysis.DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli.Contribution of the mismatch DNA repair system to the generation of stationary-phase-induced mutants of Bacillus subtilis.DNA repair and mutations during quiescence in yeast.Depletion of the cellular amounts of the MutS and MutH methyl-directed mismatch repair proteins in stationary-phase Escherichia coli K-12 cells.Carbon starvation of Salmonella typhimurium does not cause a general increase of mutation rates.Stress-Induced Mutagenesis.Involvement of error-prone DNA polymerase IV in stationary-phase mutagenesis in Pseudomonas putida.An insertion sequence prepares Pseudomonas putida S12 for severe solvent stress.Human mismatch-repair protein MutL homologue 1 (MLH1) interacts with Escherichia coli MutL and MutS in vivo and in vitro: a simple genetic system to assay MLH1 function.The microsatellites of Escherichia coli: rapidly evolving repetitive DNAs in a non-pathogenic prokaryote.
P2860
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P2860
Adaptive mutation sequences reproduced by mismatch repair deficiency.
description
1995 nî lūn-bûn
@nan
1995 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@ast
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@en
type
label
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@ast
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@en
prefLabel
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@ast
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@en
P2093
P2860
P356
P1476
Adaptive mutation sequences reproduced by mismatch repair deficiency.
@en
P2093
Galloway AM
Longerich S
Rosenberg SM
P2860
P304
12017-12020
P356
10.1073/PNAS.92.26.12017
P407
P577
1995-12-01T00:00:00Z