Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
about
Critical role of perforin-dependent CD8+ T cell immunity for rapid protective vaccination in a murine model for human smallpoxLack of both Fas ligand and perforin protects from flavivirus-mediated encephalitis in mice.The immunoproteasome and viral infection: a complex regulator of inflammationThe effector T cell response to influenza infectionA role for NKG2D in NK cell-mediated resistance to poxvirus disease.Surviving mousepox infection requires the complement systemCaspase-dependent inhibition of mousepox replication by gzmB.CD8 T cells use IFN-γ to protect against the lethal effects of a respiratory poxvirus infection.Protection against polyoma virus-induced tumors is perforin-independentCreation of killer poxvirus could have been predictedConcerted action of the FasL/Fas and perforin/granzyme A and B pathways is mandatory for the development of early viral hepatitis but not for recovery from viral infection.Obligatory requirement for antibody in recovery from a primary poxvirus infectionPolarized type 1 cytokine response and cell-mediated immunity determine genetic resistance to mousepox.The ectromelia virus SPI-2 protein causes lethal mousepox by preventing NK cell responsesInduction of natural killer cell responses by ectromelia virus controls infection.Residual active granzyme B in cathepsin C-null lymphocytes is sufficient for perforin-dependent target cell apoptosisComparable polyfunctionality of ectromelia virus- and vaccinia virus-specific murine T cells despite markedly different in vivo replication and pathogenicityT cells cause acute immunopathology and are required for long-term survival in mouse adenovirus type 1-induced encephalomyelitisGranzymes are the essential downstream effector molecules for the control of primary virus infections by cytolytic leukocytes.The cytolytic enzymes granyzme A, granzyme B, and perforin: expression patterns, cell distribution, and their relationship to cell maturity and bright CD57 expression.Fas and TNFR1, but not cytolytic granule-dependent mechanisms, mediate clearance of murine liver adenoviral infectionT cell receptor sharing by cytotoxic T lymphocytes facilitates efficient virus control.Granzymes and caspase 3 play important roles in control of gammaherpesvirus latencyContributions of CD8 T cells to the pathogenesis of mouse adenovirus type 1 respiratory infection.The level of friend retrovirus replication determines the cytolytic pathway of CD8+ T-cell-mediated pathogen control.Perforin-deficient CD8+ T cells mediate fatal lymphocytic choriomeningitis despite impaired cytokine production.
P2860
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P2860
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
description
1999 nî lūn-bûn
@nan
1999 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@ast
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@en
type
label
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@ast
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@en
prefLabel
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@ast
Perforin is essential for control of ectromelia virus but not related poxviruses in mice.
@en
P2093
P2860
P1433
P1476
Perforin is essential for control of ectromelia virus but not related poxviruses in mice
@en
P2093
P2860
P304
P577
1999-02-01T00:00:00Z