TCDD and a putative endogenous AhR ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts.
about
Retinoid-xenobiotic interactions: the Ying and the YangToward understanding the role of aryl hydrocarbon receptor in the immune system: current progress and future trendsThe Aryl Hydrocarbon Receptor: A Key Bridging Molecule of External and Internal Chemical SignalsNrf2 protects against 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced oxidative injury and steatohepatitisDioxin and estrogen signaling in lung adenocarcinoma cells with different aryl hydrocarbon receptor/estrogen receptor α phenotypes.Comparisons of differential gene expression elicited by TCDD, PCB126, βNF, or ICZ in mouse hepatoma Hepa1c1c7 cells and C57BL/6 mouse liver.Intersection of AHR and Wnt signaling in development, health, and disease.The aryl hydrocarbon receptor ligand ITE inhibits TGFβ1-induced human myofibroblast differentiationAryl hydrocarbon receptor-dependent retention of nuclear HuR suppresses cigarette smoke-induced cyclooxygenase-2 expression independent of DNA-binding.Aryl hydrocarbon receptor activation in hematopoietic stem/progenitor cells alters cell function and pathway-specific gene modulation reflecting changes in cellular trafficking and migration.Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infection.ITE and TCDD differentially regulate the vascular remodeling of rat placenta via the activation of AhR.Differential consequences of two distinct AhR ligands on innate and adaptive immune responses to influenza A virus.Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis-persistent pollutants affect programmed cell death.Differential Contribution of the Aryl-Hydrocarbon Receptor and Toll-Like Receptor Pathways to IL-8 Expression in Normal and Cystic Fibrosis Airway Epithelial Cells Exposed to Pseudomonas aeruginosa.Global gene expression changes in human embryonic lung fibroblasts induced by organic extracts from respirable air particles.Induction of a chloracne phenotype in an epidermal equivalent model by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is dependent on aryl hydrocarbon receptor activation and is not reproduced by aryl hydrocarbon receptor knock down.Aryl hydrocarbon receptor signaling modulates antiviral immune responses: ligand metabolism rather than chemical source is the stronger predictor of outcome.
P2860
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P2860
TCDD and a putative endogenous AhR ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@ast
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@en
type
label
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@ast
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@en
prefLabel
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@ast
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@en
P2093
P2860
P356
P1476
TCDD and a putative endogenous ...... ion in mouse lung fibroblasts.
@en
P2093
Ellen C Henry
Stephen L Welle
Thomas A Gasiewicz
P2860
P304
P356
10.1093/TOXSCI/KFP285
P577
2009-11-19T00:00:00Z