Disruption of Nrf2 impairs the resolution of hyperoxia-induced acute lung injury and inflammation in mice - Wikidata - wikimedia - TriplyDB

Disruption of Nrf2 impairs the resolution of hyperoxia-induced acute lung injury and inflammation in mice

Disruption of Nrf2 impairs the resolution of hyperoxia-induced acute lung injury and inflammation in mice

http://www.wikidata.org/entity/Q33646250

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Suppression of basal and carbon nanotube-induced oxidative stress, inflammation and fibrosis in mouse lungs by Nrf2 Nanomedicine delivers promising treatments for rheumatoid arthritis Roles nrf2 plays in myeloid cells and related disorders Biliverdin reductase isozymes in metabolism Src subfamily kinases regulate nuclear export and degradation of transcription factor Nrf2 to switch off Nrf2-mediated antioxidant activation of cytoprotective gene expression Reactive oxygen species in inflammation and tissue injury Th2 skewing by activation of Nrf2 in CD4(+) T cells Cardioprotective effect of paeonol and danshensu combination on isoproterenol-induced myocardial injury in rats Nrf2:INrf2 (Keap1) signaling in oxidative stress Glutathione oxidation is associated with airway macrophage functional impairment in children with severe asthma Growth and stress response mechanisms underlying post-feeding regenerative organ growth in the Burmese python.Innate immunity against bacterial infection following hyperoxia exposure is impaired in NRF2-deficient mice.A computable cellular stress network model for non-diseased pulmonary and cardiovascular tissue.Heme oxygenase in neonatal lung injury and repair Dual oxidase 2 in lung epithelia is essential for hyperoxia-induced acute lung injury in mice.Association of Nrf2 polymorphism haplotypes with acute lung injury phenotypes in inbred strains of mice.Phagocyte NADPH oxidase restrains the inflammasome in ANCA-induced GN Dual roles of NRF2 in tumor prevention and progression: possible implications in cancer treatment PI3K-AKT Signaling via Nrf2 Protects against Hyperoxia-Induced Acute Lung Injury, but Promotes Inflammation Post-Injury Independent of Nrf2 in Mice Conditional deletion of Nrf2 in airway epithelium exacerbates acute lung injury and impairs the resolution of inflammation.Nrf2 reduces allergic asthma in mice through enhanced airway epithelial cytoprotective function Coenzyme Q(1) as a probe for mitochondrial complex I activity in the intact perfused hyperoxia-exposed wild-type and Nqo1-null mouse lung.Enhanced Resolution of Hyperoxic Acute Lung Injury as a result of Aspirin Triggered Resolvin D1 Treatment.Targeted deletion of nrf2 impairs lung development and oxidant injury in neonatal mice NF-κB and Nrf2 signaling pathways contribute to wogonin-mediated inhibition of inflammation-associated colorectal carcinogenesis Thioredoxin reductase inhibition elicits Nrf2-mediated responses in Clara cells: implications for oxidant-induced lung injury Nrf2 regulates hyperoxia-induced Nox4 expression in human lung endothelium: identification of functional antioxidant response elements on the Nox4 promoter.Synthetic oleanane triterpenoids: multifunctional drugs with a broad range of applications for prevention and treatment of chronic disease.Deletion of P2X7 attenuates hyperoxia-induced acute lung injury via inflammasome suppression Abrogation of Nrf2 impairs antioxidant signaling and promotes atrial hypertrophy in response to high-intensity exercise stress.Hydrogen gas reduces hyperoxic lung injury via the Nrf2 pathway in vivo Anthocyanins protect human endothelial cells from mild hyperoxia damage through modulation of Nrf2 pathway.Role of Nrf2 and Autophagy in Acute Lung Injury NLRP3 deletion protects from hyperoxia-induced acute lung injury Nrf2 promotes alveolar mitochondrial biogenesis and resolution of lung injury in Staphylococcus aureus pneumonia in mice.Sirtuin 1 Promotes Hyperoxia-Induced Lung Epithelial Cell Death Independent of NF-E2-Related Factor 2 Activation.Activating the Nrf2-mediated antioxidant response element restores barrier function in the alveolar epithelium of HIV-1 transgenic rats.Acute exercise stress activates Nrf2/ARE signaling and promotes antioxidant mechanisms in the myocardium.Potential effects of medicinal plants and secondary metabolites on acute lung injury.The triterpenoid CDDO-imidazolide confers potent protection against hyperoxic acute lung injury in mice.

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P2860

Disruption of Nrf2 impairs the resolution of hyperoxia-induced acute lung injury and inflammation in mice

http://www.wikidata.org/entity/Q33646250

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2009 nî lūn-bûn

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2009年の論文

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JIMMUNOL.0804248

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Journal of Immunology

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Disruption of Nrf2 impairs the ...... njury and inflammation in mice

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Masayuki Yamamoto

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Narsa M Reddy

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Paul M Hassoun

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Sekhar P Reddy

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Steven R Kleeberger

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Thomas W Kensler

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P2860

Normal remodeling of the oxygen-injured lung requires the cyclin-dependent kinase inhibitor p21(Cip1/WAF1/Sdi1)

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

Resolution of inflammation: the beginning programs the end

Regulatory mechanisms controlling gene expression mediated by the antioxidant response element

Oxidants/antioxidants and chronic obstructive pulmonary disease: pathogenesis to therapy.

DNA damage and cell cycle checkpoints in hyperoxic lung injury: braking to facilitate repair.

Secretory products of macrophages.

Antioxidant responses to oxidant-mediated lung diseases.

Cytokine-mediated inflammation in acute lung injury.

Nrf2 defends the lung from oxidative stress.

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