Dual beneficial effects of (-)-epigallocatechin-3-gallate on levodopa methylation and hippocampal neurodegeneration: in vitro and in vivo studies. - Wikidata - wikimedia - TriplyDB

Dual beneficial effects of (-)-epigallocatechin-3-gallate on levodopa methylation and hippocampal neurodegeneration: in vitro and in vivo studies.

Dual beneficial effects of (-)-epigallocatechin-3-gallate on levodopa methylation and hippocampal neurodegeneration: in vitro and in vivo studies.

http://www.wikidata.org/entity/Q33654222

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Bioactive Compounds and Their Neuroprotective Effects in Diabetic Complications Potential neuroprotective properties of epigallocatechin-3-gallate (EGCG)Kainic Acid-Induced Excitotoxicity Experimental Model: Protective Merits of Natural Products and Plant Extracts Neuroprotective effect of combination therapy of glatiramer acetate and epigallocatechin-3-gallate in neuroinflammation.Catechins protect neurons against mitochondrial toxins and HIV proteins via activation of the BDNF pathway.Mitigation of H(2)O(2)-Induced Mitochondrial-Mediated Apoptosis in NG108-15 Cells by Novel Mesuagenin C from Mesua kunstleri (King) Kosterm.Can Tea Consumption be a Safe and Effective Therapy Against Diabetes Mellitus-Induced Neurodegeneration?Epigallocatechin-3-gallate: a useful, effective and safe clinical approach for targeted prevention and individualised treatment of neurological diseases?Beneficial effects of natural phenolics on levodopa methylation and oxidative neurodegeneration.Dietary polyphenol-derived protection against neurotoxic β-amyloid protein: from molecular to clinical.Mitochondria and neurodegenerative diseases: the promising role of nanotechnology in targeted drug delivery.Chronic Oral Epigallocatechin-gallate Alleviates Streptozotocin-induced Diabetic Neuropathic Hyperalgesia in Rat: Involvement of Oxidative Stress.Reduction in Autophagy by (-)-Epigallocatechin-3-Gallate (EGCG): a Potential Mechanism of Prevention of Mitochondrial Dysfunction After Subarachnoid Hemorrhage.Comparative effect of Camellia sinensis teas on object recognition test deficit and metabolic changes induced by cafeteria diet.Adult-onset brain tumors and neurodegeneration: Are polyphenols protective?A single dose of pirfenidone attenuates neuronal loss and reduces lipid peroxidation after kainic acid-induced excitotoxicity in the pubescent rat hippocampus.Signaled drug delivery and transport across the blood-brain barrier.EGCG ameliorates the suppression of long-term potentiation induced by ischemia at the Schaffer collateral-CA1 synapse in the rat.Salvianolic acid B as a substrate and weak catechol-O-methyltransferase inhibitor in rats

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Dual beneficial effects of (-)-epigallocatechin-3-gallate on levodopa methylation and hippocampal neurodegeneration: in vitro and in vivo studies.

http://www.wikidata.org/entity/Q33654222

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2010 nî lūn-bûn

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2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2010年論文

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2010年論文

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Bao Ting Zhu

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Ki Sung Kang

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Masayuki Fukui

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Noriko Yamabe

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Stephanie C Bishop

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Yujing Wen

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P2860

Pharmacokinetics and safety of green tea polyphenols after multiple-dose administration of epigallocatechin gallate and polyphenon E in healthy individuals

The significance of 3-O-methyldopa concentrations in the cerebrospinal fluid in the pathogenesis of wearing-off phenomenon in Parkinson's disease

Plasma O-methyldopa in levodopa-induced dyskinesias. A bioclinical investigation

Inhibition of poly (ADP-ribose) synthetase by gene disruption or inhibition with 5-iodo-6-amino-1,2-benzopyrone protects mice from multiple-low-dose-streptozotocin-induced diabetes

Phase I pharmacokinetic study of tea polyphenols following single-dose administration of epigallocatechin gallate and polyphenon E.

Catechol-O-methyltransferase (COMT): biochemistry, molecular biology, pharmacology, and clinical efficacy of the new selective COMT inhibitors.

Fluoro-Jade B: a high affinity fluorescent marker for the localization of neuronal degeneration.

Catechin content of 18 teas and a green tea extract supplement correlates with the antioxidant capacity.

Issues important for rational COMT inhibition.

The place of COMT inhibitors in the armamentarium of drugs for the treatment of Parkinson's disease.

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