Postnatal treatment using curcumin supplements to amend the damage in VPA-induced rodent models of autism - Wikidata - wikimedia - TriplyDB

Postnatal treatment using curcumin supplements to amend the damage in VPA-induced rodent models of autism

Postnatal treatment using curcumin supplements to amend the damage in VPA-induced rodent models of autism

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Wnt/β-catenin signaling stimulates the expression and synaptic clustering of the autism-associated Neuroligin 3 gene.Decreased Number and Expression of nNOS-Positive Interneurons in Basolateral Amygdala in Two Mouse Models of Autism

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Postnatal treatment using curcumin supplements to amend the damage in VPA-induced rodent models of autism

http://www.wikidata.org/entity/Q33654915

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2017 nî lūn-bûn

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2017 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2017 թվականի մայիսին հրատարակված գիտական հոդված

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2017年の論文

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2017年学术文章

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2017年学术文章

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name

Postnatal treatment using curc ...... nduced rodent models of autism

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Postnatal treatment using curc ...... nduced rodent models of autism

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type

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Postnatal treatment using curc ...... nduced rodent models of autism

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Postnatal treatment using curc ...... nduced rodent models of autism

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Afaf El-Ansary

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Laila Al-Ayadhi

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Maha Al-Askar

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Manar Selim

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Ramesa Shafi Bhat

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P2860

In utero exposure to valproic acid and autism--a current review of clinical and animal studies

Evaluation of animal models of neurobehavioral disorders.

Foetal antiepileptic drug exposure and verbal versus non-verbal abilities at three years of age.

Valproic acid pathway: pharmacokinetics and pharmacodynamics.

Prenatal valproate exposure and risk of autism spectrum disorders and childhood autism.

Curcumin: the story so far.

Potential therapeutic effects of curcumin, the anti-inflammatory agent, against neurodegenerative, cardiovascular, pulmonary, metabolic, autoimmune and neoplastic diseases

Curcumin: a potential neuroprotective agent in Parkinson's disease.

Curcumin protects against glutamate excitotoxicity in rat cerebral cortical neurons by increasing brain-derived neurotrophic factor level and activating TrkB.

A prospective study of cognitive fluency and originality in children exposed in utero to carbamazepine, lamotrigine, or valproate monotherapy.

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