Induction of IFN-alphabeta enables Listeria monocytogenes to suppress macrophage activation by IFN-gamma.
about
Leishmania RNA virus: when the host pays the tollNew insights into the role of RNase L in innate immunityInterferon response factors 3 and 7 protect against Chikungunya virus hemorrhagic fever and shockInnate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genomeDifferential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infectionsOverlapping, additive and counterregulatory effects of type II and I interferons on myeloid dendritic cell functionsOpposing roles for interferon regulatory factor-3 (IRF-3) and type I interferon signaling during plagueConditional Stat1 ablation reveals the importance of interferon signaling for immunity to Listeria monocytogenes infectionDisease-promoting effects of type I interferons in viral, bacterial, and coinfectionsType I interferon: friend or foe?Cyclic di-GMP-dependent signaling pathways in the pathogenic Firmicute Listeria monocytogenesA LysM and SH3-domain containing region of the Listeria monocytogenes p60 protein stimulates accessory cells to promote activation of host NK cells.Getting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial InfectionsDown regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infectionCellular reactive oxygen species inhibit MPYS induction of IFNβA fluorescence reporter model defines "Tip-DCs" as the cellular source of interferon β in murine listeriosisProduction of IFN-β during Listeria monocytogenes infection is restricted to monocyte/macrophage lineageA Listeria monocytogenes-based vaccine that secretes sand fly salivary protein LJM11 confers long-term protection against vector-transmitted Leishmania major.Activation of Ras overcomes B-cell tolerance to promote differentiation of autoreactive B cells and production of autoantibodiesMyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infectionType I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Antagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infectionsType I IFN induces IL-10 production in an IL-27-independent manner and blocks responsiveness to IFN-γ for production of IL-12 and bacterial killing in Mycobacterium tuberculosis-infected macrophages.Prevalence, distribution and functional significance of the -237C to T polymorphism in the IL-12Rβ2 promoter in Indian tuberculosis patients.Listeria monocytogenes induces IFNβ expression through an IFI16-, cGAS- and STING-dependent pathway.The timing of IFNβ production affects early innate responses to Listeria monocytogenes and determines the overall outcome of lethal infection.Bacterial programming of host responses: coordination between type I interferon and cell death.The Opportunistic Pathogen Listeria monocytogenes: Pathogenicity and Interaction with the Mucosal Immune System.A systems biological view of intracellular pathogensRoute of Infection Determines the Impact of Type I Interferons on Innate Immunity to Listeria monocytogenesType I interferons increase host susceptibility to Trypanosoma cruzi infection.The C5a anaphylatoxin receptor (C5aR1) protects against Listeria monocytogenes infection by inhibiting type 1 IFN expressionIdentification and characterization of a loss-of-function human MPYS variant.Cytokine and lipid mediator networks in tuberculosisMPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.Inflammatory monocytes recruited to the liver within 24 hours after virus-induced inflammation resemble Kupffer cells but are functionally distinct.Innate and adaptive interferons suppress IL-1α and IL-1β production by distinct pulmonary myeloid subsets during Mycobacterium tuberculosis infection.Type I interferon signaling regulates the composition of inflammatory infiltrates upon infection with Listeria monocytogenes.The Orphan Nuclear Receptor TLX Is an Enhancer of STAT1-Mediated Transcription and Immunity to Toxoplasma gondii.Hyperinduction of host beta interferon by a Listeria monocytogenes strain naturally overexpressing the multidrug efflux pump MdrT.
P2860
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P2860
Induction of IFN-alphabeta enables Listeria monocytogenes to suppress macrophage activation by IFN-gamma.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@ast
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@en
type
label
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@ast
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@en
prefLabel
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@ast
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@en
P2093
P2860
P356
P1476
Induction of IFN-alphabeta ena ...... phage activation by IFN-gamma.
@en
P2093
Jessica Humann
Karl Andreasen
Kristi Penheiter
Laurel L Lenz
Manira Rayamajhi
P2860
P304
P356
10.1084/JEM.20091746
P407
P577
2010-02-01T00:00:00Z