Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-gamma and MyD88 signaling. - Wikidata - wikimedia - TriplyDB

Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-gamma and MyD88 signaling.

Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-gamma and MyD88 signaling.

http://www.wikidata.org/entity/Q33660251

about

The Mitogen-Activated Protein Kinase (MAPK) Pathway: Role in Immune Evasion by Trypanosomatids Regulation of Dendritic Cell Function in Inflammation Estrogen receptors regulate an inflammatory pathway of dendritic cell differentiation: mechanisms and implications for immunity Dendritic cells in human Pneumovirus and Metapneumovirus infections The Regulation of CD4(+) T Cell Responses during Protozoan Infections Regulatory dendritic cells in the tumor immunoenvironment Trypanosoma brucei Co-opts NK Cells to Kill Splenic B2 B Cells MIF Promotes Classical Activation and Conversion of Inflammatory Ly6C(high) Monocytes into TipDCs during Murine Toxoplasmosis.Type I interferons promote fatal immunopathology by regulating inflammatory monocytes and neutrophils during Candida infections Monocyte recruitment during infection and inflammation Protective and pathogenic functions of macrophage subsets Interleukin-1 receptor antagonist modulates inflammation and scarring after ligament injury.MIF contributes to Trypanosoma brucei associated immunopathogenicity development Leishmania mexicana induces limited recruitment and activation of monocytes and monocyte-derived dendritic cells early during infection TNFα-mediated liver destruction by Kupffer cells and Ly6Chi monocytes during Entamoeba histolytica infection.Nitric oxide controls an inflammatory-like Ly6C(hi)PDCA1+ DC subset that regulates Th1 immune responses Interleukin expression after injury and the effects of interleukin-1 receptor antagonist A Trypanosoma brucei kinesin heavy chain promotes parasite growth by triggering host arginase activity.The role of macrophage IL-10/innate IFN interplay during virus-induced asthma.Development of a pHrodo-based assay for the assessment of in vitro and in vivo erythrophagocytosis during experimental trypanosomosis Monocyte trafficking in acute and chronic inflammation.Targeting curcusomes to inflammatory dendritic cells inhibits NF-κB and improves insulin resistance in obese mice Inflammatory monocytes recruited to the liver within 24 hours after virus-induced inflammation resemble Kupffer cells but are functionally distinct.Macrophage migration inhibitory factor (MIF): a key player in protozoan infections.Ly6C- Monocytes Regulate Parasite-Induced Liver Inflammation by Inducing the Differentiation of Pathogenic Ly6C+ Monocytes into Macrophages NK-, NKT- and CD8-Derived IFNγ Drives Myeloid Cell Activation and Erythrophagocytosis, Resulting in Trypanosomosis-Associated Acute Anemia Inflammatory monocyte effector mechanisms.IL-27 Signaling Is Crucial for Survival of Mice Infected with African Trypanosomes via Preventing Lethal Effects of CD4+ T Cells and IFN-γ.Distinct Contributions of CD4+ and CD8+ T Cells to Pathogenesis of Trypanosoma brucei Infection in the Context of Gamma Interferon and Interleukin-10 Type I and Type II Interferon Coordinately Regulate Suppressive Dendritic Cell Fate and Function during Viral Persistence Hepatic macrophage migration and differentiation critical for liver fibrosis is mediated by the chemokine receptor C-C motif chemokine receptor 8 in mice.Plasmacytoid dendritic cell depletion leads to an enhanced mononuclear phagocyte response in lungs of mice with lethal influenza virus infection Relationship among Short and Long Term of Hypoinsulinemia-Hyperglycemia, Dermatophytosis, and Immunobiology of Mononuclear Phagocytes.Recipient Myd88 Deficiency Promotes Spontaneous Resolution of Kidney Allograft Rejection Neurokinin-1 receptor agonists bias therapeutic dendritic cells to induce type 1 immunity by licensing host dendritic cells to produce IL-12 Inflammatory monocytes are potent antitumor effectors controlled by regulatory CD4+ T cells.The Trypanosoma brucei gambiense secretome impairs lipopolysaccharide-induced maturation, cytokine production, and allostimulatory capacity of dendritic cells mTORC1-Activated Monocytes Increase Tregs and Inhibit the Immune Response to Bacterial Infections.MAIT cells promote inflammatory monocyte differentiation into dendritic cells during pulmonary intracellular infection.Contribution of innate immune responses towards resistance to African trypanosome infections.

P2860

Q26768552-296B350C-5336-4519-872B-498E7C01E68C Q26800988-9918059E-CC53-4EBE-99D0-476B7C3A3CC0 Q26863531-482E5047-E26E-4DA6-9D78-1923791C630A Q26865337-33B7E0CA-8864-44BA-94D8-EADDB705A6CE Q26865518-9922233A-4A08-460D-8BDE-319F491D473B Q27023084-ABFF10E8-3B22-4245-BA8B-8E042FF2918B Q27312822-550D2D9D-34F1-47F6-9F55-E8CCF9093774 Q27348931-873DD568-9169-4662-A31D-926EECF96741 Q28481589-FD1262A3-94DE-4052-A55D-4CB7BFC0A9F0 Q29620067-97D5D649-8AEB-4DE1-9064-48AFCF5E41A5 Q29620351-6BFBDF71-725C-486D-90F7-3975433ABCB4 Q33903040-490F0DE8-FD7D-4B02-8CC0-D32815D160C6 Q34257852-D8DDB04A-273A-43D6-BC0E-F476C006BFFD Q34456673-745E13F3-EC27-4F69-B6AD-CD6434A77C2C Q34539801-D42A3033-6DD8-46D7-A19B-05DA5249DC06 Q34580296-B079AEFC-949D-47C2-9DA5-9ED96D3AADA7 Q34927037-9EC0A9D9-F11B-4D7E-A535-A2B5541CE4A5 Q35034254-FCB8A619-B5C9-472C-9572-7684BAA74874 Q35046046-06C25E61-5A86-4ECD-860B-73AC3558BA0A Q35157393-57935128-ACA8-48B4-B61E-BBEE0E1D3953 Q35230750-4E91C295-C83F-4BD1-8E7C-B3557C0B4440 Q35405562-D53D66D2-652B-49D6-A1EF-0FB6D162AD39 Q35489009-83D236AC-7F68-48A1-B789-52583C871C9C Q35565143-3F7D04F5-2F51-47D9-88FB-3E37E1F7B011 Q35644935-3736CE49-4C62-40D6-AA0B-D90A19147BF5 Q35662327-A4857F9A-380E-47FC-98AF-40B2F96F184A Q35688241-8023F19A-C23F-42F1-AFF5-CFE719AB5D4B Q35722876-08FCD9EF-95CD-48EB-83A6-EB8F4887FFBA Q35745564-AB5A00F1-37D7-48AC-A606-A859867A6FA8 Q35903800-75701FEE-26BA-4FAE-80B9-BA2D4E3810BE Q35944978-F3D8759E-01EB-402C-936D-0F4637D9FFA6 Q36010636-DA75A21C-BDB9-4476-8A4D-0FBBD67F36AC Q36204568-86F38831-4FB6-456D-9296-F121932F1DCF Q36223307-4F86F415-F1DE-4307-BD1A-EE9D1048CA8D Q36761540-4DDCAAE0-C531-49F3-A2B8-9103BDE51721 Q37088617-894E3C22-3E72-4862-8EDD-7490E67D3C31 Q37123713-9E032CD4-004B-4F47-BBF1-35E69BD008E8 Q37323597-0FEB72F6-3874-40CD-B73A-EC7F1BCA8270 Q37415246-F590E895-1967-4271-B39F-F16B361FF825 Q37932024-10232A3F-E225-43C5-945C-503A7011880D

P2860

Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-gamma and MyD88 signaling.

http://www.wikidata.org/entity/Q33660251

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@ast

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@en

type

label

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@ast

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@en

prefLabel

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@ast

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@en

P1433

Q33660251-2CEC6230-95D3-41C4-9C09-C66C00698598

P1476

Q33660251-921BA449-47A1-4A2D-B956-7D387EC3D506

P2093

Q33660251-01814AE7-4E2F-496F-98EB-A0926015C2CC

Q33660251-07BCAD21-4445-4CFB-B970-1B2244FA2734

Q33660251-421E1673-5E11-4690-92BF-1CB3FE199D1B

Q33660251-4E5D37C7-EA7C-4AC0-947E-107669BC8453

Q33660251-B97103AD-24B7-4F7E-9B67-5E88B97FF2E0

P2860

Q33660251-08859047-E178-4B78-985B-06233F6A6F29

Q33660251-16006B14-4222-402B-BE75-67D4AABA31A0

Q33660251-167E428E-79F7-4962-8ED4-46F15E07EC89

Q33660251-17F71BE3-0A8F-4352-83AD-781309A285D8

Q33660251-1AB6E054-C992-42D0-BD64-327EBDD3D9EE

Q33660251-1AE81993-2412-4C36-95B7-2D550EC4BA2F

Q33660251-26161FA2-0CCF-4D86-B7A9-9C5A78F618D6

Q33660251-27A02294-7F47-4B51-92E6-C073BA285E67

Q33660251-399617FE-A1B3-4159-B56B-6E292F553597

Q33660251-4218F2DA-8D6F-4D35-8E78-DE5CBDFC3897

P304

Q33660251-446506C4-DE17-4896-B63F-616DE79104B9

P31

Q33660251-67BC89C2-784A-41FB-A64A-9D727AC57F42

P356

Q33660251-16DBDFE2-DC74-4594-9053-FD52F8BB534D

P433

Q33660251-19168EF8-B0B5-4DD2-96CA-4A15C094A076

P478

Q33660251-29E69487-0793-4215-B0CB-7C330FCE9F78

P50

Q33660251-260FE246-C134-46B1-9065-9D94C99D989F

Q33660251-31B396EC-3EB5-4515-8541-9ABD95679A0C

Q33660251-80226DA6-6DA8-44F6-A531-A0FCD314B2C0

Q33660251-BE79D679-A854-45B3-93A5-4A8FBF33A978

P577

Q33660251-FB2162D9-D836-4BA7-B8F8-84DA5B4A6FC5

P5875

Q33660251-D3AEB810-5189-4E9C-905C-042E02F508B5

P698

Q33660251-F09141F4-84B2-4A7E-A303-4DE98205518F

P932

Q33660251-4212CE0D-946B-4DF3-886C-5FF5A8C172D5

P356

JOURNAL.PPAT.1001045

P1433

P1476

Tip-DC development during para ...... IFN-gamma and MyD88 signaling.

@en

P2093

Alain Beschin

http://www.w3.org/2001/XMLSchema#string

Frank Tacke

http://www.w3.org/2001/XMLSchema#string

Michel Hérin

http://www.w3.org/2001/XMLSchema#string

Tom Bosschaerts

http://www.w3.org/2001/XMLSchema#string

Yannick Morias

http://www.w3.org/2001/XMLSchema#string

P2860

iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice

Alternatively activated myeloid cells limit pathogenicity associated with African trypanosomiasis through the IL-10 inducible gene selenoprotein P.

Blood monocytes consist of two principal subsets with distinct migratory properties

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection.

Tumor necrosis factor alpha is a key mediator in the regulation of experimental Trypanosoma brucei infections.

Monocyte-mediated defense against microbial pathogens.

Modulation of liver injury by interleukin-10.

Tumor necrosis factor alpha- and inducible nitric oxide synthase-producing dendritic cells are rapidly recruited to the bladder in urinary tract infection but are dispensable for bacterial clearance

Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques.

P304

e1001045

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PPAT.1001045

http://www.w3.org/2001/XMLSchema#string

P433

8

http://www.w3.org/2001/XMLSchema#string

P478

6

http://www.w3.org/2001/XMLSchema#string

P50

Patrick De Baetselier

Benoît Stijlemans

Daniel Robert Engel

Martin Guilliams

P577

2010-08-12T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

45660633

http://www.w3.org/2001/XMLSchema#string

P698

20714353

http://www.w3.org/2001/XMLSchema#string

P932

2920868

http://www.w3.org/2001/XMLSchema#string