Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania - Wikidata - wikimedia - TriplyDB

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

http://www.wikidata.org/entity/Q33669426

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Cathepsin B in antigen-presenting cells controls mediators of the Th1 immune response during Leishmania major infection A transient reversal of miRNA-mediated repression controls macrophage activation.Temperature-induced protein secretion by Leishmania mexicana modulates macrophage signalling and function.Increased expression of cardiac IL-17 after burn Impact of continuous axenic cultivation in Leishmania infantum virulence Macrophage cytokines: involvement in immunity and infectious diseases The involvement of TLR2 and TLR4 in cytokine and nitric oxide production in visceral leishmaniasis patients before and after treatment with anti-leishmanial drugs.Characterization of the early inflammatory infiltrate at the feeding site of infected sand flies in mice protected from vector-transmitted Leishmania major by exposure to uninfected bites.Structurally optimized analogs of the retrograde trafficking inhibitor Retro-2cycl limit Leishmania infections Melittin induces in vitro death of Leishmania (Leishmania) infantum by triggering the cellular innate immune response.Modulation of anti-inflammatory response in lipopolysaccharide stimulated human THP-1 cell line and mouse model at gene expression level with indigenous putative probiotic lactobacilli Flavonoids Identified from Korean Scutellaria baicalensis Georgi Inhibit Inflammatory Signaling by Suppressing Activation of NF- κ B and MAPK in RAW 264.7 Cells.Leishmania and the macrophage: a multifaceted interaction.Leishmania mexicana promastigotes inhibit macrophage IL-12 production via TLR-4 dependent COX-2, iNOS and arginase-1 expression.The human parasite Leishmania amazonensis downregulates iNOS expression via NF-κB p50/p50 homodimer: role of the PI3K/Akt pathway.Sodium nitroprusside has leishmanicidal activity independent of iNOS.In vivo and in vitro phagocytosis of Leishmania (Leishmania) amazonensis promastigotes by B-1 cells.IL-23 protection against Plasmodium berghei infection in mice is partially dependent on IL-17 from macrophages.Depot Subcutaneous Injection with Chalcone CH8-Loaded Poly(Lactic-Co-Glycolic Acid) Microspheres as a Single-Dose Treatment of Cutaneous Leishmaniasis.Leishmania Hijacks Myeloid Cells for Immune Escape.

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Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

http://www.wikidata.org/entity/Q33669426

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@ast

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@en

type

label

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@ast

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@en

prefLabel

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@ast

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@en

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Journal of Inflammation

P1476

Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania

@en

P2093

Ben L Kelly

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Nicholas J Lapara

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P2860

Regulation of interleukin 12 p40 expression through an NF-kappa B half-site

The influence of restricted calorie intake on peritoneal macrophage function.

Interleukin 10- and Fcgamma receptor-deficient mice resolve Leishmania mexicana lesions

Early enhanced Th1 response after Leishmania amazonensis infection of C57BL/6 interleukin-10-deficient mice does not lead to resolution of infection.

Impaired expression of inflammatory cytokines and chemokines at early stages of infection with Leishmania amazonensis

Toll-like receptors in the pathogenesis of human disease.

TLR signaling: an emerging bridge from innate immunity to atherogenesis.

Interleukin 1alpha promotes Th1 differentiation and inhibits disease progression in Leishmania major-susceptible BALB/c mice

Leishmania major LACK antigen is required for efficient vertebrate parasitization.

Cooperation of Toll-like receptor signals in innate immune defence.

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scholarly article

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10.1186/1476-9255-7-8

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1

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2010-02-02T00:00:00Z

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41806453

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1039930668

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20205812

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2824668

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