Tumor microenvironment-based feed-forward regulation of NOS2 in breast cancer progression. - Wikidata - wikimedia - TriplyDB

Tumor microenvironment-based feed-forward regulation of NOS2 in breast cancer progression.

Tumor microenvironment-based feed-forward regulation of NOS2 in breast cancer progression.

http://www.wikidata.org/entity/Q33674627

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Metabolic, autophagic, and mitophagic activities in cancer initiation and progression The Potential Role of Nitric Oxide in Halting Cancer Progression Through Chemoprevention Gene expression profiles of NO- and HNO-donor treated breast cancer cells: insights into tumor response and resistance pathways Modulation of the Anti-Tumor Efficacy of Photodynamic Therapy by Nitric Oxide Nitric Oxide Down-Regulates Topoisomerase I and Induces Camptothecin Resistance in Human Breast MCF-7 Tumor Cells Signaling and stress: The redox landscape in NOS2 biology Genes associated with histopathologic features of triple negative breast tumors predict molecular subtypes.Roles of the cyclooxygenase 2 matrix metalloproteinase 1 pathway in brain metastasis of breast cancer Decreased miR122 in hepatocellular carcinoma leads to chemoresistance with increased arginine.NOS Inhibition Modulates Immune Polarization and Improves Radiation-Induced Tumor Growth Delay Network analysis of immunotherapy-induced regressing tumours identifies novel synergistic drug combinations.The immunogenetics of Psoriasis: A comprehensive review.Differential Effects of Glycyrrhiza Species on Genotoxic Estrogen Metabolism: Licochalcone A Downregulates P450 1B1, whereas Isoliquiritigenin Stimulates It.Biological signaling by small inorganic molecules Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications.Curcumin Nanoformulation for Cervical Cancer Treatment.Nitric oxide inhibits topoisomerase II activity and induces resistance to topoisomerase II-poisons in human tumor cells.Gasotransmitters in cancer: from pathophysiology to experimental therapy.Systemic RALA/iNOS Nanoparticles: A Potent Gene Therapy for Metastatic Breast Cancer Coupled as a Biomarker of Treatment.The brain metastatic niche.Glycative stress from advanced glycation end products (AGEs) and dicarbonyls: An emerging biological factor in cancer onset and progression.Inducible Nitric Oxide Synthase in the Carcinogenesis of Gastrointestinal Cancers.Nitric Oxide Synthase-2-Derived Nitric Oxide Drives Multiple Pathways of Breast Cancer Progression.Nitric oxide increases the invasion of pancreatic cancer cells via activation of the PI3K-AKT and RhoA pathways after carbon ion irradiation.Nitric oxide: what's new to NO?Exploiting the neoantigen landscape for immunotherapy of pancreatic ductal adenocarcinoma.Nitric oxide inhibits ATPase activity and induces resistance to topoisomerase II-poisons in human MCF-7 breast tumor cells.Integrative Analysis with Monte Carlo Cross-Validation Reveals miRNAs Regulating Pathways Cross-Talk in Aggressive Breast Cancer.Interleukin-38 is released from apoptotic cells to limit inflammatory macrophage responses.Impact of inducible nitric oxide synthase (iNOS) expression on triple negative breast cancer outcome and activation of EGFR and ERK signaling pathways.Bystander effects of nitric oxide in anti-tumor photodynamic therapy.Nitric Oxide: The Forgotten Child of Tumor Metabolism.N w-hydroxy-L-arginine as a novel ethnic specific indicator of estrogen-negative breast cancer.The dichotomous role of H2S in cancer cell biology? Déjà vu all over again.Nitric oxide is cytoprotective to breast cancer spheroids vulnerable to estrogen-induced apoptosis.IL6 derived from cancer-associated fibroblasts promotes chemoresistance via CXCR7 in esophageal squamous cell carcinoma.Multifaceted Roles of Interleukin-6 in Adipocyte-Breast Cancer Cell Interaction.Impact of l-Arginine Metabolism on Immune Response and Anticancer Immunotherapy.Glioma and microglia, a double entendre.The second annual conference of International ovarian cancer consortium and the symposium on tumor microenvironment and therapeutic resistance.

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Tumor microenvironment-based feed-forward regulation of NOS2 in breast cancer progression.

http://www.wikidata.org/entity/Q33674627

description

2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Tumor microenvironment-based f ...... in breast cancer progression.

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Tumor microenvironment-based f ...... in breast cancer progression.

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Tumor microenvironment-based f ...... in breast cancer progression.

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Tumor microenvironment-based f ...... in breast cancer progression.

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Tumor microenvironment-based f ...... in breast cancer progression.

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Chand Khanna

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Christopher H Switzer

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David A Wink

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Howard A Young

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Julie L Heinecke

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Lisa A Ridnour

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Michael M Lizardo

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Robert Y S Cheng

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P2860

Hypoxia stimulates migration of breast cancer cells via the PERK/ATF4/LAMP3-arm of the unfolded protein response

Hallmarks of Cancer: The Next Generation

miRNA-939 regulates human inducible nitric oxide synthase posttranscriptional gene expression in human hepatocytes

Nitric oxide synthase and breast cancer: role of TIMP-1 in NO-mediated Akt activation

hCAF1/CNOT7 regulates interferon signalling by targeting STAT1.

Up-regulation of pro-inflammatory genes as adaptation to hypoxia in MCF-7 cells and in human mammary invasive carcinoma microenvironment.

Ets-1 is a transcriptional mediator of oncogenic nitric oxide signaling in estrogen receptor-negative breast cancer.

Increased NOS2 predicts poor survival in estrogen receptor-negative breast cancer patients.

The two faces of interferon-γ in cancer.

S-nitrosylation of EGFR and Src activates an oncogenic signaling network in human basal-like breast cancer.

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