Higher expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 (Flk-1) and metalloproteinase-9 (MMP-9) in a rat model of peritoneal endometriosis is similar to cancer diseases.

Higher expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 (Flk-1) and metalloproteinase-9 (MMP-9) in a rat model of peritoneal endometriosis is similar to cancer diseases.

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http://www.wikidata.org/entity/Q33677926

Q33677926

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Endometriosis: where are we and where are we going?Agent-based model of angiogenesis simulates capillary sprout initiation in multicellular networks.Multicenter phase II study of apatinib, a novel VEGFR inhibitor in heavily pretreated patients with metastatic triple-negative breast cancer.Increased expression of ID2, PRELP and SMOC2 genes in patients with endometriosis.Antiangiogenesis therapy of endometriosis using PAMAM as a gene vector in a noninvasive animal model.Pigment epithelium derived factor inhibits the growth of human endometrial implants in nude mice and of ovarian endometriotic stromal cells in vitro Role of vascular endothelial growth factor polymorphisms (-2578C>A, -460 T>C, -1154G>A, +405G>C and +936C>T) in endometriosis: a case-control study with Brazilians.Inducing malignant transformation of endometriosis in rats by long-term sustaining hyperestrogenemia and type II diabetes.Inhibition of adhesion, proliferation, and invasion of primary endometriosis and endometrial stromal and ovarian carcinoma cells by a nonhyaluronan adhesion barrier gel.Vascular endothelial growth factor receptor-2 inhibitor cediranib causes regression of endometriotic lesions in a rat model Proangiogenic Tie2(+) macrophages infiltrate human and murine endometriotic lesions and dictate their growth in a mouse model of the disease.Involvement of vascular endothelial growth factor -460 C/T, +405 G/C and +936 C/T polymorphisms in the development of endometriosis.Developing a Noninvasive Procedure Using Labeled Monoclonal Antibody Anti-VEGF (Bevacizumab) for Detection of Endometriosis Molecular basis for defining the pineal gland and pinealocytes as targets for tumor necrosis factor.Capecitabine combined with (-)-epigallocatechin-3-gallate inhibits angiogenesis and tumor growth in nude mice with gastric cancer xenografts Correlation between matrix metalloproteinase-9 and endometriosis.Demographic and Clinical Features of Endometrial Polyps in Patients with Endometriosis.Towards an understanding of the molecular mechanism of endometriosis: unbalancing epithelial-stromal genetic conflict.The oil-resin of the tropical rainforest tree Copaifera langsdorffii reduces cell viability, changes cell morphology and induces cell death in human endometriotic stromal cultures.p27kip1 overexpression regulates VEGF expression, cell proliferation and apoptosis in cell culture from eutopic endometrium of women with endometriosis.Effects of dienogest on surgically induced endometriosis in rats after repeated oral administration.Expression profile of extracellular matrix and adhesion molecules in the development of endometriosis in a mouse model.Effects on in vitro fertilization-embryo transfer outcomes of vascular endothelial growth factor receptor-1, -2 and -3 in eutopic endometrial tissue of women with endometriosis.The miRNA Mirage: How Close Are We to Finding a Non-Invasive Diagnostic Biomarker in Endometriosis? A Systematic Review.Overexpression of chloride channel-3 is associated with the increased migration and invasion ability of ectopic endometrial cells from patients with endometriosis.Soluble CD44 concentration in the serum and peritoneal fluid samples of patients with different stages of endometriosis.In vivo monitoring of angiogenesis in a mouse hindlimb ischemia model using fluorescent peptide-based probes.17 β-Estradiol promotes vascular endothelial growth factor expression via the Wnt/β-catenin pathway during the pathogenesis of endometriosis.Intracellular Wnt/Beta-Catenin Signaling Underlying 17beta-Estradiol-Induced Matrix Metalloproteinase 9 Expression in Human Endometriosis.Effects of Pazopanib, Sunitinib, and Sorafenib, Anti-VEGF Agents, on the Growth of Experimental Endometriosis in Rats.Phosphorylated VEGFR2 and hypertension: potential biomarkers to indicate VEGF-dependency of advanced breast cancer in anti-angiogenic therapy.6-Shogaol reduces progression of experimental endometriosis and regulation of VGEF and inhibition of COX-2 and PGE2-mediated inflammatory responses

P2860

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P2860

Higher expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 (Flk-1) and metalloproteinase-9 (MMP-9) in a rat model of peritoneal endometriosis is similar to cancer diseases.

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http://www.wikidata.org/entity/Q33677926

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010年の論文

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