COX-2 inhibition improves immunotherapy and is associated with decreased numbers of myeloid-derived suppressor cells in mesothelioma. Celecoxib influences MDSC function.
about
The Potential Protective Effects of Polyphenols in Asbestos-Mediated Inflammation and Carcinogenesis of MesotheliumPaving the Road to Tumor Development and Spreading: Myeloid-Derived Suppressor Cells are Ruling the FateHampering immune suppressors: therapeutic targeting of myeloid-derived suppressor cells in cancerImmunity of human epithelial ovarian carcinoma: the paradigm of immune suppression in cancerImmune modulation of the tumor microenvironment for enhancing cancer immunotherapyCOX-2 and PGE2-dependent immunomodulation in breast cancerMyeloid-derived suppressor cells as therapeutic target in hematological malignanciesUpdate on the challenges and recent advances in cancer immunotherapyMyeloid derived suppressor cells-An overview of combat strategies to increase immunotherapy efficacyImmunosuppression associated with chronic inflammation in the tumor microenvironmentMesothelioma response to carbon nanotubes is associated with an early and selective accumulation of immunosuppressive monocytic cellsCoordinated regulation of myeloid cells by tumoursThe emerging immunological role of post-translational modifications by reactive nitrogen species in cancer microenvironmentMyeloid derived suppressor cells - a new therapeutic target in the treatment of cancer.Vaccination with dendritic cells loaded with allogeneic brain tumor cells for recurrent malignant brain tumors induces a CD4(+)IL17(+) response.TLR/MyD88-mediated Innate Immunity in Intestinal Graft-versus-Host DiseaseDownregulation of CD40 expression contributes to the accumulation of myeloid-derived suppressor cells in gastric tumors.New roads open up for implementing immunotherapy in mesothelioma.Combination of an agonistic anti-CD40 monoclonal antibody and the COX-2 inhibitor celecoxib induces anti-glioma effects by promotion of type-1 immunity in myeloid cells and T-cells.Patient-tailored modulation of the immune system may revolutionize future lung cancer treatment.Intermittent chemotherapy can retain the therapeutic potential of anti-CD137 antibody during the late tumor-bearing state.Positive feedback between PGE2 and COX2 redirects the differentiation of human dendritic cells toward stable myeloid-derived suppressor cells.6-Thioguanine-loaded polymeric micelles deplete myeloid-derived suppressor cells and enhance the efficacy of T cell immunotherapy in tumor-bearing mice.Finasteride Enhances the Generation of Human Myeloid-Derived Suppressor Cells by Up-Regulating the COX2/PGE2 PathwayMolecular pathways: tumor-infiltrating myeloid cells and reactive oxygen species in regulation of tumor microenvironmentCorrelation between circulating myeloid-derived suppressor cells and Th17 cells in esophageal cancer.Transcriptional regulation of myeloid-derived suppressor cells.Myeloid-derived suppressor cells from tumor-bearing mice impair TGF-β-induced differentiation of CD4+CD25+FoxP3+ Tregs from CD4+CD25-FoxP3- T cellsRepurposing Drugs in Oncology (ReDO)-diclofenac as an anti-cancer agentCox-2 inhibition enhances the activity of sunitinib in human renal cell carcinoma xenografts.Immunotherapy in pediatric malignancies: current status and future perspectivesAn inflammatory mediator, prostaglandin E2, in colorectal cancer.Microenvironment generated during EGFR targeted killing of pancreatic tumor cells by ATC inhibits myeloid-derived suppressor cells through COX2 and PGE2 dependent pathway.Myeloid derived suppressor cells: Targets for therapy.Activated hepatic stellate cells promote liver cancer by induction of myeloid-derived suppressor cells through cyclooxygenase-2.Murine mesothelioma induces locally-proliferating IL-10(+)TNF-α(+)CD206(-)CX3CR1(+) M3 macrophages that can be selectively depleted by chemotherapy or immunotherapy.Trial Watch-Small molecules targeting the immunological tumor microenvironment for cancer therapy.Potential therapeutic anti-tumor effect of a Salmonella-based vaccine.Pancreatic Cancer Cells Isolated from Muc1-Null Tumors Favor the Generation of a Mature Less Suppressive MDSC Population.Dendritic cells in the pathogenesis and treatment of human diseases: a Janus Bifrons?
P2860
Q26750451-4AE6E6AC-5BE4-452A-BF73-4EF76D841949Q26777205-4BB0485D-2AF7-4E28-B945-F3DE533D439AQ26863418-3CA1A5B5-6BE1-4F74-8737-AA4ABD4B7E7FQ26996829-C5CDF69C-A14D-43F6-8C51-205D56F24238Q27002646-10B281EE-7524-4DDB-BD37-984F343ACF24Q27003937-2276E85B-C0E1-4B34-AEB6-F31A7402B856Q27010234-BAE9DABB-1270-4D8D-A05D-F86DB48820C2Q27014693-A660BDFD-4466-48EE-ABC6-094FCFB7CFF4Q28082461-12FB044D-1847-4004-BC97-0B1D5F5AF775Q28083450-45861E46-4B63-415C-9E9E-2A8A77F7A4E4Q28387268-75579093-0222-4825-A1E6-4C995B09D56EQ28395157-0C1675BE-8E60-4BE2-94A0-797B4CB5C18BQ28396481-5DC517F2-9774-4249-B5C1-BDC28935695CQ33606107-A92E42E1-CC4E-4341-A453-4ECE53FBBA65Q33606132-BFA7D0A6-1270-4EEA-8198-43EF692AAD03Q33835099-87D9C71D-DD7D-4179-BF25-D9CB76778230Q33844271-0385B6DC-F16A-47F2-9461-3FE50ED00684Q34331993-B1864C05-3B51-4E42-944A-4373AD55BD28Q34457611-9466BA95-E73E-4E8A-BFB4-DDC999C3CA9CQ34502899-BB13EFD7-B779-4FF8-A128-4C2DC860A8DCQ35051306-79632101-F733-46A7-ADDD-E94E83CFA686Q35553327-714DDB01-EC5E-4949-A12F-8BDC8F383BB2Q35863346-CEC39B9E-A4F6-4AEA-A332-7B1494302A00Q36037921-B3552F3B-8A01-47AA-A149-A31B2B6A930CQ36243616-1DFF9D5C-4B29-44FE-BA5E-0062908353E5Q36316728-F16545B8-9940-474E-846D-68316FC19FE5Q36322351-AAF05660-3788-44B5-99BF-8E7FCEA6D4DAQ36332711-EAF344AE-4ACA-449A-B181-E3E14B96997FQ36483712-574D6F0E-485A-401B-A97B-02C5592475FBQ36594073-5684C4DC-A315-4DA8-ADAE-FE96A89443FAQ36691818-CC2B96FC-482A-4B67-9945-E394556E525DQ36701908-38E34CB4-14C2-49D0-9507-154BAF55E862Q36718698-B6814959-167A-42DF-8DF9-058331961A14Q36845851-C8BFD913-530A-46EF-8CD3-EB1DE924ED8CQ36962436-14062E8E-D46C-405B-8614-F752C1D28927Q37078960-176C85A5-0847-4D4D-B7BF-990CA67038A7Q37079141-DC96883A-08B7-427A-9C2D-473D0D040A65Q37530067-ED620049-C7A1-4303-BD57-E228C380F9FDQ37598872-517EBC0F-F19C-44B9-91FC-A5975ED7195CQ37945301-0D0AB406-62D5-4F8A-A658-F44BE41C6E2C
P2860
COX-2 inhibition improves immunotherapy and is associated with decreased numbers of myeloid-derived suppressor cells in mesothelioma. Celecoxib influences MDSC function.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@ast
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@en
type
label
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@ast
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@en
prefLabel
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@ast
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@en
P2093
P2860
P356
P1433
P1476
COX-2 inhibition improves immu ...... oxib influences MDSC function.
@en
P2093
Henk C Hoogsteden
Joachim G J V Aerts
Joost P J J Hegmans
Joris D Veltman
Margaretha E H Lambers
Menno van Nimwegen
Rudi W Hendriks
P2860
P2888
P356
10.1186/1471-2407-10-464
P407
P577
2010-08-30T00:00:00Z
P5875
P6179
1053035444