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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

http://www.wikidata.org/entity/Q33679560

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Ca2+/calmodulin-dependent protein kinase II alpha is required for the initiation and maintenance of opioid-induced hyperalgesia Neuropathic pain activates the endogenous kappa opioid system in mouse spinal cord and induces opioid receptor tolerance The role of the arginine metabolome in pain: implications for sickle cell disease The mechanism of μ-opioid receptor (MOR)-TRPV1 crosstalk in TRPV1 activation involves morphine anti-nociception, tolerance and dependence mTOR, a new potential target for chronic pain and opioid-induced tolerance and hyperalgesia Induction of synaptic long-term potentiation after opioid withdrawal Alteration in the voltage dependence of NMDA receptor channels in rat dorsal horn neurones following peripheral inflammation Involvement of subtype 1 metabotropic glutamate receptors in apoptosis and caspase-7 over-expression in spinal cord of neuropathic rats Morphine enhances microglial migration through modulation of P2X4 receptor signaling Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice Platelet-derived growth factor receptor-β antagonism restores morphine analgesic potency against neuropathic pain.Sensitization of the trigeminovascular pathway: perspective and implications to migraine pathophysiology.Morphine tolerance attenuates the resolution of postoperative pain and enhances spinal microglial p38 and extracellular receptor kinase phosphorylation.Delta-opioid receptor analgesia is independent of microglial activation in a rat model of neuropathic pain Inhibition of microglial P2X4 receptors attenuates morphine tolerance, Iba1, GFAP and mu opioid receptor protein expression while enhancing perivascular microglial ED2.Lack of effect of chronic dextromethorphan on experimental pain tolerance in methadone-maintained patients.NO as a signalling molecule in the nervous system.Cellular memory in spinal nociceptive circuitry.Exploring the neuroimmunopharmacology of opioids: an integrative review of mechanisms of central immune signaling and their implications for opioid analgesia.Biphasic effect of curcumin on morphine tolerance: a preliminary evidence from cytokine/chemokine protein array analysis.Melatonin prevents morphine-induced hyperalgesia and tolerance in rats: role of protein kinase C and N-methyl-D-aspartate receptors.The neurobiology of opiate tolerance, dependence and sensitization: mechanisms of NMDA receptor-dependent synaptic plasticity.Effects and consequences of nerve injury on the electrical properties of sensory neurons.Anticipating and treating opioid-associated adverse effects.Epigenetic regulation of chronic pain Involvement of neuronal TGF-β activated kinase 1 in the development of tolerance to morphine-induced antinociception in rat spinal cord.Current gene therapy using viral vectors for chronic pain Neuroimmune activation and neuroinflammation in chronic pain and opioid tolerance/hyperalgesia.Intrathecal rapamycin attenuates morphine-induced analgesic tolerance and hyperalgesia in rats with neuropathic pain.Prevention of Remifentanil Induced Postoperative Hyperalgesia by Dexmedetomidine via Regulating the Trafficking and Function of Spinal NMDA Receptors as well as PKC and CaMKII Level In Vivo and In Vitro The association between frequent alcohol drinking and opioid consumption after abdominal surgery: A retrospective analysis.Periaqueductal gray neuroplasticity following chronic morphine varies with age: role of oxidative stress."Listening" and "talking" to neurons: implications of immune activation for pain control and increasing the efficacy of opioids.Repeated injections of orexin-A developed behavioral tolerance to its analgesic effects in rats Norman Cousins Lecture. Glia as the "bad guys": implications for improving clinical pain control and the clinical utility of opioids.Distinct expression of synaptic NR2A and NR2B in the central nervous system and impaired morphine tolerance and physical dependence in mice deficient in postsynaptic density-93 protein.BK channels in microglia are required for morphine-induced hyperalgesia.Memantine and dizocilpine interactions with antinociceptive or discriminative stimulus effects of morphine in rats after acute or chronic treatment with morphine.Mesenchymal Stem Cells Reversed Morphine Tolerance and Opioid-induced Hyperalgesia.Downregulation of miR-219 enhances brain-derived neurotrophic factor production in mouse dorsal root ganglia to mediate morphine analgesic tolerance by upregulating CaMKIIγ.

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P2860

Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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1999 nî lūn-bûn

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1999 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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1999 թվականի հուլիսին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions.

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Potentiation of opiate analgesia and apparent reversal of morphine tolerance by proglumide

The chemistry of peroxynitrite: a product from the reaction of nitric oxide with superoxide

A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man.

Differential regulation by cAMP-dependent protein kinase and protein kinase C of the mu opioid receptor coupling to a G protein-activated K+ channel.

Expansion of receptive fields of spinal lamina I projection neurons in rats with unilateral adjuvant-induced inflammation: the contribution of dorsal horn mechanisms.

Mechanisms of hyperalgesia and morphine tolerance: a current view of their possible interactions.

Modulation of neuropathic pain behavior in rats by spinal disinhibition and NMDA receptor blockade of injury discharge.

DNA strand breakage and activation of poly-ADP ribosyltransferase: a cytotoxic pathway triggered by peroxynitrite.

Nitric oxide activation of poly(ADP-ribose) synthetase in neurotoxicity.

Generation of superoxide by purified brain nitric oxide synthase.

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