Fatty acid amide hydrolase in prostate cancer: association with disease severity and outcome, CB1 receptor expression and regulation by IL-4 - Wikidata - wikimedia - TriplyDB

Fatty acid amide hydrolase in prostate cancer: association with disease severity and outcome, CB1 receptor expression and regulation by IL-4

Fatty acid amide hydrolase in prostate cancer: association with disease severity and outcome, CB1 receptor expression and regulation by IL-4

http://www.wikidata.org/entity/Q33680523

about

Endocannabinoid system as a regulator of tumor cell malignancy - biological pathways and clinical significance Anticancer mechanisms of cannabinoids Endocannabinoids as Guardians of Metastasis Potential upstream regulators of cannabinoid receptor 1 signaling in prostate cancer: a Bayesian network analysis of data from a tissue microarray.Tumour Cannabinoid CB(1) receptor and phosphorylated epidermal growth factor receptor expression are additive prognostic markers for prostate cancer.α-synuclein reactive antibodies as diagnostic biomarkers in blood sera of Parkinson's disease patients.High levels of γ-glutamyl hydrolase (GGH) are associated with poor prognosis and unfavorable clinical outcomes in invasive breast cancer Association between cannabinoid CB₁ receptor expression and Akt signalling in prostate cancer.Regulation of circulating endocannabinoids associated with cancer and metastases in mice and humans.Monoacylglycerol lipase exerts dual control over endocannabinoid and fatty acid pathways to support prostate cancer.The endocannabinoid system and cancer: therapeutic implication.The influence of monoacylglycerol lipase inhibition upon the expression of epidermal growth factor receptor in human PC-3 prostate cancer cells.Prognosis of treatment response (pathological complete response) in breast cancer.The endocannabinoid system and sex steroid hormone-dependent cancers.Update on the endocannabinoid system as an anticancer target.The endocannabinoid system in prostate cancer.Receptor-dependent and receptor-independent endocannabinoid signaling: a therapeutic target for regulation of cancer growth.The evolving role of the endocannabinoid system in gynaecological cancer.The cannabinoid WIN 55,212-2 prevents neuroendocrine differentiation of LNCaP prostate cancer cells.Antitumorigenic targets of cannabinoids - current status and implications.Phyto-, endo- and synthetic cannabinoids: promising chemotherapeutic agents in the treatment of breast and prostate carcinomas.Systematic review of the potential role of cannabinoids as antiproliferative agents for urological cancers Effects of tumour necrosis factor α upon the metabolism of the endocannabinoid anandamide in prostate cancer cells.18F-MK-9470 PET imaging of the type 1 cannabinoid receptor in prostate carcinoma: a pilot study.Cannabinoids Modulate Neuronal Activity and Cancer by CB1 and CB2 Receptor-Independent Mechanisms.INSIGHT ON THE IMPACT OF ENDOCANNABINOID SYSTEM IN CANCER: A REVIEW.The current state and future perspectives of cannabinoids in cancer biology.

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P2860

Fatty acid amide hydrolase in prostate cancer: association with disease severity and outcome, CB1 receptor expression and regulation by IL-4

http://www.wikidata.org/entity/Q33680523

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2010 nî lūn-bûn

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2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2010 թվականի օգոստոսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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Fatty acid amide hydrolase in ...... ression and regulation by IL-4

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Anders Bergh

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Christopher J Fowler

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Emma Persson

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Torvald Granfors

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P2860

Genetic and physical mapping of the human cannabinoid receptor gene to chromosome 6q14-q15

Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation

Modulation of anxiety through blockade of anandamide hydrolysis

Low stroma androgen receptor level in normal and tumor prostate tissue is related to poor outcome in prostate cancer patients.

The expression level of CB1 and CB2 receptors determines their efficacy at inducing apoptosis in astrocytomas.

Suppression of nerve growth factor Trk receptors and prolactin receptors by endocannabinoids leads to inhibition of human breast and prostate cancer cell proliferation.

Monoacylglycerol lipase regulates a fatty acid network that promotes cancer pathogenesis.

The duplicitous effects of interleukin 4 on tumour immunity: how can the same cytokine improve or impair control of tumour growth?

A new strategy to block tumor growth by inhibiting endocannabinoid inactivation.

Receiver-operating characteristic (ROC) plots: a fundamental evaluation tool in clinical medicine.

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