Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
about
Mild cognitive impairment: pathology and mechanisms.Sortilin Fragments Deposit at Senile Plaques in Human CerebrumThe Intersection of NGF/TrkA Signaling and Amyloid Precursor Protein Processing in Alzheimer's Disease NeuropathologyNGF and proNGF regulate functionally distinct mRNAs in PC12 cells: an early gene expression profiling.Sortilin: an unusual suspect in cholesterol metabolism: from GWAS identification to in vivo biochemical analyses, sortilin has been identified as a novel mediator of human lipoprotein metabolism.A comprehensive map of the human urinary proteomeUpregulation of select rab GTPases in cholinergic basal forebrain neurons in mild cognitive impairment and Alzheimer's disease.The role of the retromer complex in aging-related neurodegeneration: a molecular and genomic review.Hippocampal proNGF signaling pathways and β-amyloid levels in mild cognitive impairment and Alzheimer diseaseVps10 family proteins and the retromer complex in aging-related neurodegeneration and diabetes.Independent and epistatic effects of variants in VPS10-d receptors on Alzheimer disease risk and processing of the amyloid precursor protein (APP)The neuropathological profile of mild cognitive impairment (MCI): a systematic review.The neurotrophins and their role in Alzheimer's disease.Dynamic nature of the p75 neurotrophin receptor in response to injury and diseasePROneurotrophins and CONSequences.ProNGF, but Not NGF, Switches from Neurotrophic to Apoptotic Activity in Response to Reductions in TrkA Receptor Levels.Amyloid beta₁₋₄₂ (Aβ₄₂) up-regulates the expression of sortilin via the p75(NTR)/RhoA signaling pathway.Prions amplify through degradation of the VPS10P sorting receptor sortilin.The pro-neurotrophin receptor sortilin is a major neuronal apolipoprotein E receptor for catabolism of amyloid-β peptide in the brainComplexity and Selectivity of γ-Secretase Cleavage on Multiple Substrates: Consequences in Alzheimer's Disease and Cancer.Trafficking in Alzheimer's Disease: Modulation of APP Transport and Processing by the Transmembrane Proteins LRP1, SorLA, SorCS1c, Sortilin, and Calsyntenin.Up-regulation of sortilin mediated by amyloid-β and p75(NTR): safety lies in the middle course.
P2860
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P2860
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@ast
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@en
type
label
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@ast
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@en
prefLabel
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@ast
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease.
@en
P2093
P2860
P1433
P1476
Preservation of cortical sortilin protein levels in MCI and Alzheimer's disease
@en
P2093
Elliott J Mufson
Joanne Wuu
Scott E Counts
P2860
P304
P356
10.1016/J.NEULET.2010.01.023
P407
P577
2010-01-18T00:00:00Z