Fas receptor is required for estrogen deficiency-induced bone loss in mice - Wikidata - wikimedia - TriplyDB

Fas receptor is required for estrogen deficiency-induced bone loss in mice

Fas receptor is required for estrogen deficiency-induced bone loss in mice

http://www.wikidata.org/entity/Q33691200

about

Targeting Fas in osteoresorptive disorders.Osteoblast-induced osteoclast apoptosis by fas ligand/FAS pathway is required for maintenance of bone mass.A new regulator of osteoclastogenesis: estrogen response element-binding protein in bone.Technetium-99 conjugated with methylene diphosphonate ameliorates ovariectomy-induced osteoporotic phenotype without causing osteonecrosis in the jaw.Fas- and Mitochondria-Mediated Signaling Pathway Involved in Osteoblast Apoptosis Induced by AlCl3.Increased chemotaxis and activity of circulatory myeloid progenitor cells may contribute to enhanced osteoclastogenesis and bone loss in the C57BL/6 mouse model of collagen-induced arthritis.Modulation of unloading-induced bone loss in mice with altered ERK signaling.The Potential of Probiotics as a Therapy for Osteoporosis.

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Fas receptor is required for estrogen deficiency-induced bone loss in mice

http://www.wikidata.org/entity/Q33691200

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

@wuu

name

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@ast

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@en

type

label

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@ast

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@en

prefLabel

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@ast

Fas receptor is required for estrogen deficiency-induced bone loss in mice

@en

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LABINVEST.2009.144

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Laboratory Investigation

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Fas receptor is required for estrogen deficiency-induced bone loss in mice

@en

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Danka Grcevic

http://www.w3.org/2001/XMLSchema#string

Hrvoje Cvija

http://www.w3.org/2001/XMLSchema#string

Ivan Kresimir Lukic

http://www.w3.org/2001/XMLSchema#string

Karlo Mihovilovic

http://www.w3.org/2001/XMLSchema#string

Natasa Kovacic

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Peter Ian Croucher

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Vedran Katavic

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Vladimir Grubisic

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P2860

Estrogen protects bone by inducing Fas ligand in osteoblasts to regulate osteoclast survival

The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the games

The multifaceted role of Fas signaling in immune cell homeostasis and autoimmunity

Apoptotic role of Fas/Fas ligand system in the regulation of erythropoiesis

Estrogen deficiency accelerates autoimmune exocrinopathy in murine Sjögren's syndrome through fas-mediated apoptosis

Estrogens up-regulate the Fas/FasL apoptotic pathway in lactotropes

The Fas death factor

Tumor necrosis factor-alpha and interleukin-1beta increase the Fas-mediated apoptosis of human osteoblasts.

Estrogen prevents bone loss via estrogen receptor alpha and induction of Fas ligand in osteoclasts.

Caspases: keys in the ignition of cell death.

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labinvest.2009.144

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402-413

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scholarly article

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10.1038/LABINVEST.2009.144

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3

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90

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2010-01-18T00:00:00Z

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20084056

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