Inhibition of hepatic mitochondrial aldehyde dehydrogenase by carbon tetrachloride through JNK-mediated phosphorylation.

Inhibition of hepatic mitochondrial aldehyde dehydrogenase by carbon tetrachloride through JNK-mediated phosphorylation.

Item
http://www.wikidata.org/entity/Q33699704
Mitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modificationsFormation of 4-hydroxynonenal from cardiolipin oxidation: Intramolecular peroxyl radical addition and decompositionAldehyde dehydrogenase 1B1: molecular cloning and characterization of a novel mitochondrial acetaldehyde-metabolizing enzymeA simple method to systematically study oxidatively modified proteins in biological samples and its applicationsMechanisms of MDMA (ecstasy)-induced oxidative stress, mitochondrial dysfunction, and organ damage.Post-translational modifications of mitochondrial aldehyde dehydrogenase and biomedical implications.Increased oxidative-modifications of cytosolic proteins in 3,4-methylenedioxymethamphetamine (MDMA, ecstasy)-exposed rat liver.Organotypic liver culture models: meeting current challenges in toxicity testing.Critical role of c-jun N-terminal protein kinase in promoting mitochondrial dysfunction and acute liver injuryMitochondrial dysfunction and cell death in neurodegenerative diseases through nitroxidative stressIncreased nitroxidative stress promotes mitochondrial dysfunction in alcoholic and nonalcoholic fatty liver disease.Free radical oxidation of cardiolipin: chemical mechanisms, detection and implication in apoptosis, mitochondrial dysfunction and human diseases.Inhibition of aldehyde dehydrogenase 2 by oxidative stress is associated with cardiac dysfunction in diabetic rats.Enhanced Phosphorylation of Bax and Its Translocation into Mitochondria in the Brains of Individuals Affiliated with Alzheimer's Disease.Preventive effects of indole-3-carbinol against alcohol-induced liver injury in mice via antioxidant, anti-inflammatory, and anti-apoptotic mechanisms: Role of gut-liver-adipose tissue axis.Ganoderma lucidum protects liver mitochondrial oxidative stress and improves the activity of electron transport chain in carbon tetrachloride intoxicated rats.Glutathione-dependent formaldehyde dehydrogenase (ADH3) and low km mitochondrial aldehyde dehydrogenase (ALDH2). New evidence for differential expression in the rat retina in response to oxidative stress
P2860

Inhibition of hepatic mitochondrial aldehyde dehydrogenase by carbon tetrachloride through JNK-mediated phosphorylation.

Item
http://www.wikidata.org/entity/Q33699704
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@ast
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@en
type
label
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@ast
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@en
prefLabel
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@ast
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921
P932
P356
P1433
P1476
Inhibition of hepatic mitochon ...... JNK-mediated phosphorylation.
@en
P2093
P2860
P304
P31
P356
10.1016/J.FREERADBIOMED.2009.11.008
P433
P478
P577
P698
P921
P932