Direct targets of Klf5 transcription factor contribute to the maintenance of mouse embryonic stem cell undifferentiated state.
about
The double life of KLF5: Opposing roles in regulation of gene-expression, cellular function, and transformationA direct physical interaction between Nanog and Sox2 regulates embryonic stem cell self-renewalKruppel-like factor 5 controls villus formation and initiation of cytodifferentiation in the embryonic intestinal epitheliumThe PR/SET domain zinc finger protein Prdm4 regulates gene expression in embryonic stem cells but plays a nonessential role in the developing mouse embryoKlf5 Mediates Odontoblastic Differentiation through Regulating Dentin-Specific Extracellular Matrix Gene Expression during Mouse Tooth DevelopmentKrüppel-like transcription factors and control of pluripotency.ChIP-Array: combinatory analysis of ChIP-seq/chip and microarray gene expression data to discover direct/indirect targets of a transcription factorCritical role of Klf5 in regulating gene expression during post-eyelid opening maturation of mouse corneas.Regulatable in vivo biotinylation expression system in mouse embryonic stem cells.Learning biomarkers of pluripotent stem cells in mouse.Krüppel-like factors 4 and 5 expression and their involvement in differentiation of oral carcinomas.Murine Embryonic Stem Cell Plasticity Is Regulated through Klf5 and Maintained by Metalloproteinase MMP1 and Hypoxia.The PRC2-associated factor C17orf96 is a novel CpG island regulator in mouse ES cells.KLF5 activates microRNA 200 transcription to maintain epithelial characteristics and prevent induced epithelial-mesenchymal transition in epithelial cells.Altered enhancer transcription underlies Huntington's disease striatal transcriptional signaturePluripotent genes in avian stem cells.Krüppel-like factors in cancer progression: three fingers on the steering wheel.Kruppel-like factor 5 is required for formation and differentiation of the bladder urotheliumKrüppel-like factor 5 is essential for maintenance of barrier function in mouse colon.Optimization of culture condition of human bone marrow stromal cells in terms of purification, proliferation, and pluripotency.Klf5 controls bone marrow homing of stem cells and progenitors through Rab5-mediated β1/β2-integrin trafficking.miR-125b regulates the early steps of ESC differentiation through dies1 in a TGF-independent manner.Candidate gene expression patterns in rabbit preimplantation embryos developed in vivo and in vitro.Inactivation of Klf5 by zinc finger nuclease downregulates expression of pluripotent genes and attenuates colony formation in embryonic stem cells.Complement 3 activates the renal renin-angiotensin system by induction of epithelial-to-mesenchymal transition of the nephrotubulus in mice.Klf4 and Klf5 differentially inhibit mesoderm and endoderm differentiation in embryonic stem cells.
P2860
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P2860
Direct targets of Klf5 transcription factor contribute to the maintenance of mouse embryonic stem cell undifferentiated state.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@ast
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@en
type
label
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@ast
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@en
prefLabel
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@ast
Direct targets of Klf5 transcr ...... m cell undifferentiated state.
@en
P2093
P2860
P50
P356
P1433
P1476
Direct targets of Klf5 transcr ...... em cell undifferentiated state
@en
P2093
Dario Antonini
Luigi Aloia
Silvia Parisi
Simona Ciriello
Vincenzo De Simone
P2860
P2888
P356
10.1186/1741-7007-8-128
P577
2010-09-27T00:00:00Z
P5875
P6179
1046557557