Ionizing radiation regulates expression of the c-jun protooncogene.
about
Role for Lyn tyrosine kinase as a regulator of stress-activated protein kinase activity in response to DNA damageAmyloid beta binds trimers as well as monomers of the 75-kDa neurotrophin receptor and activates receptor signalingATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agentsInduction by ionizing radiation of the gadd45 gene in cultured human cells: lack of mediation by protein kinase CSynergistic Gene Expression Signature Observed in TK6 Cells upon Co-Exposure to UVC-Irradiation and Protein Kinase C-Activating Tumor PromotersActivation of c-Jun N-terminal kinase 1 by UV irradiation is inhibited by wortmannin without affecting c-iun expressionThe stress response to ionizing radiation involoves c-Abl-dependent phosphorylation of SHPTP1.Histone H1 dephosphorylation is mediated through a radiation-induced signal transduction pathway dependent on ATM.Rodent cell transformation and immediate early gene expression following 60-Hz magnetic field exposure.Identification of NF-jun, a novel inducible transcription factor that regulates c-jun gene transcription.MicroRNA-21 is involved in ionizing radiation-promoted liver carcinogenesis.Ionizing radiation induces expression and binding activity of the nuclear factor kappa B.Ultraviolet radiation rapidly induces tyrosine phosphorylation and calcium signaling in lymphocytes.p53, Cip1, and Gadd153 expression following treatment of A549 cells with natural and man-made vitreous fibers.Radiation-induced heart disease: pathologic abnormalities and putative mechanismsTranscription factors activated in mammalian cells after clinically relevant doses of ionizing radiation.Differential expression of protein kinase C epsilon protein in lung cancer cell lines by ionising radiation.Tyrosine phosphorylation is a mandatory proximal step in radiation-induced activation of the protein kinase C signaling pathway in human B-lymphocyte precursors.Current status of tumor radiogenic therapy.Reactive oxygen intermediates target CC(A/T)6GG sequences to mediate activation of the early growth response 1 transcription factor gene by ionizing radiation.Isolation of x-ray-inducible transcripts from radioresistant human melanoma cells.Rapid and preferential activation of the c-jun gene during the mammalian UV response.Inducibility of the DNA repair gene encoding O6-methylguanine-DNA methyltransferase in mammalian cells by DNA-damaging treatments.Glutathione S-transferases: gene structure and regulation of expression.DNA damage by oxygen-derived species. Its mechanism and measurement in mammalian systems.Ionizing radiation activates transcription of the EGR1 gene via CArG elements.Protein kinase C mediates x-ray inducibility of nuclear signal transducers EGR1 and JUN.Proteolytic activation of protein kinase C delta by an ICE-like protease in apoptotic cells.Targeting inflammatory pathways for tumor radiosensitization.Chapter seven--Cancer treatment with gene therapy and radiation therapy.The Binding Receptors of Aβ: an Alternative Therapeutic Target for Alzheimer's Disease.Requirement of AP-1 for ceramide-induced apoptosis in human leukemia HL-60 cells.Transcriptional activation of the small GTPase gene rhoB by genotoxic stress is regulated via a CCAAT element.Ionizing radiation induces, via generation of reactive oxygen intermediates, intercellular adhesion molecule-1 (ICAM-1) gene transcription and NF kappa B-like binding activity in the ICAM-1 transcriptional regulatory region.Demonstration of Tightly Radiation-Controlled Molecular Switch Based on CArG Repeats by In Vivo Molecular Imaging.Radiation-inducible HtrA2 gene enhances radiosensitivity of uveal melanoma OCM-1 cells in vitro and in vivo.Effect of continuous high intensity focused ultrasound in a squamous cell carcinoma tumor model compared to muscle tissue evaluated by MRI, histology, and gene expression.Regulation of tumor necrosis factor gene expression by ionizing radiation in human myeloid leukemia cells and peripheral blood monocytes.The ionizing radiation-induced replication protein A phosphorylation response differs between ataxia telangiectasia and normal human cells.Development of synthetic promoters for radiation-mediated gene therapy.
P2860
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P2860
Ionizing radiation regulates expression of the c-jun protooncogene.
description
1990 nî lūn-bûn
@nan
1990 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Ionizing radiation regulates expression of the c-jun protooncogene.
@ast
Ionizing radiation regulates expression of the c-jun protooncogene.
@en
type
label
Ionizing radiation regulates expression of the c-jun protooncogene.
@ast
Ionizing radiation regulates expression of the c-jun protooncogene.
@en
prefLabel
Ionizing radiation regulates expression of the c-jun protooncogene.
@ast
Ionizing radiation regulates expression of the c-jun protooncogene.
@en
P2093
P2860
P356
P1476
Ionizing radiation regulates expression of the c-jun protooncogene.
@en
P2093
D E Hallahan
M L Sherman
R R Weichselbaum
P2860
P304
P356
10.1073/PNAS.87.15.5663
P407
P577
1990-08-01T00:00:00Z