Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
about
Intestinal nematode infection ameliorates experimental colitis in miceTreatment of experimental (Trinitrobenzene sulfonic acid) colitis by intranasal administration of transforming growth factor (TGF)-beta1 plasmid: TGF-beta1-mediated suppression of T helper cell type 1 response occurs by interleukin (IL)-10 inductionProduction of MDC/CCL22 by human intestinal epithelial cellsIncreased sensitivity to dextran sodium sulfate colitis in IRE1beta-deficient miceLessons from genetically engineered animal models XI. Novel mouse models to study pathogenic mechanisms of Crohn's disease.Th1-type responses mediate spontaneous ileitis in a novel murine model of Crohn's disease.Role of interleukin 6 in a murine model of Crohn's ileitis: are cytokine/anticytokine strategies the future for IBD therapies?Immune therapy in inflammatory bowel disease and models of colitis.Differential alteration in intestinal epithelial cell expression of toll-like receptor 3 (TLR3) and TLR4 in inflammatory bowel disease.Pseudomonas fluorescens encodes the Crohn's disease-associated I2 sequence and T-cell superantigenRole of interleukin-22 in inflammatory bowel diseaseEtiopathogenesis of inflammatory bowel disease: the importance of the pediatric perspective.Transgenic models of autoimmune disease.An important regulatory role for CD4+CD8 alpha alpha T cells in the intestinal epithelial layer in the prevention of inflammatory bowel diseaseImmune networks in animal models of inflammatory bowel disease.Appendix is a priming site in the development of ulcerative colitis.Role of the commensal microbiota in normal and pathogenic host immune responsesSystemic antibodies towards mucosal bacteria in ulcerative colitis and Crohn's disease differentially activate the innate immune response.In vivo biomarkers for targeting colorectal neoplasms.Dependence of intestinal granuloma formation on unique myeloid DC-like cells.Helicobacter bilis infection accelerates and H. hepaticus infection delays the development of colitis in multiple drug resistance-deficient (mdr1a-/-) miceMHC-bound antigens and proteomics for novel target discovery.The membrane-bound mucin Muc1 regulates T helper 17-cell responses and colitis in miceIL-22 ameliorates intestinal inflammation in a mouse model of ulcerative colitisLayers of mutualism with commensal bacteria protect us from intestinal inflammation.Role of the high affinity immunoglobulin E receptor in bacterial translocation and intestinal inflammation.T helper 1 and T helper 2 cells are pathogenic in an antigen-specific model of colitisProbiotics for the treatment of inflammatory bowel disease.Barrier dysfunction and bacterial uptake in the follicle-associated epithelium of ileal Crohn's disease.Escherichia coli strain Nissle 1917 ameliorates experimental colitis via toll-like receptor 2- and toll-like receptor 4-dependent pathways.Transforming growth factor-beta 1 inhibits non-pathogenic Gram negative bacteria-induced NF-kappa B recruitment to the interleukin-6 gene promoter in intestinal epithelial cells through modulation of histone acetylation.IKK beta and phosphatidylinositol 3-kinase/Akt participate in non-pathogenic Gram-negative enteric bacteria-induced RelA phosphorylation and NF-kappa B activation in both primary and intestinal epithelial cell lines.NOD2/CARD15 and the Paneth cell: another piece in the genetic jigsaw of inflammatory bowel disease.Epithelial dysfunction associated with the development of colitis in conventionally housed mdr1a-/- mice.Omega-3 polyunsaturated fatty acids ameliorate the severity of ileitis in the senescence accelerated mice (SAM)P1/Yit mice model.The proton-activated receptor GPR4 modulates intestinal inflammation.Visualization of IL-22-expressing Lymphocytes Using Reporter Mice.Pharmacological effects of ba-wei-xi-lei powder on ulcerative colitis in rats with enema application.A defect in epithelial barrier integrity is not required for a systemic response to bacterial antigens or intestinal injury in T cell receptor-alpha gene-deficient mice.Evaluation of Th1-like, Th2-like and immunomodulatory cytokine mRNA expression in the skin of dogs with immunomodulatory-responsive lymphocytic-plasmacytic pododermatitis.
P2860
Q24537646-C7DB4A09-A4ED-45EC-9979-18CDA93DFC8CQ24676238-8DDF20DD-1225-4557-8450-FC7D0150BB09Q28188916-3ADAB0AF-89EA-4003-BA71-A547C011E11AQ28359871-7B272F71-BBC1-401B-9A1F-21D3A98FB913Q30305634-07DC9EC6-1FEB-4375-911D-DF5A8A0ACB8AQ30307038-F896A18B-F319-43F2-8CC4-B7FF2D34EBB1Q30438775-18211C2F-FD5A-47B2-B737-321F4B947378Q30774968-55AF4653-F50B-4666-806D-BB8503E38013Q34005313-1343D59B-131D-4E5C-9664-142A592A63D3Q34259452-61409998-67C1-4289-B831-B6536BEAEEEAQ34456769-00592ED5-6141-4623-B583-A07A5853B963Q34538014-56F810F8-E47E-4D50-9E71-BDC667F7614CQ34554029-7FA061F9-EA0A-41D1-BE4D-274A9232982EQ34982863-E725E7F8-1FEF-4B91-A950-AE902481DB6FQ35193871-95C163A4-0C94-4C64-8F16-6752E9F67AFBQ35435811-6BC74EEB-5C63-48A0-9047-DF058AB8E4E7Q35448732-8CE02535-1F7D-4A44-B6F3-CA4A551AB098Q35596242-E77F1056-10C0-48E2-94D4-3FE73E56FA70Q35596304-A8102871-C455-4934-880B-4747E566C1D1Q35635502-32CBCC79-63CD-4518-B3D8-8EE002163C24Q35747283-B585FBCE-1736-43BC-A545-9C1CDFBCF16BQ35911151-765F1734-975A-4725-92CA-BBE98028F2D4Q36231773-18A31DAB-1914-4234-A6E2-0210710A9F15Q36303708-5DEB6A4E-C8CC-4264-8CD6-356EC850EC6EQ36366156-EA356FDD-488E-4BDC-A322-4F65B723D94BQ36376117-FA7F75AB-B088-4DA5-84CD-EA245A9C94D2Q36376495-E3E97507-06E3-46DA-9859-A07C5579E002Q38009716-293D95F6-69FA-48AA-A846-490566455A60Q38021501-8312F479-49D3-42A1-9045-F77B28F7BAABQ40264330-956CB7C0-A403-485A-8278-D6DC6E5C4B7DQ40658888-3E652074-6503-4BB2-8BEF-7E5281397F94Q40715411-2A9EA2F7-B36E-4C48-854E-65B7587984ADQ42105434-F5559699-B35B-47CE-AC36-CA5A74009A42Q42475738-81DD51CD-7A45-4417-AF8E-B2443893F500Q43269061-B8BE7DED-934B-472B-A3CD-C4833A9E3878Q47405007-D810DF04-6AC0-4A24-96B5-E9E2B5F42832Q48832494-0E08FBBA-0364-4779-8A35-A157DE4EDF69Q51198302-4F8395C1-B7C8-44E6-8C14-FC7A0DD066C0Q52572938-65475E4F-1D6E-454F-B09D-7450C56CED5AQ54582437-95F674DA-5571-41C8-AC18-63DC9278796A
P2860
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@ast
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@en
type
label
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@ast
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@en
prefLabel
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@ast
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@en
P2093
P2860
P1476
Colitis in transgenic and knockout animals as models of human inflammatory bowel disease.
@en
P2093
P2860
P304
P356
10.1111/J.1600-065X.1999.TB01316.X
P577
1999-06-01T00:00:00Z