Mild heat stress stimulates 20S proteasome and its 11S activator in human fibroblasts undergoing aging in vitro.
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The proteasome and its role in the degradation of oxidized proteins.The age- and sex-specific decline of the 20s proteasome and the Nrf2/CncC signal transduction pathway in adaption and resistance to oxidative stress in Drosophila melanogasterDifferential regulation of proteasome functionality in reproductive vs. somatic tissues of Drosophila during aging or oxidative stress.Basic leucine zipper protein Cnc-C is a substrate and transcriptional regulator of the Drosophila 26S proteasome.Late-onset Alzheimer's disease, heating up and foxed by several proteins: pathomolecular effects of the aging process.Aging and dietary restriction alter proteasome biogenesis and composition in the brain and liverPrinciples and practice of hormetic treatment of aging and age-related diseases.Impairment of lon-induced protection against the accumulation of oxidized proteins in senescent wi-38 fibroblastsMechanisms of hormesis through mild heat stress on human cells.Hormesis-based anti-aging products: a case study of a novel cosmetic.The nuclear proteasome and the degradation of oxidatively damaged proteins.Hormetic modulation of aging and longevity by mild heat stress.Hormetic prevention of molecular damage during cellular aging of human skin fibroblasts and keratinocytes.Multiple mild heat-shocks decrease the Gompertz component of mortality in Caenorhabditis elegans.Targeting the age-related occurrence, removal, and accumulation of molecular damage by hormesis.Oxidative protein damage and the proteasome.Hormesis, cellular stress response and neuroinflammation in schizophrenia: Early onset versus late onset state.TLC-based detection of methylated cytosine: application to aging epigenetics.Aging intervention, prevention, and therapy through hormesis.Heat stress and hormetin-induced hormesis in human cells: effects on aging, wound healing, angiogenesis, and differentiationHormesis and Cellular Quality Control: A Possible Explanation for the Molecular Mechanisms that Underlie the Benefits of Mild Stress.Molecular mechanisms of anti-aging hormetic effects of mild heat stress on human cells.Proteasome activity or expression is not altered by activation of the heat shock transcription factor Hsf1 in cultured fibroblasts or myoblasts.Anti-ageing strategies: prevention or therapy? Showing ageing from within.Introducing Professor Stuart Calderwood, Stress Response Translational Research Section Editor.Age-related and sex-specific differences in proteasome activity in individual Drosophila flies from wild type, longevity-selected and stress resistant strains.Cellular degradation activity is maintained during aging in long-living queen bees.
P2860
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P2860
Mild heat stress stimulates 20S proteasome and its 11S activator in human fibroblasts undergoing aging in vitro.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@ast
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@en
type
label
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@ast
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@en
prefLabel
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@ast
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@en
P2093
P2860
P1476
Mild heat stress stimulates 20 ...... sts undergoing aging in vitro.
@en
P2093
Brian F C Clark
Rasmus Beedholm
Suresh I S Rattan
P2860
P356
10.1379/1466-1268(2004)009<0049:MHSSSP>2.0.CO;2
P577
2004-03-01T00:00:00Z