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Sodium and potassium balance depends on αENaC expression in connecting tubule

Sodium and potassium balance depends on αENaC expression in connecting tubule

http://www.wikidata.org/entity/Q33718954

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Epigenetics and the control of the collecting duct epithelial sodium channel Scaffold protein connector enhancer of kinase suppressor of Ras isoform 3 (CNK3) coordinates assembly of a multiprotein epithelial sodium channel (ENaC)-regulatory complex Renal intercalated cells are rather energized by a proton than a sodium pump.The glucocorticoid-induced leucine zipper (gilz/Tsc22d3-2) gene locus plays a crucial role in male fertility.Colon-specific deletion of epithelial sodium channel causes sodium loss and aldosterone resistance Aldosterone Modulates the Association between NCC and ENaC.Deletion of the angiotensin II type 1 receptor-associated protein enhances renal sodium reabsorption and exacerbates angiotensin II-mediated hypertension Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension Aldosterone regulates microRNAs in the cortical collecting duct to alter sodium transport.Genetic ablation of aquaporin-2 in the mouse connecting tubules results in defective renal water handling.The Cap1-claudin-4 regulatory pathway is important for renal chloride reabsorption and blood pressure regulation.KLHL3 regulates paracellular chloride transport in the kidney by ubiquitination of claudin-8.AT1 receptors in the collecting duct directly modulate the concentration of urine.Epithelial Sodium Channel-Mediated Sodium Transport Is Not Dependent on the Membrane-Bound Serine Protease CAP2/Tmprss4.Reducing αENaC expression in the kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading.Ion channels in renal disease.mPGES-1-derived PGE2 mediates dehydration natriuresis An Af9 cis-element directly targets Dot1a to mediate transcriptional repression of the αENaC gene Maintaining K+ balance on the low-Na+, high-K+ diet.Enhanced angiotensin receptor-associated protein in renal tubule suppresses angiotensin-dependent hypertension Impact of Angiotensin Type 1A Receptors in Principal Cells of the Collecting Duct on Blood Pressure and Hypertension.Acetazolamide Attenuates Lithium-Induced Nephrogenic Diabetes Insipidus Sp1 trans-activates and is required for maximal aldosterone induction of the αENaC gene in collecting duct cells.Aldosterone reprograms promoter methylation to regulate αENaC transcription in the collecting duct.Physical and functional interaction of Rnf2 with Af9 regulates basal and aldosterone-stimulated transcription of the α-ENaC gene in a renal collecting duct cell line.Role of adenylyl cyclase 6 in the development of lithium-induced nephrogenic diabetes insipidus.Independent regulation of Na+ and K+ balance by the kidney.Ion channel drug discovery: challenges and future directions.Blood pressure regulation via the epithelial sodium channel: from gene to kidney and beyond.Molecular physiology of the medullary collecting duct.Dietary potassium and the renal control of salt balance and blood pressure.Hypertensive epigenetics: from DNA methylation to microRNAs.Relative roles of principal and intercalated cells in the regulation of sodium balance and blood pressure.Vasopressin regulation of sodium transport in the distal nephron and collecting duct.Severe hyperkalemia is rescued by low-potassium diet in renal βENaC-deficient mice.The sodium chloride cotransporter (NCC) and epithelial sodium channel (ENaC) associate.Ion transport in the zebrafish kidney from a human disease angle: possibilities, considerations, and future perspectives.The function and regulation of acid-sensing ion channels (ASICs) and the epithelial Na(+) channel (ENaC): IUPHAR Review 19.Diurnal variation of urinary sodium-to-potassium ratio in free-living Japanese individuals.Severe acute dehydration in a desert rodent elicits a transcriptional response that effectively prevents kidney injury.

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P2860

Sodium and potassium balance depends on αENaC expression in connecting tubule

http://www.wikidata.org/entity/Q33718954

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Sodium and potassium balance depends on αENaC expression in connecting tubule

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Sodium and potassium balance depends on αENaC expression in connecting tubule

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type

label

Sodium and potassium balance depends on αENaC expression in connecting tubule

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Sodium and potassium balance depends on αENaC expression in connecting tubule

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prefLabel

Sodium and potassium balance depends on αENaC expression in connecting tubule

@ast

Sodium and potassium balance depends on αENaC expression in connecting tubule

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Journal of the American Society of Nephrology

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Sodium and potassium balance depends on αENaC expression in connecting tubule

@en

P2093

Annie Mercier Zuber

http://www.w3.org/2001/XMLSchema#string

Bernard C Rossier

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Caroline Ronzaud

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David Mordasini

http://www.w3.org/2001/XMLSchema#string

Edith Hummler

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Jean-Christophe Stehle

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Marc Maillard

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Qing Wang

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Sumedha Malsure

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P2860

Liddle's syndrome: heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel

Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1

Hypertension caused by a truncated epithelial sodium channel gamma subunit: genetic heterogeneity of Liddle syndrome

Kidney function in mice lacking aldosterone

Regulation of maturation and processing of ENaC subunits in the rat kidney

Salt restriction induces pseudohypoaldosteronism type 1 in mice expressing low levels of the beta-subunit of the amiloride-sensitive epithelial sodium channel

Disruption of the beta subunit of the epithelial Na+ channel in mice: hyperkalemia and neonatal death associated with a pseudohypoaldosteronism phenotype.

Collecting duct-specific gene inactivation of alphaENaC in the mouse kidney does not impair sodium and potassium balance.

Sodium and potassium handling by the aldosterone-sensitive distal nephron: the pivotal role of the distal and connecting tubule.

Cellular and subcellular immunolocalization of vasopressin-regulated water channel in rat kidney.

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1942-1951

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scholarly article

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10.1681/ASN.2009101077

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11

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21

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Birgitte Mønster Christensen

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2010-10-14T00:00:00Z

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20947633

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3014008

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