Placenta growth factor in sickle cell disease: association with hemolysis and inflammation.
about
Biomarkers and recent advances in the management and therapy of sickle cell diseaseHigh levels of placenta growth factor in sickle cell disease promote pulmonary hypertensionHeme-bound iron activates placenta growth factor in erythroid cells via erythroid Krüppel-like factorUrinary albumin excretion is associated with pulmonary hypertension in sickle cell disease: potential role of soluble fms-like tyrosine kinase-1.Association of coagulation activation with clinical complications in sickle cell diseaseKey endothelial cell angiogenic mechanisms are stimulated by the circulating milieu in sickle cell disease and attenuated by hydroxyureaChronic transfusion therapy improves but does not normalize systemic and pulmonary vasculopathy in sickle cell disease.Decades after the cooperative study: a re-examination of systemic blood pressure in sickle cell disease.Erythropoietin-mediated expression of placenta growth factor is regulated via activation of hypoxia-inducible factor-1α and post-transcriptionally by miR-214 in sickle cell disease.Peroxisome proliferator-activated receptor-α-mediated transcription of miR-301a and miR-454 and their host gene SKA2 regulates endothelin-1 and PAI-1 expression in sickle cell diseaseActivated Transcription Factor 3 in Association with Histone Deacetylase 6 Negatively Regulates MicroRNA 199a2 Transcription by Chromatin Remodeling and Reduces Endothelin-1 Expression.PlGF: a multitasking cytokine with disease-restricted activity.Intravascular hemolysis and the pathophysiology of sickle cell disease.PlGF enhances TLR-dependent inflammatory responses in human mononuclear phagocytes.Clinical significance of assessment of thrombospondin and placenta growth factor levels in patients with sickle cell anemia: two centers egyptian studies.Angiogenic growth factors augment K-Cl cotransporter expression in erythroid cells via hypoxia-inducible factor-1α.Ironing out placenta growth factorPlacenta growth factor mediated gene regulation in sickle cell disease.Pathways to pulmonary hypertension in sickle cell disease: the search for prevention and early intervention.Endothelin-A Receptor Antagonism Retards the Progression of Murine Sickle Cell Nephropathy.Association of soluble fms-like tyrosine kinase-1 with pulmonary hypertension and haemolysis in sickle cell disease.Monocytosis is associated with hemolysis in sickle cell disease.Beyond the definitions of the phenotypic complications of sickle cell disease: an update on management.
P2860
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P2860
Placenta growth factor in sickle cell disease: association with hemolysis and inflammation.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Placenta growth factor in sick ...... th hemolysis and inflammation.
@ast
Placenta growth factor in sick ...... th hemolysis and inflammation.
@en
type
label
Placenta growth factor in sick ...... th hemolysis and inflammation.
@ast
Placenta growth factor in sick ...... th hemolysis and inflammation.
@en
prefLabel
Placenta growth factor in sick ...... th hemolysis and inflammation.
@ast
Placenta growth factor in sick ...... th hemolysis and inflammation.
@en
P2093
P2860
P1433
P1476
Placenta growth factor in sick ...... th hemolysis and inflammation.
@en
P2093
Alan Hinderliter
Ben Hulkower
Dell Strayhorn
Eugene P Orringer
Julia E Brittain
Kenneth I Ataga
Laura De Castro
Marilyn J Telen
Susan K Jones
P2860
P304
P356
10.1182/BLOOD-2009-04-217950
P407
P577
2009-12-29T00:00:00Z