A potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake. - Wikidata - wikimedia - TriplyDB

A potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake.

A potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake.

http://www.wikidata.org/entity/Q33728140

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The Role of Hypothalamic Neuropeptides in Neurogenesis and Neuritogenesis From neuroanatomy to behavior: central integration of peripheral signals regulating feeding behavior Brain regulation of energy balance and body weight.Neuroendocrine regulation of appetitive ingestive behavior The Role of PVH Circuits in Leptin Action and Energy Balance Hindbrain noradrenergic A2 neurons: diverse roles in autonomic, endocrine, cognitive, and behavioral functions Relative number and distribution of murine hypothalamic proopiomelanocortin neurons innervating distinct target sites.Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist.The CNS glucagon-like peptide-2 receptor in the control of energy balance and glucose homeostasis The effects of beta-endorphin: state change modification.NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation.Modulation of the central melanocortin system by leptin, insulin, and serotonin: co-ordinated actions in a dispersed neuronal network.Sixteen years and counting: an update on leptin in energy balance Deficiency of PTP1B in POMC neurons leads to alterations in energy balance and homeostatic response to cold exposure.The ovarian hormone estradiol plays a crucial role in the control of food intake in females.Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors.Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.Volume transmission of beta-endorphin via the cerebrospinal fluid; a review Food intake reductions and increases in energetic responses by hindbrain leptin and melanotan II are enhanced in mice with POMC-specific PTP1B deficiency.Parabrachial CGRP Neurons Control Meal Termination.Vagal afferent NMDA receptors modulate CCK-induced reduction of food intake through synapsin I phosphorylation in adult male rats Melanocortin signaling in the brainstem influences vagal outflow to the stomach.The brain subfornical organ mediates leptin-induced increases in renal sympathetic activity but not its metabolic effects Differential control of metabolic and cardiovascular functions by melanocortin-4 receptors in proopiomelanocortin neurons.Central insulin and leptin-mediated autonomic control of glucose homeostasis.Lateral thinking about leptin: a review of leptin action via the lateral hypothalamus Hindbrain neurons as an essential hub in the neuroanatomically distributed control of energy balance.Unraveling the central proopiomelanocortin neural circuits.Molecular anatomy of the gut-brain axis revealed with transgenic technologies: implications in metabolic research.Glycemic state regulates melanocortin, but not nesfatin-1, responsiveness of glucose-sensing neurons in the nucleus of the solitary tract.Hexosamine biosynthetic pathway activity in leptin resistant sucrose-drinking rats Melanocortin-4 receptors expressed by cholinergic neurons regulate energy balance and glucose homeostasis.Acute activation of GLP-1-expressing neurons promotes glucose homeostasis and insulin sensitivity.Activation of anorexigenic pro-opiomelanocortin neurones during refeeding is independent of vagal and brainstem inputs.Subdiaphragmatic Vagotomy With Pyloroplasty Ameliorates the Obesity Caused by Genetic Deletion of the Melanocortin 4 Receptor in the Mouse.

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P2860

A potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake.

http://www.wikidata.org/entity/Q33728140

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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A potential role for hypothala ...... ed suppression of food intake.

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A potential role for hypothala ...... ed suppression of food intake.

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A potential role for hypothala ...... ed suppression of food intake.

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American Journal of Physiology - Regulatory, Integrative and Comparative Physiology

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A potential role for hypothala ...... ed suppression of food intake.

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Christopher J Rhodes

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Gwendolyn W Louis

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Harvey J Grill

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Huiyuan Zheng

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Karolina P Skibicka

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Laurel M Patterson

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Martin G Myers

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P2860

A simplified system for generating recombinant adenoviruses

Evidence that the caudal brainstem is a target for the inhibitory effect of leptin on food intake

Leptin activates anorexigenic POMC neurons through a neural network in the arcuate nucleus

Forebrain melanocortin signaling enhances the hindbrain satiety response to CCK-8.

Phenotype of neurons in the nucleus of the solitary tract that express CCK-induced activation of the ERK signaling pathway

Chronically decerebrate rats demonstrate satiation but not bait shyness.

Principles for interpreting interactions among the multiple systems that influence food intake.

The central melanocortin system and the integration of short- and long-term regulators of energy homeostasis.

Brainstem mechanisms integrating gut-derived satiety signals and descending forebrain information in the control of meal size.

Distributed neural control of energy balance: contributions from hindbrain and hypothalamus.

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Hans-Rudolf Berthoud

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20071607

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