NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
about
Renal autoregulation in health and diseaseAngiotensin-II type 1 receptor and NOX2 mediate TCF/LEF and CREB dependent WISP1 induction and cardiomyocyte hypertrophyAngiotensin II stimulates thick ascending limb superoxide production via protein kinase C(α)-dependent NADPH oxidase activationNew targets for treatment of diabetic nephropathy: what we have learned from animal models.Adenosine A2A receptor activation attenuates tubuloglomerular feedback responses by stimulation of endothelial nitric oxide synthase.Testosterone enhances tubuloglomerular feedback by increasing superoxide production in the macula densa.Enhanced expression and activity of Nox2 and Nox4 in the macula densa in ANG II-induced hypertensive mice.Oxidative stress contributes to orthopedic trauma-induced acute kidney injury in obese rats.Aldosterone stimulates superoxide production in macula densa cellsOxidative status in the macula densa modulates tubuloglomerular feedback responsiveness in angiotensin II-induced hypertensionMechanisms of carbon monoxide attenuation of tubuloglomerular feedback.The NOX toolbox: validating the role of NADPH oxidases in physiology and disease.Mechanisms of angiotensin II-enhanced connecting tubule glomerular feedback.Vitamin D and Parathyroid Hormone Relationships with Urinary Nitric Oxide Metabolites and Plasma Isoprostanes in African-Americans.Angiotensin II stimulates superoxide production in the thick ascending limb by activating NOX4.Shear stress blunts tubuloglomerular feedback partially mediated by primary cilia and nitric oxide at the macula densa.Interaction between nitric oxide and superoxide in the macula densa in aldosterone-induced alterations of tubuloglomerular feedbackInhibition of Nitric Oxide Synthase 1 Induces Salt-Sensitive Hypertension in Nitric Oxide Synthase 1α Knockout and Wild-Type MiceMechanism of inhibition of tubuloglomerular feedback by CO and cGMPOxidative stress in hypertension: role of the kidney.Combination treatment of angiotensin II type I receptor blocker and new oral iron chelator attenuates progression of nonalcoholic steatohepatitis in rats.Role of the Primary Cilia on the Macula Densa and Thick Ascending Limbs in Regulation of Sodium Excretion and Hemodynamics.Macula Densa Nitric Oxide Synthase 1β Protects against Salt-Sensitive Hypertension.Prorenin receptor (PRR)-mediated NADPH oxidase (Nox) signaling regulates VEGF synthesis under hyperglycemic condition in ARPE-19 cells.Combined treatment with dipeptidyl peptidase-4 inhibitor (sitagliptin) and angiotensin-II type 1 receptor blocker (losartan) suppresses progression in a non-diabetic rat model of steatohepatitis.Daily intake of vitamin D- or calcium-vitamin D-fortified Persian yogurt drink (doogh) attenuates diabetes-induced oxidative stress: evidence for antioxidative properties of vitamin D.
P2860
Q28082365-0F8BA98B-3E17-4BC9-93B4-4146DEB11B06Q28566875-9F8742DF-42FE-4D4C-BBCD-630E205E195CQ33967364-AE531D9C-C8F7-44E8-A082-7C6A548799BDQ34498964-68D33A2D-6921-487A-8CF8-923D8807012CQ34599952-EE395AA5-4B1F-48C9-BC18-00C565F18A63Q34610651-FF415840-E2E2-4AD8-9116-CC1A7E5C756DQ35055893-74FAE862-4A0F-4209-832E-0498A5E3527AQ35115779-FDCCFBC1-7E5F-4497-8DD6-61D4ABCA4D12Q35216795-75975B42-9C7F-463A-8E98-AA658ABBAA96Q35316853-DDFF9DA4-5C72-4370-ABD0-432583E9E447Q35975943-CDF1CA79-A469-403D-B1D9-CFC4A6F07DD2Q36060782-CC36030E-1EF0-4392-8434-0815B3C91990Q36114951-1D3C721A-60A7-40AC-9B79-B81E09FA349AQ36206119-8F7F6C75-78B0-485E-BBB3-CB4118C4D747Q36312399-C0413DC2-C80D-4761-BA28-02928C0D4D1DQ36339259-2A95A542-7808-45EF-A421-6105C1D0A267Q36593137-4FF7F139-A163-428E-9E97-374E4D005D68Q36713325-DC73CDCD-5A3F-4802-BE2A-EF23613B9AB3Q37290108-00004F08-1AC6-4DDC-A431-95A1CA5E1D7DQ37434021-75987F66-7C0D-4E56-A894-8AF1ED9596FBQ39775863-34EED7B1-A0AE-4977-86A5-93E854808668Q40998120-DBC56328-7739-4788-A9C2-8BDB41F0975EQ42371202-F6AD9F85-1375-4AC1-A941-DB9E0A89E2C7Q46316434-4812E4CB-D5DA-41DA-97F6-2F161B2130ECQ48886971-B3050DA8-E198-4E38-997C-B3AF41665444Q50856187-DBD7EA06-94E7-426F-9248-C67CB715B1AD
P2860
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@ast
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@en
type
label
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@ast
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@en
prefLabel
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@ast
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@en
P2093
P2860
P1476
NOX2 is the primary source of angiotensin II-induced superoxide in the macula densa.
@en
P2093
Haifeng Liu
Kiran Chandrashekar
Luis A Juncos
Ruisheng Liu
P2860
P304
P356
10.1152/AJPREGU.00762.2009
P577
2010-01-06T00:00:00Z