Mechanisms controlling the smooth muscle cell death in progeria via down-regulation of poly(ADP-ribose) polymerase 1.
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Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cellsAdvancing cardiovascular tissue engineeringInterruption of progerin-lamin A/C binding ameliorates Hutchinson-Gilford progeria syndrome phenotypeDNA repair defects and genome instability in Hutchinson-Gilford Progeria Syndrome.Nesprin-2-dependent ERK1/2 compartmentalisation regulates the DNA damage response in vascular smooth muscle cell ageingPoly(ADP-ribose) protects vascular smooth muscle cells from oxidative DNA damage.Loss of H3K9me3 Correlates with ATM Activation and Histone H2AX Phosphorylation Deficiencies in Hutchinson-Gilford Progeria SyndromeReprogramming progeria fibroblasts re-establishes a normal epigenetic landscape.All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype.CD38 Dictates Age-Related NAD Decline and Mitochondrial Dysfunction through an SIRT3-Dependent Mechanism.Hutchinson-Gilford progeria syndrome as a model for vascular aging.Disruption of PCNA-lamins A/C interactions by prelamin A induces DNA replication fork stalling.Methylene blue alleviates nuclear and mitochondrial abnormalities in progeria.NAD and the aging process: Role in life, death and everything in between.Progerin expression disrupts critical adult stem cell functions involved in tissue repair.Vitamin D receptor signaling improves Hutchinson-Gilford progeria syndrome cellular phenotypes.A Tissue Engineered Blood Vessel Model of Hutchinson-Gilford Progeria Syndrome Using Human iPSC-derived Smooth Muscle CellsRoles of Cross-Membrane Transport and Signaling in the Maintenance of Cellular Homeostasis.Shared molecular and cellular mechanisms of premature ageing and ageing-associated diseases.Human induced pluripotent stem cell-derived vascular smooth muscle cells: differentiation and therapeutic potential.Pathological modelling of pigmentation disorders associated with Hutchinson-Gilford Progeria Syndrome (HGPS) revealed an impaired melanogenesis pathway in iPS-derived melanocytes.
P2860
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P2860
Mechanisms controlling the smooth muscle cell death in progeria via down-regulation of poly(ADP-ribose) polymerase 1.
description
2014 nî lūn-bûn
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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
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2014年论文
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name
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@ast
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@en
type
label
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@ast
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@en
prefLabel
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@ast
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@en
P2093
P2860
P356
P1476
Mechanisms controlling the smo ...... poly(ADP-ribose) polymerase 1.
@en
P2093
Haoyue Zhang
Zheng-Mei Xiong
P2860
P304
P356
10.1073/PNAS.1320843111
P407
P577
2014-05-19T00:00:00Z