Placental insufficiency results in temporal alterations in the renin angiotensin system in male hypertensive growth restricted offspring. - Wikidata - wikimedia - TriplyDB

Placental insufficiency results in temporal alterations in the renin angiotensin system in male hypertensive growth restricted offspring.

Placental insufficiency results in temporal alterations in the renin angiotensin system in male hypertensive growth restricted offspring.

http://www.wikidata.org/entity/Q33736611

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Fetal programming and early identification of newborns at high risk of free radical-mediated diseases Gender differences in developmental programming of cardiovascular diseases How the kidney is impacted by the perinatal maternal environment to develop hypertension Sex differences in the developmental programming of hypertension.Role of the kidney in the prenatal and early postnatal programming of hypertension Early renal denervation prevents development of hypertension in growth-restricted offspring Estrogen protects against increased blood pressure in postpubertal female growth restricted offspring.Enhanced sensitivity to acute angiotensin II is testosterone dependent in adult male growth-restricted offspring Effect of prenatal dexamethasone on postnatal serum and urinary angiotensin II levels Changes in amniotic fluid and umbilical cord serum proteomic profiles of foetuses with intrauterine growth retardation.Childhood kidney outcomes in relation to fetal blood flow and kidney size.Fetal and infant growth patterns and kidney function at school age.Low birth weight activates the renin-angiotensin system, but limits cardiac angiogenesis in early postnatal life.Hypersensitivity to acute ANG II in female growth-restricted offspring is exacerbated by ovariectomy.Maternal protein restriction reduces expression of angiotensin I-converting enzyme 2 in rat placental labyrinth zone in late pregnancy.Prenatal programming of hypertension in the rat: effect of postnatal rearing Pre-eclampsia and offspring cardiovascular health: mechanistic insights from experimental studies.Protein restriction to pregnant rats increases the plasma levels of angiotensin II and expression of angiotensin II receptors in uterine arteries.Transient Exposure of Enalapril Normalizes Prenatal Programming of Hypertension and Urinary Angiotensinogen Excretion.Sex-Specific Effect of Endothelin in the Blood Pressure Response to Acute Angiotensin II in Growth-Restricted Rats.Developmental programming of hypertension and kidney disease Intrauterine growth restriction: fetal programming of hypertension and kidney disease.Fetal programming and cardiovascular pathology.Sex differences in the enhanced responsiveness to acute angiotensin II in growth-restricted rats: role of fasudil, a Rho kinase inhibitor.Sex differences in the fetal programming of hypertension.Developmental programming of hypertension: insight from animal models of nutritional manipulation.Alterations in circulatory and renal angiotensin-converting enzyme and angiotensin-converting enzyme 2 in fetal programmed hypertension Sex differences in the developmental origins of hypertension and cardiorenal disease.Role of fetal programming in the development of hypertension.Sex differences in the developmental origins of cardiovascular disease.Epigenetics and developmental programming of adult onset diseases.Ouabain rescues rat nephrogenesis during intrauterine growth restriction by regulating the complement and coagulation cascades and calcium signaling pathway.Prenatal hypoxia leads to hypertension, renal renin-angiotensin system activation and exacerbates salt-induced pathology in a sex-specific manner.In Utero Origins of Hypertension: Mechanisms and Targets for Therapy.Role of renal sympathetic nerve activity in prenatal programming of hypertension.Intrauterine growth restriction programs an accelerated age-related increase in cardiovascular risk in male offspring.Prenatal nutrition: a critical window of opportunity for mother and child.Influence of birth weight on the renal development and kidney diseases in adulthood: experimental and clinical evidence.Activation of the sympathetic nervous system, is it key to the developmental origins of enhanced cardiovascular risk?Birth weight is inversely associated with blood pressure and serum aldosterone and cortisol levels in children.

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P2860

Placental insufficiency results in temporal alterations in the renin angiotensin system in male hypertensive growth restricted offspring.

http://www.wikidata.org/entity/Q33736611

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2007 nî lūn-bûn

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Placental insufficiency result ...... e growth restricted offspring.

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Placental insufficiency result ...... e growth restricted offspring.

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Antoinette S Dawson

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Barbara T Alexander

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Contrina A Huffman

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Daniela Grigore

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Elliot B Robertson

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Erick A Bourassa

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K Bridget Brosnihan

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Robert C Speth

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P2860

Renin and angiotensinogen gene expression in maturing rat kidney.

Effects of AT1 receptor blockade on renal injury and mitogen-activated protein activity in Dahl salt-sensitive rats.

Androgen-dependent regulation of human angiotensinogen expression in KAP-hAGT transgenic mice.

Prenatal programming of adult hypertension in the rat.

Growth in utero and during childhood among women who develop coronary heart disease: longitudinal study.

An intact renin-angiotensin system is a prerequisite for normal renal development.

Fetal origins of adult hypertension: a renal mechanism?

The role of size at birth and postnatal catch-up growth in determining systolic blood pressure: a systematic review of the literature.

Sympathetic nervous system and the kidney in hypertension.

Intrarenal oxidative stress and augmented angiotensinogen are precedent to renal injury in Zucker diabetic fatty rats.

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