alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
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Osmotic homeostasisGenetic ablation of aquaporin-2 in the mouse connecting tubules results in defective renal water handling.Effects of chronic lithium administration on renal acid excretion in humans and rats.Absence of PKC-alpha attenuates lithium-induced nephrogenic diabetes insipidus.AT1 receptors in the collecting duct directly modulate the concentration of urine.Attenuation of lithium-induced natriuresis and kaliuresis in P2Y₂ receptor knockout miceClopidogrel attenuates lithium-induced alterations in renal water and sodium channels/transporters in mice.Long-Term Lithium Use and Risk of Renal and Upper Urinary Tract Cancers.Regulation of transport in the connecting tubule and cortical collecting ductNephrogenic diabetes insipidus: essential insights into the molecular background and potential therapies for treatment.Lithium: a versatile tool for understanding renal physiology.Acetazolamide Attenuates Lithium-Induced Nephrogenic Diabetes InsipidusLithium causes G2 arrest of renal principal cellsLithium induces microcysts and polyuria in adolescent rat kidney independent of cyclooxygenase-2.Lithium in the Kidney: Friend and Foe?Lithium-induced NDI: acetazolamide reduces polyuria but does not improve urine concentrating ability.What we need to know about the effect of lithium on the kidney.Molecular mechanisms in lithium-associated renal disease: a systematic review.The function and regulation of acid-sensing ion channels (ASICs) and the epithelial Na(+) channel (ENaC): IUPHAR Review 19.Development and Diseases of the Collecting Duct System.EGF Receptor Inhibition by Erlotinib Increases Aquaporin 2-Mediated Renal Water Reabsorption.Genetic deletion of the P2Y2 receptor offers significant resistance to development of lithium-induced polyuria accompanied by alterations in PGE2 signaling.Hydrochlorothiazide attenuates lithium-induced nephrogenic diabetes insipidus independently of the sodium-chloride cotransporter.Tissue injury after lithium treatment in human and rat postnatal kidney involves glycogen synthase kinase-3β-positive epithelium.Lithium increases ammonium excretion leading to altered urinary acid-base buffer composition.Lithium reduces aquaporin-2 transcription independent of prostaglandins.
P2860
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P2860
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@ast
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@en
type
label
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@ast
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@en
prefLabel
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@ast
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@en
P2093
P2860
P356
P1476
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
@en
P2093
Bernard C Rossier
Birgitte Mønster Christensen
Edith Hummler
Jean-Christophe Stehle
Johannes Loffing
Peter M T Deen
P2860
P304
P356
10.1681/ASN.2010070734
P577
2010-11-04T00:00:00Z