Dystonia in neurodegeneration with brain iron accumulation: outcome of bilateral pallidal stimulation. - Wikidata - wikimedia - TriplyDB

Dystonia in neurodegeneration with brain iron accumulation: outcome of bilateral pallidal stimulation.

Dystonia in neurodegeneration with brain iron accumulation: outcome of bilateral pallidal stimulation.

http://www.wikidata.org/entity/Q33742432

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Dystonic opisthotonus: a "red flag" for neurodegeneration with brain iron accumulation syndromes?Differences in globus pallidus neuronal firing rates and patterns relate to different disease biology in children with dystonia.Deep Brain Stimulation in Huntington's Disease-Preliminary Evidence on Pathophysiology, Efficacy and Safety Neurodegeneration with brain iron accumulation: diagnosis and management Short and long term outcome of bilateral pallidal stimulation in chorea-acanthocytosis Open-Label Fosmetpantotenate, a Phosphopantothenate Replacement Therapy in a Single Patient with Atypical PKAN.Deep brain stimulation for pantothenate kinase-associated neurodegeneration.The "curved lead pathway" method to enable a single lead to reach any two intracranial targets.Childhood disorders of neurodegeneration with brain iron accumulation (NBIA).Dystonia rating scales: critique and recommendations Deep brain stimulation: current and future clinical applications.Deep Brain Stimulation in Neurological and Psychiatric Disorders Clinical Heterogeneity of Atypical Pantothenate Kinase-Associated Neurodegeneration in Koreans.Neuropsychological, neuropsychiatric, and quality of life issues in DBS for dystonia.Syndromes of neurodegeneration with brain iron accumulation (NBIA): an update on clinical presentations, histological and genetic underpinnings, and treatment considerations.Treatment for dystonia in childhood.Pediatric indications for deep brain stimulation.Surgical treatment for secondary dystonia.Excess iron harms the brain: the syndromes of neurodegeneration with brain iron accumulation (NBIA).Pathophysiology and treatment of neurodegeneration with brain iron accumulation in the pediatric population.Pantothenate kinase-associated neurodegeneration (PKAN) and PLA2G6-associated neurodegeneration (PLAN): review of two major neurodegeneration with brain iron accumulation (NBIA) phenotypes.Treatment of Wilson's disease motor complications with deep brain stimulation.Defective lipid metabolism in neurodegeneration with brain iron accumulation (NBIA) syndromes: not only a matter of iron.Childhood dystonias.Subthalamic Nuclei Stimulation in Patients With Pantothenate Kinase-Associated Neurodegeneration (PKAN).Clinical rating scale for pantothenate kinase-associated neurodegeneration: A pilot study.DBS in Dystonia and Other Hyperkinetic Movement Disorders.Deep brain stimulation in the globus pallidus compensates response inhibition deficits: evidence from pantothenate kinase-associated neurodegeneration.Genetics and Pathophysiology of Neurodegeneration with Brain Iron Accumulation (NBIA).Deep brain stimulation improves quality of life in pantothenate kinase-associated neurodegeneration.Proportion of life lived with dystonia inversely correlates with response to pallidal deep brain stimulation in both primary and secondary childhood dystonia.Pallidal stimulation in atypical pantothenate kinase-associated neurodegeneration: six-year follow-up.Mitochondrial membrane protein associated neurodegenration: a novel variant of neurodegeneration with brain iron accumulation.Brain stimulation in Huntington's disease.Rechargeable deep brain stimulators in the management of paediatric dystonia: well tolerated with a low complication rate.A new phenotype of brain iron accumulation with dystonia, optic atrophy, and peripheral neuropathy.Patient perspectives on the efficacy of a new kind of rechargeable deep brain stimulators(1).Clinical outcome of deep brain stimulation for dystonia: constant-current or constant-voltage stimulation? A non-randomized study.Reply: bilateral pallidal stimulation for Wilson's disease.Deep brain stimulation in the ventrolateral thalamus/subthalamic area in dystonia with head tremor.

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P2860

Dystonia in neurodegeneration with brain iron accumulation: outcome of bilateral pallidal stimulation.

http://www.wikidata.org/entity/Q33742432

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Dystonia in neurodegeneration ...... ilateral pallidal stimulation.

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Dystonia in neurodegeneration ...... ilateral pallidal stimulation.

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Dystonia in neurodegeneration ...... bilateral pallidal stimulation

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C A Haenggeli

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D Woitalla

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E Moro

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F Valldeoriola

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K A M Pauls

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K L Leenders

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P2860

A novel pantothenate kinase gene (PANK2) is defective in Hallervorden-Spatz syndrome

Clinical heterogeneity of neurodegeneration with brain iron accumulation (Hallervorden-Spatz syndrome) and pantothenate kinase-associated neurodegeneration

Clinical and genetic delineation of neurodegeneration with brain iron accumulation

Hallervorden spatz disease: MR and pathological findings of a rare case.

Neurodegeneration with brain iron accumulation.

Mutation in the gene encoding ferritin light polypeptide causes dominant adult-onset basal ganglia disease.

Hallervorden-Spatz syndrome.

Genetic, clinical, and radiographic delineation of Hallervorden-Spatz syndrome.

Bilateral deep-brain stimulation of the globus pallidus in primary generalized dystonia.

PLA2G6, encoding a phospholipase A2, is mutated in neurodegenerative disorders with high brain iron.

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10.1093/BRAIN/AWQ022

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Pt 3

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133

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Alfons Schnitzler

Patrick Santens

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41808335

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20207700

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neurodegeneration with brain iron accumulation

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2842517

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