Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrity - Wikidata - wikimedia - TriplyDB

Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrity

Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrity

http://www.wikidata.org/entity/Q33753767

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The role of neutrophils in immune dysfunction during severe inflammation Neutrophil Integrins and Matrix Ligands and NET Release Circulating neutrophil counts and mortality in septic shock.Properdin levels in human sepsis.Blood cells and endothelial barrier function.Regulation of autophagy by the nuclear factor κB signaling pathway in the hippocampus of rats with sepsis.Low molecular weight heparin prevents lipopolysaccharide induced-hippocampus-dependent cognitive impairments in mice.Streptococcus pneumoniae disrupts pulmonary immune defence via elastase release following pneumolysin-dependent neutrophil lysis.Nur77 attenuates endothelin-1 expression via downregulation of NF-κB and p38 MAPK in A549 cells and in an ARDS rat model.The mercurial nature of neutrophils: still an enigma in ARDS?ECIS technology reveals that monocytes isolated by CD14+ve selection mediate greater loss of BBB integrity than untouched monocytes, which occurs to a greater extent with IL-1β activated endothelium in comparison to TNFα.Extracellular granzyme K mediates endothelial activation through the cleavage of protease-activated receptor-1.Regulation of human cerebro-microvascular endothelial baso-lateral adhesion and barrier function by S1P through dual involvement of S1P1 and S1P2 receptors.Pathogenic, immunologic, and clinical aspects of sepsis - update 2016.The Use of Redox Expression and Associated Molecular Damage to Evaluate the Inflammatory Response in Critically Ill Patient with Severe Burn.Urinary trypsin inhibitor attenuates LPS-induced endothelial barrier dysfunction by upregulation of vascular endothelial-cadherin expression.Septicemia is associated with increased risk for dementia: a population-based longitudinal study.Shear resistance of endothelial cells in a pathological environment.Endothelial biomarkers in human sepsis: pathogenesis and prognosis for ARDS.Leukadherin-1 ameliorates endothelial barrier damage mediated by neutrophils from critically ill patients.Neutrophils in Tissue Trauma of the Skin, Bone, and Lung: Two Sides of the Same Coin.

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Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrity

http://www.wikidata.org/entity/Q33753767

description

2013 nî lūn-bûn

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2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Neutrophils from critically il ...... endothelial barrier integrity

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Neutrophils from critically il ...... endothelial barrier integrity

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type

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Neutrophils from critically il ...... endothelial barrier integrity

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Neutrophils from critically il ...... endothelial barrier integrity

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Neutrophils from critically il ...... endothelial barrier integrity

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Neutrophils from critically il ...... endothelial barrier integrity

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Neutrophils from critically il ...... endothelial barrier integrity

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Elizabeth D Fox

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Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

CAP37, a neutrophil-derived inflammatory mediator, augments leukocyte adhesion to endothelial monolayers

Randomized phase I/II trial of a macrophage-specific immunomodulator (PGG-glucan) in high-risk surgical patients

Time course of endothelial damage in septic shock: prediction of outcome.

Inflection points in sepsis biology: from local defense to systemic organ injury

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Effect of PGG-glucan on the rate of serious postoperative infection or death observed after high-risk gastrointestinal operations. Betafectin Gastrointestinal Study Group

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Getting to the site of inflammation: the leukocyte adhesion cascade updated

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