Inactivation of the gbpA gene of Streptococcus mutans increases virulence and promotes in vivo accumulation of recombinations between the glucosyltransferase B and C genes.
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Distribution of putative virulence genes in Streptococcus mutans strains does not correlate with caries experienceQuantitative analyses of Streptococcus mutans biofilms with quartz crystal microbalance, microjet impingement and confocal microscopyTrigger factor in Streptococcus mutans is involved in stress tolerance, competence development, and biofilm formationBiofilm formation and virulence expression by Streptococcus mutans are altered when grown in dual-species modelLigand-binding properties of the carboxyl-terminal repeat domain of Streptococcus mutans glucan-binding protein A.Inactivation of the gbpA gene of Streptococcus mutans alters structural and functional aspects of plaque biofilm which are compensated by recombination of the gtfB and gtfC genes.Stress-induced membrane association of the Streptococcus mutans GTP-binding protein, an essential G protein, and investigation of its physiological role by utilizing an antisense RNA strategy.Evidence that accumulation of mutants in a biofilm reflects natural selection rather than stress-induced adaptive mutation.Transcriptional repressor Rex is involved in regulation of oxidative stress response and biofilm formation by Streptococcus mutans.Effect of an orphan response regulator on Streptococcus mutans sucrose-dependent adherence and cariogenesisStreptococcus mutans glucan-binding protein-A affects Streptococcus gordonii biofilm architectureBiological and Immunogenicity Property of IgY Anti S. mutans ComDFunctional analysis of glucan binding protein B from Streptococcus mutans.Recombination between gtfB and gtfC is required for survival of a dTDP-rhamnose synthesis-deficient mutant of Streptococcus mutans in the presence of sucrose.Functional genomics approach to identifying genes required for biofilm development by Streptococcus mutans.Cariogenicity of Streptococcus mutans glucan-binding protein deletion mutants.Psr is involved in regulation of glucan production, and double deficiency of BrpA and Psr is lethal in Streptococcus mutans.Glucan-binding proteins are essential for shaping Streptococcus mutans biofilm architecture.Manganese affects Streptococcus mutans virulence gene expression.Regulation of recombination between gtfB/gtfC genes in Streptococcus mutans by recombinase A.
P2860
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P2860
Inactivation of the gbpA gene of Streptococcus mutans increases virulence and promotes in vivo accumulation of recombinations between the glucosyltransferase B and C genes.
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@ast
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@en
type
label
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@ast
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@en
prefLabel
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@ast
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@en
P2093
P2860
P1476
Inactivation of the gbpA gene ...... osyltransferase B and C genes.
@en
P2093
K R Hazlett
S M Michalek
P2860
P304
P407
P577
1998-05-01T00:00:00Z