Conserved cysteine residues within the attachment G glycoprotein of respiratory syncytial virus play a critical role in the enhancement of cytotoxic T-lymphocyte responses - Wikidata - wikimedia - TriplyDB

Conserved cysteine residues within the attachment G glycoprotein of respiratory syncytial virus play a critical role in the enhancement of cytotoxic T-lymphocyte responses

Conserved cysteine residues within the attachment G glycoprotein of respiratory syncytial virus play a critical role in the enhancement of cytotoxic T-lymphocyte responses

http://www.wikidata.org/entity/Q33754172

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The respiratory syncytial virus G protein conserved domain induces a persistent and protective antibody response in rodents Prophylaxis with a respiratory syncytial virus (RSV) anti-G protein monoclonal antibody shifts the adaptive immune response to RSV rA2-line19F infection from Th2 to Th1 in BALB/c mice.Nanoparticle vaccines encompassing the respiratory syncytial virus (RSV) G protein CX3C chemokine motif induce robust immunity protecting from challenge and disease.Functional Analysis of the 60-Nucleotide Duplication in the Respiratory Syncytial Virus Buenos Aires Strain Attachment Glycoprotein.The Central Conserved Region (CCR) of Respiratory Syncytial Virus (RSV) G Protein Modulates Host miRNA Expression and Alters the Cellular Response to Infection.Modulation of the host immune response by respiratory syncytial virus proteins.Mutating the CX3C Motif in the G Protein Should Make a Live Respiratory Syncytial Virus Vaccine Safer and More Effective.Respiratory syncytial virus G protein CX3C motif impairs human airway epithelial and immune cell responses.

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P2860

Conserved cysteine residues within the attachment G glycoprotein of respiratory syncytial virus play a critical role in the enhancement of cytotoxic T-lymphocyte responses

http://www.wikidata.org/entity/Q33754172

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2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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type

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Conserved cysteine residues wi ...... totoxic T-lymphocyte responses

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Ana C Monsalvo

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Dowd Bridget

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Federico F Laham

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Fernando P Polack

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Guillermina A Melendi

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Maria Florencia Delgado

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Pablo M Irusta

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Patricio Acosta

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P2860

CX3CR1-mediated dendritic cell access to the intestinal lumen and bacterial clearance

Induction of Th-1 and Th-2 responses by respiratory syncytial virus attachment glycoprotein is epitope and major histocompatibility complex independent.

Respiratory syncytial virus infection suppresses lung CD8+ T-cell effector activity and peripheral CD8+ T-cell memory in the respiratory tract.

Virus-specific CD8+ T lymphocytes downregulate T helper cell type 2 cytokine secretion and pulmonary eosinophilia during experimental murine respiratory syncytial virus infection.

A previously unrecognized H-2D(b)-restricted peptide prominent in the primary influenza A virus-specific CD8(+) T-cell response is much less apparent following secondary challenge.

The cysteine-rich region of respiratory syncytial virus attachment protein inhibits innate immunity elicited by the virus and endotoxin

Respiratory syncytial virus and parainfluenza virus.

Anti-IL-4 treatment at immunization modulates cytokine expression, reduces illness, and increases cytotoxic T lymphocyte activity in mice challenged with respiratory syncytial virus.

Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice.

Lack of antibody affinity maturation due to poor Toll-like receptor stimulation leads to enhanced respiratory syncytial virus disease

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1

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