IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
about
Enigma of IL-17 and Th17 Cells in Rheumatoid Arthritis and in Autoimmune Animal Models of ArthritisA cytokine-centric view of the pathogenesis and treatment of autoimmune arthritisProteoglycan aggrecan conducting T cell activation and apoptosis in a murine model of rheumatoid arthritisViolacein Treatment Modulates Acute and Chronic Inflammation through the Suppression of Cytokine Production and Induction of Regulatory T CellsExposure to Candida albicans polarizes a T-cell driven arthritis model towards Th17 responses, resulting in a more destructive arthritisLocation of CD4+ T cell priming regulates the differentiation of Th1 and Th17 cells and their contribution to arthritis.Commercial bovine proteoglycan is highly arthritogenic and can be used as an alternative antigen source for PGIA model.The immunomodulator AS101 suppresses production of inflammatory cytokines and ameliorates the pathogenesis of experimental autoimmune encephalomyelitisBrief report: Autologous stem cell transplantation restores immune tolerance in experimental arthritis by renewal and modulation of the Teff cell compartmentImpaired activation-induced cell death promotes spontaneous arthritis in antigen (cartilage proteoglycan)-specific T cell receptor-transgenic miceTissue specific CD4+ T cell priming determines the requirement for interleukin-23 in experimental arthritisMechanisms of NDV-3 vaccine efficacy in MRSA skin versus invasive infection.Proteoglycan-induced arthritis and recombinant human proteoglycan aggrecan G1 domain-induced arthritis in BALB/c mice resembling two subtypes of rheumatoid arthritis.T-bet controls severity of hypersensitivity pneumonitisB cell depletion enhances T regulatory cell activity essential in the suppression of arthritis.B cells expressing IFN-γ suppress Treg-cell differentiation and promote autoimmune experimental arthritis.The Additive Inflammatory In Vivo and In Vitro Effects of IL-7 and TSLP in Arthritis Underscore the Therapeutic Rationale for Dual BlockadeInterferon-γ regulates discordant mechanisms of uveitis versus joint and axial disease in a murine model resembling spondylarthritis.Neutralization of IL-17 ameliorates uveitis but damages photoreceptors in a murine model of spondyloarthritis.Anti-Arthritic Activity of Schistosoma mansoni and Trichinella spiralis Derived-Antigens in Adjuvant Arthritis in Rats: Role of FOXP3+ Treg Cells.Rheumatoid arthritis vaccine therapies: perspectives and lessons from therapeutic ligand epitope antigen presentation system vaccines for models of rheumatoid arthritisExcessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitorsAdvances in rheumatoid arthritis animal models.Are Th17 cells an appropriate new target in the treatment of rheumatoid arthritis?Effector T cells in rheumatoid arthritis: lessons from animal models.T-bet in disease.Therapeutic Potential of Interferon-γ and Its Antagonists in Autoinflammation: Lessons from Murine Models of Systemic Juvenile Idiopathic Arthritis and Macrophage Activation Syndrome.An epitope-specific DerG-PG70 LEAPS vaccine modulates T cell responses and suppresses arthritis progression in two related murine models of rheumatoid arthritisMHC class II alleles associated with Th1 rather than Th17 type immunity drive the onset of early arthritis in a rat model of rheumatoid arthritis.Immunomodulation of autoimmune arthritis by pro-inflammatory cytokines.Cutting edge: the pathogenicity of IFN-γ-producing Th17 cells is independent of T-bet.Borrelia-primed and -infected mice deficient of interleukin-17 develop arthritis after neutralization of gamma-interferon.Arthritis is developed in Borrelia-primed and -infected mice deficient of interleukin-17.Exacerbated egg-induced immunopathology in murine Schistosoma mansoni infection is primarily mediated by IL-17 and restrained by IFN-γ.The roles of IFN-γ versus IL-17 in pathogenic effects of human Th17 cells on synovial fibroblasts.Mesenchymal stem cell therapy in proteoglycan induced arthritis.T-bet over-expression regulates aryl hydrocarbon receptor-mediated T helper type 17 differentiation through an interferon (IFN)γ-independent pathway.Invariant NKT cells promote skin wound healing by preventing a prolonged neutrophilic inflammatory response.
P2860
Q26768254-34C6200E-DBB2-4EFF-96D6-5ED62A9A4CBFQ26823326-87697380-534F-410F-80F2-BA47401C80D6Q27014005-3E7A31E6-5453-48B6-99E5-59C1E4C84DC6Q28546985-95245DD2-2DA4-4539-BADB-F5DD77FD871EQ28728570-0FCD0FDC-7EB7-4945-8E4A-64111869F045Q30412824-3BDA1766-F9F9-433C-9EC5-D74150624F21Q33757939-B42400CD-9BD5-407F-8974-BE944AC298CBQ33963074-B743815A-9E9E-4D1D-8F22-28FF330E1C30Q34039955-054961D1-39A1-42B3-A52C-18FFD33A4322Q34185573-11DDD569-FF59-4D30-8309-7A871B3A20C5Q34375258-CE0EB95B-E517-4677-8343-7A224F3D7E64Q34792962-CB0298BA-0969-42FC-823B-37A6686882B3Q34917019-0469540D-6D30-4D5F-A318-69128B09D85FQ35090644-D28AAD2F-3648-489A-86A1-0E39F137EA84Q35623403-353F9AF0-DDE8-4F50-A76D-02CA27D16F91Q35627016-D7A543D1-8C09-4AD7-9704-9DA4C656B316Q35674548-CB256A95-ED18-4D64-997C-F5F52E15371DQ35730023-66E8E253-8577-42AF-B307-9DEF13CC40EEQ36084943-C55C99F3-EB59-40A0-BE7B-4CBEFFE30522Q36179435-B683C3A7-19E2-4159-81A8-6E2B5F8214DAQ36673061-A46D571E-AEE2-4FED-88D8-5610FF23043DQ36907665-CCC4C658-805F-4A51-AB85-F09380869E83Q37205119-7F381F30-1F11-4ECE-B4A0-DD92105CD24CQ37821982-E40097A2-BC1E-43D5-85EA-BFD68F8FEF06Q37868325-8190B081-5E70-4254-98A6-F09425C177C9Q37891668-9C66B53B-9B26-4569-AFB2-6BAC5B85A2A9Q38648790-1CE13BC1-A921-4077-8B87-DE7DA62D9991Q38674546-6B830623-73F5-46AC-A93A-9DEBFFBEFF43Q39067148-68B25A9B-4D31-4865-AEEE-4C6AE0AC89CBQ39259474-637FBD24-82E4-424F-BEE5-8D409DB685B7Q40021485-BEA35E60-135E-42CD-A5F7-6BC90B0E314EQ40340934-3DF812BC-1D33-45AE-B546-31A67F13B4A2Q40587751-29827F80-3879-42F8-AF16-FDCCD0E257A3Q41941686-3C78AEC2-9ABB-4F3C-AEFD-6B044FC50A3AQ42129662-1DC503F8-33E7-4A62-B416-24C0BA0898CAQ42233976-13E9300C-E755-435B-AC05-F1917AFC404FQ47785882-467BE216-201D-443C-BEE0-C097386E173DQ49898897-4AE7F528-734F-404D-AC39-4ECF7F2B7BD9
P2860
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@ast
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@en
type
label
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@ast
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@en
prefLabel
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@ast
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@en
P2093
P2860
P356
P1476
IFN-gamma regulates the requirement for IL-17 in proteoglycan-induced arthritis.
@en
P2093
Alison Finnegan
Keith M Hamel
Paul D Doodes
Rachel L Rodeghero
Tibor T Glant
Yanxia Cao
Yumei Wang
P2860
P304
P356
10.4049/JIMMUNOL.0902907
P407
P577
2009-12-18T00:00:00Z