Gender dimorphic formation of mouse Mallory-Denk bodies and the role of xenobiotic metabolism and oxidative stress
about
A Guide to Non-Alcoholic Fatty Liver Disease in Childhood and AdolescenceThe relevance of liver histology to predicting clinically meaningful outcomes in nonalcoholic steatohepatitis.Keratin hypersumoylation alters filament dynamics and is a marker for human liver disease and keratin mutation.Transition between acute and chronic hepatotoxicity in mice is associated with impaired energy metabolism and induction of mitochondrial heme oxygenase-1.Energy determinants GAPDH and NDPK act as genetic modifiers for hepatocyte inclusion formation.Renal Impairment with Sublethal Tubular Cell Injury in a Chronic Liver Disease Mouse ModelRb and p53 Liver Functions Are Essential for Xenobiotic Metabolism and Tumor Suppression.Keratin 8 phosphorylation regulates its transamidation and hepatocyte Mallory-Denk body formation.Association between puberty and features of nonalcoholic fatty liver disease.Oxidative stress, Nrf2 and keratin up-regulation associate with Mallory-Denk body formation in mouse erythropoietic protoporphyriaAging modulates susceptibility to mouse liver Mallory-Denk body formationAmbient Light Promotes Selective Subcellular Proteotoxicity after Endogenous and Exogenous Porphyrinogenic StressMallory-Denk bodies are associated with outcomes and histologic features in patients with chronic hepatitis C.Absence of keratin 8 or 18 promotes antimitochondrial autoantibody formation in aging male miceWnt/β-catenin signaling protects mouse liver against oxidative stress-induced apoptosis through the inhibition of forkhead transcription factor FoxO3.Lamin aggregation is an early sensor of porphyria-induced liver injuryCD73 (ecto-5'-nucleotidase) hepatocyte levels differ across mouse strains and contribute to mallory-denk body formation.Keratin 18-deficiency results in steatohepatitis and liver tumors in old mice: A model of steatohepatitis-associated liver carcinogenesis.The cytoskeleton in nonalcoholic steatohepatitis: 100 years old but still youthful.Xenobiotic-induced liver injury and fibrosis.Broad spectrum of hepatocyte inclusions in humans, animals, and experimental models.Modulation of cytoskeletal dynamics by mammalian nucleoside diphosphate kinase (NDPK) proteinsLoss of keratin 19 favours the development of cholestatic liver disease through decreased ductular reaction.
P2860
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P2860
Gender dimorphic formation of mouse Mallory-Denk bodies and the role of xenobiotic metabolism and oxidative stress
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Gender dimorphic formation of ...... etabolism and oxidative stress
@ast
Gender dimorphic formation of ...... etabolism and oxidative stress
@en
type
label
Gender dimorphic formation of ...... etabolism and oxidative stress
@ast
Gender dimorphic formation of ...... etabolism and oxidative stress
@en
prefLabel
Gender dimorphic formation of ...... etabolism and oxidative stress
@ast
Gender dimorphic formation of ...... etabolism and oxidative stress
@en
P2093
P2860
P1433
P1476
Gender dimorphic formation of ...... etabolism and oxidative stress
@en
P2093
Elizabeth M Brunt
M Bishr Omary
Natasha T Snider
Paul F Hollenberg
Shinichiro Hanada
P2860
P304
P356
10.1053/J.GASTRO.2009.12.055
P407
P577
2010-01-11T00:00:00Z