Vitamin C transporter Slc23a1 links renal reabsorption, vitamin C tissue accumulation, and perinatal survival in mice. - Wikidata - wikimedia - TriplyDB

Vitamin C transporter Slc23a1 links renal reabsorption, vitamin C tissue accumulation, and perinatal survival in mice.

Vitamin C transporter Slc23a1 links renal reabsorption, vitamin C tissue accumulation, and perinatal survival in mice.

http://www.wikidata.org/entity/Q33761086

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Nitric-oxide dioxygenase function of human cytoglobin with cellular reductants and in rat hepatocytes A critical review of vitamin C for the prevention of age-related cognitive decline and Alzheimer's disease Treatment of Pancreatic Cancer with Pharmacological Ascorbate Recent applications of engineered animal antioxidant deficiency models in human nutrition and chronic disease Regulation of vitamin C homeostasis during deficiency Human genetic variation influences vitamin C homeostasis by altering vitamin C transport and antioxidant enzyme function Expression, purification and low-resolution structure of human vitamin C transporter SVCT1 (SLC23A1)Behavioral and monoamine changes following severe vitamin C deficiency Ascorbic acid and the brain: rationale for the use against cognitive decline.Vitamin C in Stem Cell Biology: Impact on Extracellular Matrix Homeostasis and Epigenetics.1H NMR-based profiling reveals differential immune-metabolic networks during influenza virus infection in obese mice.Low ascorbic acid and increased oxidative stress in gulo(-/-) mice during development Low vitamin C and increased oxidative stress and cell death in mice that lack the sodium-dependent vitamin C transporter SVCT2.Increased expression of SVCT2 in a new mouse model raises ascorbic acid in tissues and protects against paraquat-induced oxidative damage in lung.Current limitations of murine models in oncology for ascorbate research.Tubular transport: core curriculum 2010.Vitamin C: a concentration-function approach yields pharmacology and therapeutic discoveries.Differential regulation of the ascorbic acid transporter SVCT2 during development and in response to ascorbic acid depletion.Epididymal Region-Specific miRNA Expression and DNA Methylation and Their Roles in Controlling Gene Expression in Rats.The SLC23 family of ascorbate transporters: ensuring that you get and keep your daily dose of vitamin C Aging Selectively Modulates Vitamin C Transporter Expression Patterns in the Kidney.Genetic Variation in Human Vitamin C Transporter Genes in Common Complex Diseases.Genetic variation at the SLC23A1 locus is associated with circulating concentrations of L-ascorbic acid (vitamin C): evidence from 5 independent studies with >15,000 participants.Ascorbic acid: chemistry, biology and the treatment of cancer.The epigenetic role of vitamin C in health and disease.Intestinal dehydroascorbic acid (DHA) transport mediated by the facilitative sugar transporters, GLUT2 and GLUT8.Marginal Ascorbate Status (Hypovitaminosis C) Results in an Attenuated Response to Vitamin C Supplementation Molecular basis of cardioprotective effect of antioxidant vitamins in myocardial infarction.Chronic ethanol exposure closes the door to vitamin C in pancreatic acinar cells. Focus on "Uptake of ascorbic acid by pancreatic acinar cells is negatively impacted by chronic alcohol exposure".Parenteral ascorbate as a cancer therapeutic: a reassessment based on pharmacokinetics.The human sodium-dependent ascorbic acid transporters SLC23A1 and SLC23A2 do not mediate ascorbic acid release in the proximal renal epithelial cell.Myths, artifacts, and fatal flaws: identifying limitations and opportunities in vitamin C research.Nuclear receptor-mediated alleviation of alcoholic fatty liver by polyphenols contained in alcoholic beverages.New criteria for supplementation of selected micronutrients in the era of nutrigenetics and nutrigenomics.Vitamin C: the known and the unknown and Goldilocks.Oxidative stress, serotonergic changes and decreased ultrasonic vocalizations in a mouse model of Smith-Lemli-Opitz syndrome.Uptake of ascorbic acid by pancreatic acinar cells is negatively impacted by chronic alcohol exposure.Does Vitamin C Influence Neurodegenerative Diseases and Psychiatric Disorders?Chemical Transport Knockout for Oxidized Vitamin C, Dehydroascorbic Acid, Reveals Its Functions in vivo.Maternal vitamin C deficiency during pregnancy results in transient fetal and placental growth retardation in guinea pigs.

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P2860

Vitamin C transporter Slc23a1 links renal reabsorption, vitamin C tissue accumulation, and perinatal survival in mice.

http://www.wikidata.org/entity/Q33761086

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Vitamin C transporter Slc23a1 ...... nd perinatal survival in mice.

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Christopher P Corpe

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He Sun

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Hongbin Tu

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Jin Wang

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Jurgen Schnermann

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Mark Levine

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Michael Graham Espey

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Peter Eck

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Robert Faulhaber-Walter

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Robert L Nussbaum

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P2860

Vitamin C pharmacokinetics in healthy volunteers: evidence for a recommended dietary allowance

Aortic wall damage in mice unable to synthesize ascorbic acid

A simple salting out procedure for extracting DNA from human nucleated cells

Glucose transporter isoforms GLUT1 and GLUT3 transport dehydroascorbic acid

A family of mammalian Na+-dependent L-ascorbic acid transporters

High-dose vitamin therapy stimulates variant enzymes with decreased coenzyme binding affinity (increased K(m)): relevance to genetic disease and polymorphisms

Genetic variation in the sodium-dependent vitamin C transporters, SLC23A1, and SLC23A2 and risk for preterm delivery

Vitamin C is not essential for carnitine biosynthesis in vivo: verification in vitamin C-depleted senescence marker protein-30/gluconolactonase knockout mice

Ascorbic acid and dehydroascorbic acid measurements in human plasma and serum

Senescence marker protein 30 functions as gluconolactonase in L-ascorbic acid biosynthesis, and its knockout mice are prone to scurvy

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10.1172/JCI39191

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