Fate tracing reveals hepatic stellate cells as dominant contributors to liver fibrosis independent of its aetiology. - Wikidata - wikimedia - TriplyDB

Fate tracing reveals hepatic stellate cells as dominant contributors to liver fibrosis independent of its aetiology.

Fate tracing reveals hepatic stellate cells as dominant contributors to liver fibrosis independent of its aetiology.

http://www.wikidata.org/entity/Q33762356

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Type-1 pericytes accumulate after tissue injury and produce collagen in an organ-dependent manner Homing in on the hepatic scar: recent advances in cell-specific targeting of liver fibrosis Evolving Insights on Metabolism, Autophagy, and Epigenetics in Liver Myofibroblasts Promising Therapy Candidates for Liver Fibrosis Advances in Liver Regeneration: Revisiting Hepatic Stem/Progenitor Cells and Their Origin Controversies over the Epithelial-to-Mesenchymal Transition in Liver Fibrosis Cytoglobin as a Marker of Hepatic Stellate Cell-derived Myofibroblasts Potential Use of Biological Proteins for Liver Failure Therapy Animal Models for Fibrotic Liver Diseases: What We Have, What We Need, and What Is under Development The role of miRNAs in stress-responsive hepatic stellate cells during liver fibrosis Healing scars: targeting pericytes to treat fibrosis Therapeutic targeting of liver inflammation and fibrosis by nanomedicine Bone marrow-derived fibrocytes contribute to liver fibrosis Reversibility and heritability of liver fibrosis: Implications for research and therapy Culture Model of Rat Portal Myofibroblasts High-yield and high-purity isolation of hepatic stellate cells from normal and fibrotic mouse livers.Stiffening hydrogels for investigating the dynamics of hepatic stellate cell mechanotransduction during myofibroblast activation Molecular mechanism and treatment of viral hepatitis-related liver fibrosis.The Role of Mesothelial Cells in Liver Development, Injury, and Regeneration Targeting Mechanotransduction at the Transcriptional Level: YAP and BRD4 Are Novel Therapeutic Targets for the Reversal of Liver Fibrosis New Concepts on Pathogenesis and Diagnosis of Liver Fibrosis; A Review Article Elastin in the Liver Strategies and endpoints of antifibrotic drug trials: Summary and recommendations from the AASLD Emerging Trends Conference, Chicago, June 2014.Membrane-to-Nucleus Signals and Epigenetic Mechanisms for Myofibroblastic Activation and Desmoplastic Stroma: Potential Therapeutic Targets for Liver Metastasis?Interaction of TWEAK with Fn14 leads to the progression of fibrotic liver disease by directly modulating hepatic stellate cell proliferation Epithelial-mesenchymal transition in tissue repair and fibrosis Genetic tools for identifying and manipulating fibroblasts in the mouse p62/SQSTM1 by Binding to Vitamin D Receptor Inhibits Hepatic Stellate Cell Activity, Fibrosis, and Liver Cancer Loss of pericyte smoothened activity in mice with genetic deficiency of leptin.Imidazoline I2 receptor inhibitor idazoxan regulates the progression of hepatic fibrosis via Akt-Nrf2-Smad2/3 signaling pathway.Human Menstrual Blood-Derived Stem Cells Ameliorate Liver Fibrosis in Mice by Targeting Hepatic Stellate Cells via Paracrine Mediators.TGF-β-induced hepatocyte lincRNA-p21 contributes to liver fibrosis in mice SerpinB3 Promotes Pro-fibrogenic Responses in Activated Hepatic Stellate Cells.The origin of fibrogenic myofibroblasts in fibrotic liver.The roles of Toll-like receptor 4 in the pathogenesis of pathogen-associated biliary fibrosis caused by Clonorchis sinensis.Focal Adhesion Kinase Regulates Hepatic Stellate Cell Activation and Liver Fibrosis.Interspecies NASH disease activity whole-genome profiling identifies a fibrogenic role of PPARα-regulated dermatopontin Platelet-derived growth factor receptor α in hepatocellular carcinoma is a prognostic marker independent of underlying liver cirrhosis Integrins, myofibroblasts, and organ fibrosis.Origin of myofibroblasts in the fibrotic liver in mice

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Fate tracing reveals hepatic stellate cells as dominant contributors to liver fibrosis independent of its aetiology.

http://www.wikidata.org/entity/Q33762356

description

2013 nî lūn-bûn

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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2013 թվականի հունվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Fate tracing reveals hepatic s ...... independent of its aetiology.

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Fate tracing reveals hepatic s ...... independent of its aetiology.

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Fate tracing reveals hepatic s ...... independent of its aetiology.

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Fate tracing reveals hepatic s ...... independent of its aetiology.

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Fate tracing reveals hepatic s ...... independent of its aetiology.

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Fate tracing reveals hepatic s ...... independent of its aetiology.

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Nature Communications

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Fate tracing reveals hepatic s ...... s independent of its aetiology

@en

P2093

Christine C Hsu

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Dianne H Dapito

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Ingmar Mederacke

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Juliane S Troeger

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Robert F Schwabe

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Xueru Mu

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P2860

What's new in liver fibrosis? The origin of myofibroblasts in liver fibrosis

Pericytes: developmental, physiological, and pathological perspectives, problems, and promises

Lecithin: retinol acyltransferase protein is distributed in both hepatic stellate cells and endothelial cells of normal rodent and human liver

A global double-fluorescent Cre reporter mouse

A robust and high-throughput Cre reporting and characterization system for the whole mouse brain

Hepatic stellate cells: protean, multifunctional, and enigmatic cells of the liver

Mechanisms of hepatic fibrogenesis

TLR4 enhances TGF-beta signaling and hepatic fibrosis

Mechanisms of fibrosis: therapeutic translation for fibrotic disease

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Jean-Philippe Pradère

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